Jiří Kalabis

1.3k total citations
17 papers, 1.1k citations indexed

About

Jiří Kalabis is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Jiří Kalabis has authored 17 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 6 papers in Oncology and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Jiří Kalabis's work include Cancer Cells and Metastasis (3 papers), Cancer-related gene regulation (3 papers) and TGF-β signaling in diseases (2 papers). Jiří Kalabis is often cited by papers focused on Cancer Cells and Metastasis (3 papers), Cancer-related gene regulation (3 papers) and TGF-β signaling in diseases (2 papers). Jiří Kalabis collaborates with scholars based in United States, Japan and Czechia. Jiří Kalabis's co-authors include Meenhard Herlyn, Anil K. Rustgi, Hiroshi Nakagawa, Douglas B. Stairs, Maria E. Vega, Andres J. Klein–Szanto, Carmen Z. Michaylira, Gabrielle S. Wong, Mitsuteru Natsuizaka and Cameron N. Johnstone and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Genes & Development.

In The Last Decade

Jiří Kalabis

17 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jiří Kalabis United States 12 625 421 254 192 152 17 1.1k
Maria E. Vega United States 11 441 0.7× 285 0.7× 186 0.7× 113 0.6× 108 0.7× 18 820
Lidia Moserle Italy 18 647 1.0× 496 1.2× 158 0.6× 341 1.8× 173 1.1× 26 1.1k
Koroku Kato Japan 22 466 0.7× 433 1.0× 141 0.6× 250 1.3× 125 0.8× 51 1.1k
Cornelius F.J. Jansen Netherlands 13 779 1.2× 319 0.8× 233 0.9× 156 0.8× 79 0.5× 21 1.3k
Sudheer Kumar Gara United States 20 545 0.9× 283 0.7× 212 0.8× 260 1.4× 183 1.2× 35 1.2k
Meghna Waghray United States 15 663 1.1× 623 1.5× 229 0.9× 261 1.4× 192 1.3× 15 1.4k
Simona Podgrabinska United States 9 633 1.0× 957 2.3× 211 0.8× 111 0.6× 256 1.7× 11 1.3k
Joseph L. Sottnik United States 17 342 0.5× 341 0.8× 150 0.6× 163 0.8× 105 0.7× 26 837
Eva Allonca Spain 23 570 0.9× 410 1.0× 144 0.6× 269 1.4× 114 0.8× 46 1.1k
Christine Gjerdrum Norway 3 514 0.8× 488 1.2× 100 0.4× 158 0.8× 385 2.5× 6 1.1k

Countries citing papers authored by Jiří Kalabis

Since Specialization
Citations

This map shows the geographic impact of Jiří Kalabis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jiří Kalabis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jiří Kalabis more than expected).

Fields of papers citing papers by Jiří Kalabis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jiří Kalabis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jiří Kalabis. The network helps show where Jiří Kalabis may publish in the future.

Co-authorship network of co-authors of Jiří Kalabis

This figure shows the co-authorship network connecting the top 25 collaborators of Jiří Kalabis. A scholar is included among the top collaborators of Jiří Kalabis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jiří Kalabis. Jiří Kalabis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Kalabis, Jiří, Gabrielle S. Wong, Maria E. Vega, et al.. (2012). Isolation and characterization of mouse and human esophageal epithelial cells in 3D organotypic culture. Nature Protocols. 7(2). 235–246. 130 indexed citations
2.
Mongroo, Perry S., Felicite K. Noubissi, Míriam Cuatrecasas, et al.. (2011). IMP-1 Displays Cross-Talk with K-Ras and Modulates Colon Cancer Cell Survival through the Novel Proapoptotic Protein CYFIP2. Cancer Research. 71(6). 2172–2182. 98 indexed citations
3.
Stairs, Douglas B., Lauren J. Bayne, Ben Rhoades, et al.. (2011). Deletion of p120-Catenin Results in a Tumor Microenvironment with Inflammation and Cancer that Establishes It as a Tumor Suppressor Gene. Cancer Cell. 19(4). 470–483. 158 indexed citations
4.
Ohashi, Shinya, Mitsuteru Natsuizaka, Gabrielle S. Wong, et al.. (2010). Epidermal Growth Factor Receptor and Mutant p53 Expand an Esophageal Cellular Subpopulation Capable of Epithelial-to-Mesenchymal Transition through ZEB Transcription Factors. Cancer Research. 70(10). 4174–4184. 126 indexed citations
5.
6.
Kalabis, Jiří, Gang Li, Mizuho Fukunaga‐Kalabis, Anil K. Rustgi, & Meenhard Herlyn. (2008). Endothelin-3 stimulates survival of goblet cells in organotypic cultures of fetal human colonic epithelium. American Journal of Physiology-Gastrointestinal and Liver Physiology. 295(6). G1182–G1189. 7 indexed citations
7.
Kalabis, Jiří, Kenji Oyama, Takaomi Okawa, et al.. (2008). A subpopulation of mouse esophageal basal cells has properties of stem cells with the capacity for self-renewal and lineage specification. Journal of Clinical Investigation. 118(12). 3860–9. 105 indexed citations
8.
Lee, James, Munenori Takaoka, Carmen Z. Michaylira, et al.. (2008). Hypoxia inducible factor-1α regulates cyclooxgenase-2 and prostaglandin E synthase in esophageal cancer.. 68. 2049–2049. 1 indexed citations
9.
10.
Fukunaga‐Kalabis, Mizuho, Gabriela Martínez, Zhaojun Liu, et al.. (2006). CCN3 controls 3D spatial localization of melanocytes in the human skin through DDR1. The Journal of Cell Biology. 175(4). 563–569. 83 indexed citations
11.
Berencsi, Klára, Neal J. Meropol, John P. Hoffman, et al.. (2006). Colon carcinoma cells induce CXCL11-dependent migration of CXCR3-expressing cytotoxic T lymphocytes in organotypic culture. Cancer Immunology Immunotherapy. 56(3). 359–370. 21 indexed citations
12.
Kalabis, Jiří, Ian M. Rosenberg, & Daniel K. Podolsky. (2006). Vangl1 Protein Acts as a Downstream Effector of Intestinal Trefoil Factor (ITF)/TFF3 Signaling and Regulates Wound Healing of Intestinal Epithelium. Journal of Biological Chemistry. 281(10). 6434–6441. 44 indexed citations
13.
Li, Gang, Jiří Kalabis, Xiaowei Xu, et al.. (2003). Reciprocal regulation of MelCAM and AKT in human melanoma. Oncogene. 22(44). 6891–6899. 69 indexed citations
14.
Kalabis, Jiří, Greg H. Enders, Brigitte Marian, et al.. (2003). Stimulation of human colonic epithelial cells by leukemia inhibitory factor is dependent on collagen‐embedded fibroblasts in organotypic culture. The FASEB Journal. 17(9). 1115–1117. 30 indexed citations
15.
Satyamoorthy, Kapaettu, Gang Li, Bhavesh Vaidya, Jiří Kalabis, & Meenhard Herlyn. (2002). Insulin-like growth factor-I-induced migration of melanoma cells is mediated by interleukin-8 induction.. PubMed. 13(2). 87–93. 44 indexed citations
16.
Kalabis, Jiří, et al.. (2000). Correlation between apoptotic figure counting and the TUNEL technique.. PubMed. 22(4). 307–10. 3 indexed citations
17.
Žaloudík, Jan, et al.. (1997). Biomarkers for predicting response to regional chemoimmunotherapy in liver metastases from colorectal carcinoma. International Journal of Immunopharmacology. 19(9-10). 481–485. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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