Jocelyn D. Weissman

1.1k total citations
33 papers, 950 citations indexed

About

Jocelyn D. Weissman is a scholar working on Immunology, Molecular Biology and Cancer Research. According to data from OpenAlex, Jocelyn D. Weissman has authored 33 papers receiving a total of 950 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Immunology, 16 papers in Molecular Biology and 4 papers in Cancer Research. Recurrent topics in Jocelyn D. Weissman's work include T-cell and B-cell Immunology (16 papers), Immune Cell Function and Interaction (12 papers) and Genomics and Chromatin Dynamics (12 papers). Jocelyn D. Weissman is often cited by papers focused on T-cell and B-cell Immunology (16 papers), Immune Cell Function and Interaction (12 papers) and Genomics and Chromatin Dynamics (12 papers). Jocelyn D. Weissman collaborates with scholars based in United States, Malaysia and Japan. Jocelyn D. Weissman's co-authors include Dinah S. Singer, T. Kevin Howcroft, Anne Gégonne, Aparna Raval, Jae Ryoung Hwang, Kelly P. Kearse, Jie Mu, Yoshihiro Nakatani, Louis Schiltz and Paul A. Roche and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Molecular Cell.

In The Last Decade

Jocelyn D. Weissman

33 papers receiving 943 citations

Peers

Countries citing papers authored by Jocelyn D. Weissman

Since Specialization

Citations

This map shows the geographic impact of Jocelyn D. Weissman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jocelyn D. Weissman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jocelyn D. Weissman more than expected).

Fields of papers citing papers by Jocelyn D. Weissman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jocelyn D. Weissman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jocelyn D. Weissman. The network helps show where Jocelyn D. Weissman may publish in the future.

Co-authorship network of co-authors of Jocelyn D. Weissman

This figure shows the co-authorship network connecting the top 25 collaborators of Jocelyn D. Weissman. A scholar is included among the top collaborators of Jocelyn D. Weissman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jocelyn D. Weissman. Jocelyn D. Weissman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	The intrinsic kinase activity of BRD4 spans its BD2-B-BID domains 2021 ·Journal of Biological Chemistry ·Jocelyn D. Weissman, Amit Kumar Singh, Ballachanda N. Devaiah, Peter Schuck, Ross C. Larue, Dinah S. Singer	13
2	MYC protein stability is negatively regulated by BRD4 2020 ·Proceedings of the National Academy of Sciences ·Ballachanda N. Devaiah, Jie Mu, (unknown), Sheetal Uppal, Jocelyn D. Weissman, Dan Cheng, Laura Baranello, Zuqin Nie, David Levens, Dinah S. Singer	108
3	Differential context-specific impact of individual core promoter elements on transcriptional dynamics 2017 ·Molecular Biology of the Cell ·(unknown), (unknown), Jocelyn D. Weissman, Daniel R. Larson, Dinah S. Singer	24
4	Major Histocompatibility Complex Class I Core Promoter Elements Are Not Essential for Transcription in vivo 2013 ·Molecular and Cellular Biology ·(unknown), Jocelyn D. Weissman, (unknown), Jie Mu, Dinah S. Singer	9
5	Three Novel Downstream Promoter Elements Regulate MHC Class I Promoter Activity in Mammalian Cells 2010 ·PLoS ONE ·Namhoon Lee, Shankar S. Iyer, Jie Mu, Jocelyn D. Weissman, Anat Ohali, T. Kevin Howcroft, Brian A. Lewis, Dinah S. Singer	13
6	In Vivo Expression of MHC Class I Genes Depends on the Presence of a Downstream Barrier Element 2009 ·PLoS ONE ·Helit Cohen, Palak R. Parekh, (unknown), (unknown), Jocelyn D. Weissman, Dinah S. Singer	3
7	Histone Modifications, but Not Nucleosomal Positioning, Correlate with Major Histocompatibility Complex Class I Promoter Activity in Different Tissues In Vivo 2008 ·Molecular and Cellular Biology ·(unknown), Jocelyn D. Weissman, Anne Gégonne, Helit Cohen, Dinah S. Singer	16
8	TFIID component TAF7 functionally interacts with both TFIIH and P-TEFb 2008 ·Proceedings of the National Academy of Sciences ·Anne Gégonne, Jocelyn D. Weissman, Hanxin Lu, Meisheng Zhou, Arindam Das-Gupta, R. Ribble, John Brady, Dinah S. Singer	31
9	A T Lymphocyte-Specific Transcription Complex Containing RUNX1 Activates MHC Class I Expression 2005 ·The Journal of Immunology ·T. Kevin Howcroft, Jocelyn D. Weissman, Anne Gégonne, Dinah S. Singer	20
10	Distinct Transcriptional Pathways Regulate Basal and Activated Major Histocompatibility Complex Class I Expression 2003 ·Molecular and Cellular Biology ·T. Kevin Howcroft, Aparna Raval, Jocelyn D. Weissman, Anne Gégonne, Dinah S. Singer	35
11	Assay of an Intrinsic Acetyltransferase Activity of the Transcriptional Coactivator CIITA 2003 ·Methods in enzymology on CD-ROM/Methods in enzymology ·Jocelyn D. Weissman, Aparna Raval, Dinah S. Singer	8
12	Transcriptional Coactivator, CIITA, Is an Acetyltransferase that Bypasses a Promoter Requirement for TAFII250 2001 ·Molecular Cell ·Aparna Raval, T. Kevin Howcroft, Jocelyn D. Weissman, Susan Kirshner, (unknown), Kazunari K. Yokoyama, Jenny P.‐Y. Ting, Dinah S. Singer	98
13	Extensive interactions between HIV TAT and TAFII250 2001 ·Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology ·Jocelyn D. Weissman, Jae Ryoung Hwang, Dinah S. Singer	9
14	TAFII250-independent Transcription Can Be Conferred on a TAFII250-dependent Basal Promoter by Upstream Activators 2000 ·Journal of Biological Chemistry ·Jocelyn D. Weissman, T. Kevin Howcroft, Dinah S. Singer	20
15	Upstream Stimulatory Factor Regulates Major Histocompatibility Complex Class I Gene Expression: the U2ΔE4 Splice Variant Abrogates E-Box Activity 1999 ·Molecular and Cellular Biology ·T. Kevin Howcroft, Charles J. Murphy, Jocelyn D. Weissman, (unknown), Michèle Sawadogo, Dinah S. Singer	33
16	Repression of the nonclassicalMHC class I geneH2-M1 bycis-acting silencer DNA elements 1996 ·Immunogenetics ·T. Kevin Howcroft, Jocelyn D. Weissman, Stuart Rudikoff, William I. Frels, Dinah S. Singer	5
17	Active Repression of Major Histocompatibility Complex Class I Genes in a Human Neuroblastoma Cell Line 1996 ·Journal of Biological Chemistry ·Charles J. Murphy, (unknown), (unknown), Jocelyn D. Weissman, Dinah S. Singer	33
18	In Vivo Function of Regulatory DNA Sequence Elements of a Major Histocompatibility Complex Class I Gene 1992 ·Molecular and Cellular Biology ·Jean E. Maguire, William I. Frels, (unknown), Jocelyn D. Weissman, Dinah S. Singer	19
19	A Complex Regulatory DNA Element Associated with a Major Histocompatibility Complex Class I Gene Consists of Both a Silencer and an Enhancer 1991 ·Molecular and Cellular Biology ·Jocelyn D. Weissman, Dinah S. Singer	10
20	Striking Similarities between the Regulatory Mechanisms Governing Yeast Mating-Type Genes and Mammalian Major Histocompatibility Complex Genes 1991 ·Molecular and Cellular Biology ·Jocelyn D. Weissman, Dinah S. Singer	15

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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