John J. Eppig

28.0k total citations · 6 hit papers
211 papers, 22.5k citations indexed

About

John J. Eppig is a scholar working on Public Health, Environmental and Occupational Health, Molecular Biology and Reproductive Medicine. According to data from OpenAlex, John J. Eppig has authored 211 papers receiving a total of 22.5k indexed citations (citations by other indexed papers that have themselves been cited), including 169 papers in Public Health, Environmental and Occupational Health, 105 papers in Molecular Biology and 84 papers in Reproductive Medicine. Recurrent topics in John J. Eppig's work include Reproductive Biology and Fertility (168 papers), Ovarian function and disorders (59 papers) and Sperm and Testicular Function (51 papers). John J. Eppig is often cited by papers focused on Reproductive Biology and Fertility (168 papers), Ovarian function and disorders (59 papers) and Sperm and Testicular Function (51 papers). John J. Eppig collaborates with scholars based in United States, Japan and China. John J. Eppig's co-authors include Karen Wigglesworth, Marilyn J. O’Brien, Allen C. Schroeder, Frank L. Pendola, Koji Sugiura, You‐Qiang Su, Martin M. Matzuk, Stephen M. Downs, Maria M. Viveiros and Kathleen H. Burns and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

John J. Eppig

210 papers receiving 21.9k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Development in vitro of mouse oocytes from primordial follicles

1996 783 citations John J. Eppig Biology of Reproduction profile →
Oocyte control of ovarian follicular development and function in mammals

2001 767 citations John J. Eppig Reproduction profile →
Intercellular Communication in the Mammalian Ovary: Oocytes Carry the Conversation

2002 744 citations Martin M. Matzuk, John J. Eppig et al. Science profile →
Cyclin D2 is an FSH-responsive gene involved in gonadal cell proliferation and oncogenesis

1996 593 citations John J. Eppig et al. profile →
Capacity of Mouse Oocytes from Preantral Follicles to Undergo Embryogenesis and Development to Live Young after Growth, Maturation, and Fertilization in Vitro1

1989 507 citations John J. Eppig, Allen C. Schroeder Biology of Reproduction profile →
Granulosa Cell Ligand NPPC and Its Receptor NPR2 Maintain Meiotic Arrest in Mouse Oocytes

2010 455 citations Meijia Zhang, You‐Qiang Su et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
John J. Eppig United States	84	17.4k	11.3k	10.1k	3.8k	1.7k	211	22.5k
Jonathan L. Tilly United States	65	9.0k 0.5×	7.7k 0.7×	5.4k 0.5×	2.1k 0.6×	2.1k 1.2×	161	15.8k
JoAnne S. Richards United States	81	9.4k 0.5×	6.5k 0.6×	6.8k 0.7×	6.7k 1.8×	4.1k 2.3×	225	19.9k
Dirk G. de Rooij Netherlands	82	9.1k 0.5×	12.4k 1.1×	11.9k 1.2×	8.3k 2.2×	847 0.5×	288	22.5k
Qing‐Yuan Sun China	60	7.4k 0.4×	7.9k 0.7×	3.5k 0.3×	2.2k 0.6×	888 0.5×	467	13.7k
R. M. Moor United Kingdom	59	6.3k 0.4×	3.4k 0.3×	3.5k 0.3×	2.6k 0.7×	815 0.5×	188	10.0k
Gregory S. Kopf United States	60	7.8k 0.4×	3.3k 0.3×	8.2k 0.8×	1.7k 0.4×	645 0.4×	137	11.7k
Rex A. Hess United States	68	3.5k 0.2×	4.8k 0.4×	7.3k 0.7×	5.9k 1.6×	792 0.5×	232	14.7k
Atsuo Ogura Japan	66	7.2k 0.4×	10.9k 1.0×	5.3k 0.5×	5.3k 1.4×	736 0.4×	325	16.6k
Shunichi Shimasaki United States	69	6.1k 0.4×	8.6k 0.8×	3.0k 0.3×	2.4k 0.6×	780 0.4×	158	13.7k
Alfonso Gutiérrez‐Adán Spain	59	5.9k 0.3×	4.5k 0.4×	3.5k 0.3×	2.9k 0.8×	1.2k 0.7×	304	11.5k

Countries citing papers authored by John J. Eppig

Since Specialization

Citations

This map shows the geographic impact of John J. Eppig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John J. Eppig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John J. Eppig more than expected).

Fields of papers citing papers by John J. Eppig

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John J. Eppig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John J. Eppig. The network helps show where John J. Eppig may publish in the future.

Co-authorship network of co-authors of John J. Eppig

This figure shows the co-authorship network connecting the top 25 collaborators of John J. Eppig. A scholar is included among the top collaborators of John J. Eppig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John J. Eppig. John J. Eppig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Robinson, Jerid W., Meijia Zhang, Leia C. Shuhaibar, et al.. (2012). Luteinizing hormone reduces the activity of the NPR2 guanylyl cyclase in mouse ovarian follicles, contributing to the cyclic GMP decrease that promotes resumption of meiosis in oocytes. Developmental Biology. 366(2). 308–316. 111 indexed citations

Zhang, Meijia, You‐Qiang Su, Koji Sugiura, et al.. (2011). Estradiol Promotes and Maintains Cumulus Cell Expression of Natriuretic Peptide Receptor 2 (NPR2) and Meiotic Arrest in Mouse Oocytes In Vitro. Endocrinology. 152(11). 4377–4385. 130 indexed citations

Su, You‐Qiang, Koji Sugiura, Qinglei Li, et al.. (2010). Mouse Oocytes Enable LH-Induced Maturation of the Cumulus-Oocyte Complex via Promoting EGF Receptor-Dependent Signaling. Molecular Endocrinology. 24(6). 1230–1239. 101 indexed citations

Zhang, Meijia, You‐Qiang Su, Koji Sugiura, Guoliang Xia, & John J. Eppig. (2010). Granulosa Cell Ligand NPPC and Its Receptor NPR2 Maintain Meiotic Arrest in Mouse Oocytes. Science. 330(6002). 366–369. 455 indexed citations breakdown →

Su, You‐Qiang, Koji Sugiura, Karen Wigglesworth, et al.. (2007). Oocyte regulation of metabolic cooperativity between mouse cumulus cells and oocytes: BMP15 and GDF9 control cholesterol biosynthesis in cumulus cells. Development. 135(1). 111–121. 306 indexed citations

Mehlmann, Lisa M., Yoshinaga Saeki, Shigeru Tanaka, et al.. (2004). The G _s -Linked Receptor GPR3 Maintains Meiotic Arrest in Mammalian Oocytes. Science. 306(5703). 1947–1950. 264 indexed citations

Su, You‐Qiang, James M. Denegre, Karen Wigglesworth, et al.. (2003). Oocyte-dependent activation of mitogen-activated protein kinase (ERK1/2) in cumulus cells is required for the maturation of the mouse oocyte–cumulus cell complex. Developmental Biology. 263(1). 126–138. 182 indexed citations

Eppig, John J., et al.. (2002). The future of the oocyte : basic and clinical aspects. Springer eBooks. 1 indexed citations

Fuente, Rabindranath De La, Marilyn J. O’Brien, & John J. Eppig. (1999). Epidermal growth factor enhances preimplantation developmental competence of maturing mouse oocytes. Human Reproduction. 14(12). 3060–3068. 100 indexed citations

10.

Hirao, Yuji & John J. Eppig. (1999). Analysis of the mechanism(s) of metaphase I‐arrest in strain LT mouse oocytes: Delay in the acquisition of competence to undergo the metaphase I/anaphase transition. Molecular Reproduction and Development. 54(3). 311–318. 2 indexed citations

11.

Wandji, S.-A., et al.. (1997). Initiation of growth of baboon primordial follicles in vitro. Human Reproduction. 12(9). 1993–2001. 180 indexed citations

12.

Eppig, John J.. (1996). Development in vitro of mouse oocytes from primordial follicles. Biology of Reproduction. 54(1). 197–207. 783 indexed citations breakdown →

13.

Eppig, John J., Marilyn J. O’Brien, & Karen Wigglesworth. (1996). Mammalian oocyte growth and development in vitro. Molecular Reproduction and Development. 44(2). 260–273. 18 indexed citations

14.

Eppig, John J., Marilyn J. O’Brien, & Karen Wigglesworth. (1996). Mammalian oocyte growth and development in vitro. Molecular Reproduction and Development. 44(2). 260–273. 194 indexed citations

15.

Eppig, John J., Karen Wigglesworth, & Marilyn J. O’Brien. (1992). Comparison of embryonic developmental competence of mouse oocytes grown with and without serum. Molecular Reproduction and Development. 32(1). 33–40. 61 indexed citations

16.

Eppig, John J., Susan A.J. Daniel, & Allen C. Schroeder. (1988). Growth and Development of Mouse Oocytes in Vitro^a. Annals of the New York Academy of Sciences. 541(1). 205–210. 5 indexed citations

17.

Eppig, John J., Rolf Freter, Patricia F. Ward‐Bailey, & Richard M. Schultz. (1983). Inhibition of oocyte maturation in the mouse: Participation of cAMP, steroid hormones, and a putative maturation-inhibitory factor. Developmental Biology. 100(1). 39–49. 132 indexed citations

18.

Eppig, John J.. (1981). Regulation by Sulfated Glycosaminoglycans of the Expansion of Cumuli Oophori Isolated from Mice. Biology of Reproduction. 25(3). 599–608. 33 indexed citations

19.

Eppig, John J.. (1981). Preimplantation embryonic development of spontaneous mouse parthenotes after oocyte meiotic maturation in vitro. Gamete Research. 4(1). 3–13. 13 indexed citations

20.

Eppig, John J., et al.. (1978). Effects of progesterone and oestradiol-17β on the spontaneous meiotic maturation of mouse oocytes. Reproduction. 53(1). 99–101. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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