Jesse R. Raab

1.9k total citations
26 papers, 1.3k citations indexed

About

Jesse R. Raab is a scholar working on Molecular Biology, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, Jesse R. Raab has authored 26 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 8 papers in Immunology and 6 papers in Pathology and Forensic Medicine. Recurrent topics in Jesse R. Raab's work include Genomics and Chromatin Dynamics (9 papers), Chromatin Remodeling and Cancer (8 papers) and Immunotherapy and Immune Responses (7 papers). Jesse R. Raab is often cited by papers focused on Genomics and Chromatin Dynamics (9 papers), Chromatin Remodeling and Cancer (8 papers) and Immunotherapy and Immune Responses (7 papers). Jesse R. Raab collaborates with scholars based in United States, Canada and India. Jesse R. Raab's co-authors include Rohinton T. Kamakaka, Terry Magnuson, T. Peter Kingham, Ronald P. DeMatteo, Umer I. Chaudhry, George Plitas, Steven C. Katz, Joshua Starmer, Jonathan C. Schisler and Ronald L. Chandler and has published in prestigious journals such as Nature Communications, Nature Genetics and The EMBO Journal.

In The Last Decade

Jesse R. Raab

25 papers receiving 1.2k citations

Peers

Countries citing papers authored by Jesse R. Raab

Since Specialization

Citations

This map shows the geographic impact of Jesse R. Raab's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jesse R. Raab with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jesse R. Raab more than expected).

Fields of papers citing papers by Jesse R. Raab

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jesse R. Raab. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jesse R. Raab. The network helps show where Jesse R. Raab may publish in the future.

Co-authorship network of co-authors of Jesse R. Raab

This figure shows the co-authorship network connecting the top 25 collaborators of Jesse R. Raab. A scholar is included among the top collaborators of Jesse R. Raab based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jesse R. Raab. Jesse R. Raab is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Expanded detection and impact of BAP1 alterations in cancer 2024 ·NAR Cancer ·Ian R. Sturgill, Jesse R. Raab, Katherine A. Hoadley	0
2	Sox9EGFP Defines Biliary Epithelial Heterogeneity Downstream of Yap Activity 2021 ·Cellular and Molecular Gastroenterology and Hepatology ·(unknown), (unknown), (unknown), (unknown), Karel P. Alcedo, Jesse R. Raab, Adam D. Gracz	10
3	Quantitative classification of chromatin dynamics reveals regulators of intestinal stem cell differentiation 2019 ·Development ·Jesse R. Raab, (unknown), (unknown), (unknown), Scott T. Magness, Adam D. Gracz	23
4	Sox4 Promotes Atoh1-Independent Intestinal Secretory Differentiation Toward Tuft and Enteroendocrine Fates 2018 ·Gastroenterology ·Adam D. Gracz, Leigh Ann Samsa, (unknown), (unknown), Bailey Zwarycz, Yuan-Hung Lo, Katherine Bao, Joshua Starmer, Jesse R. Raab, Noah F. Shroyer, R. Lee Reinhardt, Scott T. Magness	66
5	SWI/SNF remains localized to chromatin in the presence of SCHLAP1 2018 ·Nature Genetics ·Jesse R. Raab, Keriayn N. Smith, (unknown), (unknown), J. Mauro Calabrese, Terry Magnuson	23
6	Co-regulation of transcription by BRG1 and BRM, two mutually exclusive SWI/SNF ATPase subunits 2017 ·Epigenetics & Chromatin ·Jesse R. Raab, (unknown), (unknown), Terry Magnuson	38
7	Epigenetic Regulation by ATP-Dependent Chromatin-Remodeling Enzymes 2016 ·Current topics in developmental biology ·(unknown), Jesse R. Raab, Terry Magnuson	10
8	Genome-Wide Transcriptional Regulation Mediated by Biochemically Distinct SWI/SNF Complexes 2015 ·PLoS Genetics ·Jesse R. Raab, (unknown), Terry Magnuson	92
9	An Interactive Database for the Assessment of Histone Antibody Specificity 2015 ·Molecular Cell ·Scott B. Rothbart, Bradley M. Dickson, Jesse R. Raab, Adrian T. Grzybowski, Krzysztof Krajewski, Angela H. Guo, Erin K. Shanle, Steven Z. Josefowicz, Stephen M. Fuchs, (unknown), Terry Magnuson, Alexander J. Ruthenburg, Brian D. Strahl	121
10	Coexistent ARID1A–PIK3CA mutations promote ovarian clear-cell tumorigenesis through pro-tumorigenic inflammatory cytokine signalling 2015 ·Nature Communications ·Ronald L. Chandler, Jeffrey S. Damrauer, Jesse R. Raab, Jonathan C. Schisler, Matthew D. Wilkerson, John P. Didion, Joshua Starmer, Daniel Serber, Della Yee, Jessie Xiong, David B. Darr, Fernando Pardo‐Manuel de Villena, William Y. Kim, Terry Magnuson	230
11	Global gene expression profiling of a mouse model of ovarian clear cell carcinoma caused by ARID1A and PIK3CA mutations implicates a role for inflammatory cytokine signaling 2015 ·Genomics Data ·Ronald L. Chandler, Jesse R. Raab, (unknown), Terry Magnuson, Jonathan C. Schisler	5
12	TFIIIC bound DNA elements in nuclear organization and insulation 2012 ·Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms ·Jacob G. Kirkland, Jesse R. Raab, Rohinton T. Kamakaka	41
13	Human tRNA genes function as chromatin insulators 2011 ·The EMBO Journal ·Jesse R. Raab, Jonathan Chiu, Jingchun Zhu, Sol Katzman, Sreenivasulu Kurukuti, Paul A. Wade, David Haussler, Rohinton T. Kamakaka	109
14	Insulators and promoters: closer than we think 2010 ·Nature Reviews Genetics ·Jesse R. Raab, Rohinton T. Kamakaka	120
15	DNA polymerase ε, acetylases and remodellers cooperate to form a specialized chromatin structure at a tRNA insulator 2009 ·The EMBO Journal ·Namrita Dhillon, Jesse R. Raab, (unknown), Shawn J. Szyjka, (unknown), Oscar M. Aparicio, Brenda Andrews, Rohinton T. Kamakaka	39
16	NK Dendritic Cells Expanded in IL-15 Exhibit Antitumor Responses In Vivo 2007 ·The Journal of Immunology ·Umer I. Chaudhry, George Plitas, Bryan M. Burt, T. Peter Kingham, Jesse R. Raab, Ronald P. DeMatteo	21
17	Biliary Obstruction Selectively Expands and Activates Liver Myeloid Dendritic Cells 2006 ·The Journal of Immunology ·Joshua I.S. Bleier, Steven C. Katz, Umer I. Chaudhry, Venu G. Pillarisetty, T. Peter Kingham, Alaap B. Shah, Jesse R. Raab, Ronald P. DeMatteo	30
18	Interleukin-18 and the toll-like receptor 9 ligand CpG stimulate murine splenic natural killer dendritic cells (NKDC) to proliferate and secrete IFN-gamma 2006 ·Journal of the American College of Surgeons ·Umer I. Chaudhry, T. Peter Kingham, George Plitas, Steven C. Katz, (unknown), Jesse R. Raab, Ronald P. DeMatteo	1
19	Combined Stimulation with Interleukin-18 and CpG Induces Murine Natural Killer Dendritic Cells to Produce IFN-γ and Inhibit Tumor Growth 2006 ·Cancer Research ·Umer I. Chaudhry, T. Peter Kingham, George Plitas, Steven C. Katz, Jesse R. Raab, Ronald P. DeMatteo	57
20	Crm1-Mediated Nuclear Export of Cdc14 is Required for the Completion of Cytokinesis in Budding Yeast 2005 ·Cell Cycle ·Joshua N. Bembenek, Jungseog Kang, Cornelia Kurischko, Bing Li, Jesse R. Raab, Kenneth D. Belanger, Francis C. Luca, Hongtao Yu	42

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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