Jingyi Shi

2.3k citations
32 papers · 1.1k indexed · h-index 12
Topics
Acute Myeloid Leukemia Research (7 papers)Chronic Myeloid Leukemia Treatments (5 papers)Gastric Cancer Management and Outcomes (3 papers)

In The Last Decade

Jingyi Shi

28 papers receiving 1.1k citations

Peers

Jingyi Shi
Comparison fields: 5 of 97
  • Molecular Biology 536
  • Hematology 468
  • Genetics 227
  • Oncology 183
  • Cancer Research 170
Replace Omar Benzakour with:
Omar Benzakour United Kingdom
Nicolas Prévost United States
Ming-jiang Xu Japan
Majid Ali United States
Marina R. Carpinelli Australia
Amy S. Gardiner United States
Maria Mesuraca Italy
Christopher R. Marlein United Kingdom
Karen Seeberger Canada
Hendrik Nogai Germany
Jingyi Shi relative to Omar Benzakour United Kingdom Omar Benzakour's profile →
Citations per field
00.5×6.2×
Omar Benzakour · 1×
Citations per year

Countries citing papers authored by Jingyi Shi

Since Specialization
Citations

This map shows the geographic impact of Jingyi Shi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jingyi Shi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jingyi Shi more than expected).

Fields of papers citing papers by Jingyi Shi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jingyi Shi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jingyi Shi. The network helps show where Jingyi Shi may publish in the future.

Co-authorship network of co-authors of Jingyi Shi

This figure shows the co-authorship network connecting the top 25 collaborators of Jingyi Shi. A scholar is included among the top collaborators of Jingyi Shi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jingyi Shi. Jingyi Shi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 0
3 1
4 1
5 7
6 6
7 3
8 1
9 2
10 7
11 30
12 0
13 1
14 1
15 31
16 87
17 30
18 129
19
[Study of mutation and single nucleotide polymorphism of PDGFRbeta and SHIP gene in acute myeloid leukemia].
1
20 57

About Jingyi Shi

Jingyi Shi is a scholar working on Hematology, Pathology and Forensic Medicine and Oncology, having authored 32 papers that have together received 1.1k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (7 papers), Chronic Myeloid Leukemia Treatments (5 papers) and Gastric Cancer Management and Outcomes (3 papers). The work is most often cited by research in Hematology (468 citations), Developmental Neuroscience (143 citations) and Genetics (227 citations). Jingyi Shi has collaborated with scholars based in China, United States and Ethiopia. Frequent co-authors include Adrian Marinovich, Ben A. Barres, Sai‐Juan Chen, Bing Chen, Chen Zhu, Yueying Wang, An Huang, Yongmei Zhu, Jin Gu and Zhixiang Shen. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Communications and Journal of Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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