Jirô Arata
- Molecular Biology
- Oncology
- Cellular and Molecular Neuroscience top 10%
- Cancer Research
- Immunology and Allergy top 5%
- Co-authors
- Jôji TadaHisanori AkiyamaWataru FujimotoTakashi OonoHiroyuki KodamaYoshifumi NinomiyaHiroko KanzakiSatoshi Hirakawa
- Topics
- Peptidase Inhibition and Analysis (16 papers)Neuropeptides and Animal Physiology (10 papers)Protease and Inhibitor Mechanisms (8 papers)
In The Last Decade
Jirô Arata
31 papers receiving 615 citations
Peers
Comparison fields: 5 of 88
- Molecular Biology 255
- Oncology 228
- Cellular and Molecular Neuroscience 152
- Cancer Research 132
- Immunology and Allergy 105
Countries citing papers authored by Jirô Arata
This map shows the geographic impact of Jirô Arata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jirô Arata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jirô Arata more than expected).
Fields of papers citing papers by Jirô Arata
This network shows the impact of papers produced by Jirô Arata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jirô Arata. The network helps show where Jirô Arata may publish in the future.
Co-authorship network of co-authors of Jirô Arata
This figure shows the co-authorship network connecting the top 25 collaborators of Jirô Arata. A scholar is included among the top collaborators of Jirô Arata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jirô Arata. Jirô Arata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 59 | |
| 2 | 1 | |
| 3 | Expression of the bcl-2 homologue bax in normal human skin, psoriasis vulgaris and non-melanoma skin cancers. | 36 |
| 4 | 56 | |
| 5 | 27 | |
| 6 | 8 | |
| 7 | 63 | |
| 8 | 19 | |
| 9 | 16 | |
| 10 | 4 | |
| 11 | 3 | |
| 12 | Angiopathic pathogenesis of clinical manifestations in prolidase deficiency. | 15 |
| 13 | 20 | |
| 14 | 3 | |
| 15 | 16 | |
| 16 | Characterization of fibroblasts-derived prolidase-presence of two forms of prolidase | 1 |
| 17 | 38 | |
| 18 | 9 | |
| 19 | 4 | |
| 20 | 64 |
About Jirô Arata
Jirô Arata is a scholar working on Immunology and Allergy, Oncology and Cellular and Molecular Neuroscience, having authored 31 papers that have together received 651 indexed citations. Recurring topics across this work include Peptidase Inhibition and Analysis (16 papers), Neuropeptides and Animal Physiology (10 papers) and Protease and Inhibitor Mechanisms (8 papers). The work is most often cited by research in Immunology and Allergy (105 citations), Microbiology (9 citations) and Cancer Research (132 citations). Jirô Arata has collaborated with scholars based in Japan and Russia. Frequent co-authors include Jôji Tada, Hisanori Akiyama, Wataru Fujimoto, Takashi Oono, Hiroyuki Kodama, Yoshifumi Ninomiya, Hiroko Kanzaki, Satoshi Hirakawa, Osamu Yamasaki and Sumihare Noji. Their work appears in journals such as Biochemical and Biophysical Research Communications, FEBS Letters and Clinica Chimica Acta.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.