Johan Östman

1.0k total citations · 1 hit paper
5 papers, 792 citations indexed

About

Johan Östman is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Johan Östman has authored 5 papers receiving a total of 792 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 4 papers in Physiology and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Johan Östman's work include Ion channel regulation and function (2 papers), Pain Mechanisms and Treatments (2 papers) and Alzheimer's disease research and treatments (2 papers). Johan Östman is often cited by papers focused on Ion channel regulation and function (2 papers), Pain Mechanisms and Treatments (2 papers) and Alzheimer's disease research and treatments (2 papers). Johan Östman collaborates with scholars based in United Kingdom, Sweden and Germany. Johan Östman's co-authors include John N. Wood, Bjarke Abrahamsen, Norbert Klugbauer, Caroline Fertleman, Frances Elmslie, R. Mark Gardiner, Mark D. Baker, Keith A. Parker, Michele Rees and Mark D. Baker and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and The Journal of Physiology.

In The Last Decade

Johan Östman

5 papers receiving 775 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

SCN9A Mutations in Paroxysmal Extreme Pain Disorder: Allelic Variants Underlie Distinct Channel Defects and Phenotypes

2006 559 citations Caroline Fertleman, Mark D. Baker et al. Neuron profile →

Peers

Johan Östman

MB

PHYSI

CMN

SS

CCM

Ruirui Lu Germany

Derek J. Trezise United Kingdom

Peihan Orestes United States

María A. Gandini Canada

Iuliia Vitko United States

Tomonari Watabiki Japan

Joe Mikusa United States

Tal Hoffmann Germany

Alessandra Levato Italy

Ho‐Won Kang South Korea

Ruirui Lu Germany

Johan Östman

327 ×0.6

MB

427 ×0.9

PHYSI

243 ×0.9

CMN

116 ×1.1

SS

44 ×0.5

CCM

Citations per year, relative to Johan Östman Johan Östman (= 1×) peers Ruirui Lu

Countries citing papers authored by Johan Östman

Since Specialization

Citations

This map shows the geographic impact of Johan Östman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Johan Östman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Johan Östman more than expected).

Fields of papers citing papers by Johan Östman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Johan Östman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Johan Östman. The network helps show where Johan Östman may publish in the future.

Co-authorship network of co-authors of Johan Östman

This figure shows the co-authorship network connecting the top 25 collaborators of Johan Östman. A scholar is included among the top collaborators of Johan Östman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Johan Östman. Johan Östman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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5 of 5 papers shown

1.

Östman, Johan, et al.. (2012). 20-Hydroxyeicosatetraenoic Acid (20-HETE) Is a Novel Activator of Transient Receptor Potential Vanilloid 1 (TRPV1) Channel. Journal of Biological Chemistry. 287(17). 13868–13876. 61 indexed citations

2.

Östman, Johan, Mohammed A. Nassar, John N. Wood, & Mark D. Baker. (2007). GTP up‐regulated persistent Na⁺ current and enhanced nociceptor excitability require Na_V1.9. The Journal of Physiology. 586(4). 1077–1087. 93 indexed citations

3.

Fertleman, Caroline, Mark D. Baker, Keith A. Parker, et al.. (2006). SCN9A Mutations in Paroxysmal Extreme Pain Disorder: Allelic Variants Underlie Distinct Channel Defects and Phenotypes. Neuron. 52(5). 767–774. 559 indexed citations breakdown →

4.

Mališauskas, Mantas, Johan Östman, Adas Darinskas, et al.. (2004). Does the Cytotoxic Effect of Transient Amyloid Oligomers from Common Equine Lysozyme in Vitro Imply Innate Amyloid Toxicity?. Journal of Biological Chemistry. 280(8). 6269–6275. 61 indexed citations

5.

Olofsson, Anders, Johan Östman, & Erik Lundgren. (2002). Amyloid: Morphology and Toxicity. Clinical Chemistry and Laboratory Medicine (CCLM). 40(12). 1266–70. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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