Jennifer Haas

800 total citations
8 papers, 630 citations indexed

About

Jennifer Haas is a scholar working on Physiology, Immunology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Jennifer Haas has authored 8 papers receiving a total of 630 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Physiology, 5 papers in Immunology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Jennifer Haas's work include Asthma and respiratory diseases (5 papers), Pediatric health and respiratory diseases (3 papers) and IL-33, ST2, and ILC Pathways (3 papers). Jennifer Haas is often cited by papers focused on Asthma and respiratory diseases (5 papers), Pediatric health and respiratory diseases (3 papers) and IL-33, ST2, and ILC Pathways (3 papers). Jennifer Haas collaborates with scholars based in United Kingdom, United States and Japan. Jennifer Haas's co-authors include Michael R. Edwards, Sebastian L. Johnston, Onn Min Kon, Annemarie Sykes, Simon Message, Nathan W. Bartlett, Jie Zhu, Peter K. Jeffery, Deborah Clarke and Louise Slater and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and European Respiratory Journal.

In The Last Decade

Jennifer Haas

8 papers receiving 625 citations

Peers

Countries citing papers authored by Jennifer Haas

Since Specialization

Citations

This map shows the geographic impact of Jennifer Haas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jennifer Haas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jennifer Haas more than expected).

Fields of papers citing papers by Jennifer Haas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jennifer Haas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jennifer Haas. The network helps show where Jennifer Haas may publish in the future.

Co-authorship network of co-authors of Jennifer Haas

This figure shows the co-authorship network connecting the top 25 collaborators of Jennifer Haas. A scholar is included among the top collaborators of Jennifer Haas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jennifer Haas. Jennifer Haas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	CXC chemokines and antimicrobial peptides in rhinovirus‐induced experimental asthma exacerbations 2014 ·Clinical & Experimental Allergy ·Gernot Rohde, Simon Message, Jennifer Haas, Tatiana Kebadze, Hayley L. Parker, Vasile Laza‐Stanca, Musa Khaitov, Onn Min Kon, L Stanciu, Patrick Mallia, Michael R. Edwards, Sebastian L. Johnston	41
2	Rhinovirus-induced interferon production is not deficient in well controlled asthma 2013 ·Thorax ·Annemarie Sykes, Jonathan Macintyre, Michael R. Edwards, Ajerico del Rosario, Jennifer Haas, Vera Gielen, Onn Min Kon, Mark McHale, Sebastian L. Johnston	97
3	TLR3, TLR4 and TLRs7–9 Induced Interferons Are Not Impaired in Airway and Blood Cells in Well Controlled Asthma 2013 ·PLoS ONE ·Annemarie Sykes, Michael R. Edwards, Jonathan Macintyre, Ajerico del Rosario, Vera Gielen, Jennifer Haas, Onn Min Kon, Mark McHale, Sebastian L. Johnston	33
4	Correction: Co-ordinated Role of TLR3, RIG-I and MDA5 in the Innate Response to Rhinovirus in Bronchial Epithelium 2012 ·PLoS Pathogens ·(unknown), Nathan W. Bartlett, Jennifer Haas, Jie Zhu, Simon Message, Ross P. Walton, Annemarie Sykes, (unknown), Deborah Clarke, Maria G. Belvisi, Onn Min Kon, Takashi Fujita, Peter K. Jeffery, Sebastian L. Johnston, Michael R. Edwards	2
5	Defining critical roles for NF‐κB p65 and type I interferon in innate immunity to rhinovirus 2012 ·EMBO Molecular Medicine ·Nathan W. Bartlett, Louise Slater, Nicholas Glanville, Jennifer Haas, Gaetano Caramori, Paolo Casolari, Deborah Clarke, Simon Message, Julia Aniscenko, Tatiana Kebadze, Jie Zhu, Patrick Mallia, Joseph P. Mizgerd, Maria G. Belvisi, Alberto Papi, Sergei V. Kotenko, Sebastian L. Johnston, Michael R. Edwards	64
6	Rhinovirus induces MUC5AC in a human infection model andin vitroviaNF-κB and EGFR pathways 2010 ·European Respiratory Journal ·Christopher A. Hewson, Jennifer Haas, Nathan W. Bartlett, Simon Message, Vasile Laza‐Stanca, Tatiana Kebadze, Gaetano Caramori, Jie Zhu, Mark R. Edbrooke, L Stanciu, Onn Min Kon, Alberto Papi, Peter K. Jeffery, Michael R. Edwards, Sebastian L. Johnston	90
7	Co-ordinated Role of TLR3, RIG-I and MDA5 in the Innate Response to Rhinovirus in Bronchial Epithelium 2010 ·PLoS Pathogens ·Louise Slater, Nathan W. Bartlett, Jennifer Haas, Jie Zhu, Simon Message, Ross P. Walton, Annemarie Sykes, (unknown), Deborah Clarke, Maria G. Belvisi, Onn Min Kon, Takashi Fujita, Peter K. Jeffery, Sebastian L. Johnston, Michael R. Edwards	245
8	Corticosteroids and β2 Agonists Differentially Regulate Rhinovirus-induced Interleukin-6 via Distinct Cis-acting Elements 2007 ·Journal of Biological Chemistry ·Michael R. Edwards, Jennifer Haas, (unknown), Malcolm Johnson, Sebastian L. Johnston	58

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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