Erik Espling

711 total citations
7 papers, 553 citations indexed

About

Erik Espling is a scholar working on Oncology, Radiology, Nuclear Medicine and Imaging and Molecular Biology. According to data from OpenAlex, Erik Espling has authored 7 papers receiving a total of 553 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Oncology, 3 papers in Radiology, Nuclear Medicine and Imaging and 2 papers in Molecular Biology. Recurrent topics in Erik Espling's work include Monoclonal and Polyclonal Antibodies Research (3 papers), CAR-T cell therapy research (2 papers) and Cancer-related Molecular Pathways (2 papers). Erik Espling is often cited by papers focused on Monoclonal and Polyclonal Antibodies Research (3 papers), CAR-T cell therapy research (2 papers) and Cancer-related Molecular Pathways (2 papers). Erik Espling collaborates with scholars based in United States. Erik Espling's co-authors include Denise A. Galloway, Jo Beth Harry, Jens Oliver Funk, Bruce Stillman, S Waga, G W Demers, Beth G. Etscheid, Peter A. Thompson, Jeffrey A. Ledbetter and D. Barone and has published in prestigious journals such as Journal of Clinical Oncology, Genes & Development and Blood.

In The Last Decade

Erik Espling

6 papers receiving 540 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Erik Espling United States	5	319	284	245	101	86	7	553
Siribang-on Piboonniyom United States	7	287 0.9×	254 0.9×	320 1.3×	93 0.9×	55 0.6×	7	696
Nicole A. Spardy United States	10	227 0.7×	220 0.8×	300 1.2×	64 0.6×	27 0.3×	10	504
Jasmine I. Daksis Canada	6	243 0.8×	168 0.6×	338 1.4×	115 1.1×	61 0.7×	10	544
Suruchi Mittal Italy	7	242 0.8×	198 0.7×	331 1.4×	49 0.5×	116 1.3×	7	641
Xianming Yu United States	16	403 1.3×	398 1.4×	253 1.0×	62 0.6×	180 2.1×	20	793
Arie Zauberman Israel	9	295 0.9×	172 0.6×	314 1.3×	40 0.4×	47 0.5×	9	560
Qi‐Sheng Feng China	13	297 0.9×	96 0.3×	219 0.9×	47 0.5×	93 1.1×	31	553
Rémy Poirey Germany	13	289 0.9×	97 0.3×	172 0.7×	53 0.5×	103 1.2×	20	467
Stacy R. Hagemeier United States	11	395 1.2×	227 0.8×	171 0.7×	28 0.3×	120 1.4×	13	549
Tim Mosbruger United States	12	166 0.5×	100 0.4×	324 1.3×	76 0.8×	57 0.7×	14	575

Countries citing papers authored by Erik Espling

Since Specialization

Citations

This map shows the geographic impact of Erik Espling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Erik Espling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Erik Espling more than expected).

Fields of papers citing papers by Erik Espling

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Erik Espling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Erik Espling. The network helps show where Erik Espling may publish in the future.

Co-authorship network of co-authors of Erik Espling

This figure shows the co-authorship network connecting the top 25 collaborators of Erik Espling. A scholar is included among the top collaborators of Erik Espling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Erik Espling. Erik Espling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Gavin, Marc A., et al.. (2020). Abstract B45: Phosphatidylserine suppresses T cells through GPR174, and co-inhibition of adenosine receptors and GPR174 synergistically enhances T cell responses. Cancer Immunology Research. 8(3_Supplement). B45–B45.

2.

Hayden-Ledbetter, Martha, Erik Espling, William Brady, et al.. (2009). CD20-Directed Small Modular Immunopharmaceutical, TRU-015, Depletes Normal and Malignant B Cells. Clinical Cancer Research. 15(8). 2739–2746. 27 indexed citations

3.

Grosmaire, Laura S., Erik Espling, C. Nilsson, et al.. (2007). Depletion of Normal and Malignant B cells with a CD37-specific SMIP molecule. Journal of Clinical Oncology. 25(18_suppl). 3063–3063. 1 indexed citations

4.

Zhao, Xiaobin, Sabyasachi Biswas, Andrew P. Mone, et al.. (2004). Novel Anti-CD37 Small Modular Immunopharmaceutical (SMIP) Induces B-Cell-Specific, Caspase-Independent Apoptosis in Human CLL Cells.. Blood. 104(11). 2515–2515. 4 indexed citations

5.

Metz, Stewart A., Scott K. Holland, Lisa M. Johnson, et al.. (2001). Inosine-5′-Monophosphate Dehydrogenase Is Required for Mitogenic Competence of Transformed Pancreatic β Cells**This work was supported in part by NIH Grant DK-37312.. Endocrinology. 142(1). 193–204. 11 indexed citations

6.

Funk, Jens Oliver, S Waga, Jo Beth Harry, et al.. (1997). Inhibition of CDK activity and PCNA-dependent DNA replication by p21 is blocked by interaction with the HPV-16 E7 oncoprotein. Genes & Development. 11(16). 2090–2100. 403 indexed citations

7.

Demers, G W, Erik Espling, Jo Beth Harry, Beth G. Etscheid, & Denise A. Galloway. (1996). Abrogation of growth arrest signals by human papillomavirus type 16 E7 is mediated by sequences required for transformation. Journal of Virology. 70(10). 6862–6869. 107 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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