Jo Beth Harry
Impact in
- Epidemiology top 5%
- Cervical Cancer and HPV Research
- Hepatitis B Virus Studies
- Oncology top 10%
- Cancer-related Molecular Pathways
Papers in
-
- Cardiac electrophysiology and arrhythmias 5
- Virology 1
- Co-authors
- Denise A. GallowayErik EsplingJens Oliver FunkBruce StillmanS WagaF O WettsteinNikolai M. SoldatovEvgeny Kobrinsky
- Journals
- Journal of Virology (4 papers)Journal of Biological Chemistry (3 papers)Antimicrobial Agents and Chemotherapy (2 papers)Genes & Development (1 paper)Proceedings of the National Academy of Sciences (1 paper)
- Partner nations
- United StatesAustriaGermany
In The Last Decade
Jo Beth Harry
14 papers receiving 934 citations
Peers
Comparison fields: 5 of 71
- Epidemiology 500
- Oncology 377
- Virology 31
- Immunology 138
- Molecular Biology 442
Countries citing papers authored by Jo Beth Harry
This map shows the geographic impact of Jo Beth Harry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jo Beth Harry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jo Beth Harry more than expected).
Fields of papers citing papers by Jo Beth Harry
This network shows the impact of papers produced by Jo Beth Harry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jo Beth Harry. The network helps show where Jo Beth Harry may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Jo Beth Harry, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 11 | |
| 2 | 2008 | 23 | |
| 3 | 2008 | 29 | |
| 4 | 2005 | 48 | |
| 5 | 2005 | 40 | |
| 6 | 2004 | 26 | |
| 7 | 2004 | 29 | |
| 8 | 2004 | 46 | |
| 9 | 1997 | 403 | |
| 10 | 1996 | 107 | |
| 11 | 1996 | 22 | |
| 12 | 1994 | 113 | |
| 13 | 1992 | 48 | |
| 14 | 1991 | 25 |
About Jo Beth Harry
Jo Beth Harry is a scholar working on Cardiology and Cardiovascular Medicine, Virology, Cellular and Molecular Neuroscience, Epidemiology and Genetics, having authored 14 papers that have together received 970 indexed citations. Recurring topics across this work include Ion channel regulation and function (6 papers), Cardiac electrophysiology and arrhythmias (5 papers), Cervical Cancer and HPV Research (4 papers), Virus-based gene therapy research (3 papers), Cancer-related Molecular Pathways (3 papers), Antifungal resistance and susceptibility (2 papers), Nicotinic Acetylcholine Receptors Study (2 papers) and Neuroscience and Neural Engineering (2 papers). The work is most often cited by research in Epidemiology (500 citations), Oncology (377 citations), Virology (31 citations), Immunology (138 citations) and Molecular Biology (442 citations). Jo Beth Harry has collaborated with scholars based in United States, Austria and Germany. Frequent co-authors include Denise A. Galloway, Erik Espling, Jens Oliver Funk, Bruce Stillman, S Waga, F O Wettstein, Nikolai M. Soldatov, Evgeny Kobrinsky, Anthony P. Schmitt and Thomas Iftner. Their work appears in journals such as Journal of Virology, Journal of Biological Chemistry, Antimicrobial Agents and Chemotherapy, Genes & Development and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.