Michele Rees

2.9k total citations · 2 hit papers
13 papers, 2.2k citations indexed

About

Michele Rees is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Michele Rees has authored 13 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 3 papers in Genetics. Recurrent topics in Michele Rees's work include Ion channel regulation and function (8 papers), Neuroscience and Neuropharmacology Research (8 papers) and Nicotinic Acetylcholine Receptors Study (4 papers). Michele Rees is often cited by papers focused on Ion channel regulation and function (8 papers), Neuroscience and Neuropharmacology Research (8 papers) and Nicotinic Acetylcholine Receptors Study (4 papers). Michele Rees collaborates with scholars based in United Kingdom, United States and Germany. Michele Rees's co-authors include J. Elaine Barclay, Edward Perez‐Reyes, Magali Williamson, Jung‐Ha Lee, Leanne L. Cribbs, Margaret Fox, Asif N. Daud, R. Mark Gardiner, Antonio E. Lacerda and John N. Wood and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Neuron.

In The Last Decade

Michele Rees

13 papers receiving 2.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Molecular characterization of a neuronal low-voltage-activated T-type calcium channel

1998 615 citations Edward Perez‐Reyes, Leanne L. Cribbs et al. Nature profile →
SCN9A Mutations in Paroxysmal Extreme Pain Disorder: Allelic Variants Underlie Distinct Channel Defects and Phenotypes

2006 559 citations Caroline Fertleman, Mark D. Baker et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Michele Rees United Kingdom	11	1.8k	1.2k	558	492	149	13	2.2k
Asif N. Daud United States	11	2.1k 1.2×	1.6k 1.3×	753 1.3×	340 0.7×	163 1.1×	12	2.6k
T P Snutch Canada	14	2.0k 1.1×	1.7k 1.4×	361 0.6×	400 0.8×	137 0.9×	15	2.4k
Jawed Hamid Canada	25	1.9k 1.1×	1.3k 1.1×	338 0.6×	449 0.9×	154 1.0×	30	2.3k
Anthony Stea Canada	20	2.2k 1.2×	1.8k 1.5×	581 1.0×	240 0.5×	97 0.7×	23	2.7k
Gayle M. Passmore United Kingdom	9	1.1k 0.6×	804 0.7×	320 0.6×	557 1.1×	156 1.0×	11	1.4k
Lakshmi Sangameswaran United States	20	1.5k 0.8×	1.1k 0.9×	180 0.3×	963 2.0×	255 1.7×	32	2.2k
Hai Yan United States	29	1.1k 0.6×	774 0.6×	275 0.5×	195 0.4×	201 1.3×	50	2.1k
Brian W. Strassle United States	14	1.6k 0.9×	1.2k 1.0×	734 1.3×	324 0.7×	94 0.6×	20	2.3k
Paola Imbrici Italy	26	1.7k 0.9×	1.0k 0.8×	814 1.5×	107 0.2×	101 0.7×	97	2.1k
Gábor Czirják Hungary	22	2.0k 1.1×	957 0.8×	522 0.9×	373 0.8×	354 2.4×	42	2.4k

Countries citing papers authored by Michele Rees

Since Specialization

Citations

This map shows the geographic impact of Michele Rees's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michele Rees with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michele Rees more than expected).

Fields of papers citing papers by Michele Rees

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michele Rees. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michele Rees. The network helps show where Michele Rees may publish in the future.

Co-authorship network of co-authors of Michele Rees

This figure shows the co-authorship network connecting the top 25 collaborators of Michele Rees. A scholar is included among the top collaborators of Michele Rees based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michele Rees. Michele Rees is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Turner, Katherine J., Jacqueline Hoyle, Leonardo E. Valdivia, et al.. (2019). Abrogation of Stem Loop Binding Protein (Slbp) function leads to a failure of cells to transition from proliferation to differentiation, retinal coloboma and midline axon guidance deficits. PLoS ONE. 14(1). e0211073–e0211073. 6 indexed citations

Fertleman, Caroline, Mark D. Baker, Keith A. Parker, et al.. (2006). SCN9A Mutations in Paroxysmal Extreme Pain Disorder: Allelic Variants Underlie Distinct Channel Defects and Phenotypes. Neuron. 52(5). 767–774. 559 indexed citations breakdown →

Donato, R. J., Karen Page, Manuela Nieto‐Rostro, et al.. (2006). The ducky^2JMutation inCacna2d2Results in Reduced Spontaneous Purkinje Cell Activity and Altered Gene Expression. Journal of Neuroscience. 26(48). 12576–12586. 55 indexed citations

Brodbeck, Jens, Anthony Davies, Jo‐Maree Courtney, et al.. (2002). The Ducky Mutation in Cacna2d2 Results in Altered Purkinje Cell Morphology and Is Associated with the Expression of a Truncated α2δ-2 Protein with Abnormal Function. Journal of Biological Chemistry. 277(10). 7684–7693. 122 indexed citations

Taske, Nichole, Magali Williamson, Andrew Makoff, et al.. (2002). Evaluation of the positional candidate gene CHRNA7 at the juvenile myoclonic epilepsy locus (EJM2) on chromosome 15q13–14. Epilepsy Research. 49(2). 157–172. 39 indexed citations

Robinson, Robert, Nichole Taske, Thomas Sander, et al.. (2002). Linkage analysis between childhood absence epilepsy and genes encoding GABAA and GABAB receptors, voltage-dependent calcium channels, and the ECA1 region on chromosome 8q. Epilepsy Research. 48(3). 169–179. 23 indexed citations

Barclay, J. Elaine, Marina Mione, Susan L. Ackerman, et al.. (2001). Ducky Mouse Phenotype of Epilepsy and Ataxia Is Associated with Mutations in theCacna2d2Gene and Decreased Calcium Channel Current in Cerebellar Purkinje Cells. Journal of Neuroscience. 21(16). 6095–6104. 242 indexed citations

Barclay, J. Elaine & Michele Rees. (2000). Genomic organization of the mouse and human α2δ2 voltage-dependent calcium channel subunit genes. Mammalian Genome. 11(12). 1142–1144. 18 indexed citations

Formstone, Caroline J., J. Elaine Barclay, Michele Rees, & Peter Little. (2000). Chromosomal localization of Celsr2 and Celsr3 in the mouse; Celsr3 is a candidate for the tippy (tip) lethal mutant on Chromosome 9. Mammalian Genome. 11(5). 392–394. 10 indexed citations

10.

Barclay, J. Elaine & Michele Rees. (1999). Mouse Models of Spike‐Wave Epilepsy. Epilepsia. 40(s3). 17–22. 19 indexed citations

11.

Perez‐Reyes, Edward, Leanne L. Cribbs, Asif N. Daud, et al.. (1998). Molecular characterization of a neuronal low-voltage-activated T-type calcium channel. Nature. 391(6670). 896–900. 615 indexed citations breakdown →

12.

Cribbs, Leanne L., Jung‐Ha Lee, Jie Yang, et al.. (1998). Cloning and Characterization of α1H From Human Heart, a Member of the T-Type Ca²⁺Channel Gene Family. Circulation Research. 83(1). 103–109. 481 indexed citations

13.

Parkinson, Nick, J. Elaine Barclay, Mark Gardiner, & Michele Rees. (1997). An inverse PCR strategy for the recovery of end fragments from the pRML YAC vector. 2(1). 133–135. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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