Jean‐Pierre Pujol

2.3k total citations
36 papers, 2.0k citations indexed

About

Jean‐Pierre Pujol is a scholar working on Rheumatology, Immunology and Allergy and Molecular Biology. According to data from OpenAlex, Jean‐Pierre Pujol has authored 36 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Rheumatology, 14 papers in Immunology and Allergy and 12 papers in Molecular Biology. Recurrent topics in Jean‐Pierre Pujol's work include Osteoarthritis Treatment and Mechanisms (22 papers), Cell Adhesion Molecules Research (14 papers) and Proteoglycans and glycosaminoglycans research (11 papers). Jean‐Pierre Pujol is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (22 papers), Cell Adhesion Molecules Research (14 papers) and Proteoglycans and glycosaminoglycans research (11 papers). Jean‐Pierre Pujol collaborates with scholars based in France, United States and Finland. Jean‐Pierre Pujol's co-authors include Philippe Galéra, Patrick Bogdanowicz, Florence Legendre, Karim Boumédiene, Christos Chadjichristos, Gérard Loyau, Rina Andriamanalijaona, Chafik Ghayor, Françoise Rédiní and Leena Ala‐Kokko and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical and Biophysical Research Communications and FEBS Letters.

In The Last Decade

Jean‐Pierre Pujol

36 papers receiving 2.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jean‐Pierre Pujol France	23	950	683	334	330	309	36	2.0k
Karim Boumédiene France	31	1.2k 1.2×	885 1.3×	290 0.9×	404 1.2×	347 1.1×	83	2.5k
Kosei Ijiri Japan	23	1.3k 1.4×	1.0k 1.5×	257 0.8×	477 1.4×	434 1.4×	70	3.0k
Reinout Stoop Netherlands	30	1.1k 1.2×	601 0.9×	290 0.9×	202 0.6×	225 0.7×	53	2.7k
Diane Peluso Canada	6	1.7k 1.8×	547 0.8×	171 0.5×	421 1.3×	440 1.4×	8	2.3k
Glyn D. Palmer United States	23	958 1.0×	656 1.0×	138 0.4×	194 0.6×	255 0.8×	43	2.1k
Chiara Gentili Italy	29	824 0.9×	877 1.3×	189 0.6×	228 0.7×	119 0.4×	53	2.2k
Henk M. van Beuningen Netherlands	25	2.2k 2.4×	1.1k 1.6×	258 0.8×	262 0.8×	625 2.0×	48	3.0k
Florence Legendre France	23	887 0.9×	469 0.7×	128 0.4×	246 0.7×	216 0.7×	46	1.6k
Kenneth B. Marcu United States	18	1.2k 1.3×	1.2k 1.7×	120 0.4×	554 1.7×	477 1.5×	23	2.4k
Pia Margarethe Gebhard Germany	15	1.6k 1.7×	619 0.9×	146 0.4×	526 1.6×	462 1.5×	20	2.0k

Countries citing papers authored by Jean‐Pierre Pujol

Since Specialization

Citations

This map shows the geographic impact of Jean‐Pierre Pujol's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jean‐Pierre Pujol with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jean‐Pierre Pujol more than expected).

Fields of papers citing papers by Jean‐Pierre Pujol

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jean‐Pierre Pujol. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jean‐Pierre Pujol. The network helps show where Jean‐Pierre Pujol may publish in the future.

Co-authorship network of co-authors of Jean‐Pierre Pujol

This figure shows the co-authorship network connecting the top 25 collaborators of Jean‐Pierre Pujol. A scholar is included among the top collaborators of Jean‐Pierre Pujol based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jean‐Pierre Pujol. Jean‐Pierre Pujol is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Deschrevel, Brigitte, et al.. (2009). Chondroitin sulfate increases hyaluronan production by human synoviocytes through differential regulation of hyaluronan synthases: Role of p38 and Akt. Arthritis & Rheumatism. 60(3). 760–770. 64 indexed citations

Rochette, Jacques, et al.. (2009). Human insulin A‐chain peptide analog(s) with in vitro biological activity. Cell Biochemistry and Function. 27(6). 370–377. 1 indexed citations

Pujol, Jean‐Pierre, Christos Chadjichristos, Florence Legendre, et al.. (2008). Interleukin-1 and Transforming Growth Factor-ß 1 as Crucial Factors in Osteoarthritic Cartilage Metabolism. Connective Tissue Research. 49(3-4). 293–297. 114 indexed citations

Renard, Emmanuelle, Christos Chadjichristos, Magdalini Kypriotou, et al.. (2008). Chondroitin sulphate decreases collagen synthesis in normal and scleroderma fibroblasts through a Smad‐independent TGF‐β pathway – implication of C‐Krox and Sp1. Journal of Cellular and Molecular Medicine. 12(6b). 2836–2847. 6 indexed citations

Kypriotou, Magdalini, Gallic Beauchef, Christos Chadjichristos, et al.. (2007). Human Collagen Krox Up-regulates Type I Collagen Expression in Normal and Scleroderma Fibroblasts through Interaction with Sp1 and Sp3 Transcription Factors. Journal of Biological Chemistry. 282(44). 32000–32014. 40 indexed citations

Kypriotou, Magdalini, Christos Chadjichristos, Gallic Beauchef, et al.. (2007). Interleukin-6 (IL-6) and/or Soluble IL-6 Receptor Down-regulation of Human Type II Collagen Gene Expression in Articular Chondrocytes Requires a Decrease of Sp1·Sp3 Ratio and of the Binding Activity of Both Factors to the COL2A1 Promoter. Journal of Biological Chemistry. 283(8). 4850–4865. 128 indexed citations

Potier, Esther, Elisabeth Ferreira, Rina Andriamanalijaona, et al.. (2007). Hypoxia affects mesenchymal stromal cell osteogenic differentiation and angiogenic factor expression. Bone. 40(4). 1078–1087. 236 indexed citations

Beauchef, Gallic, Magdalini Kypriotou, Christos Chadjichristos, et al.. (2005). c-Krox down-regulates the expression of UDP–glucose dehydrogenase in chondrocytes. Biochemical and Biophysical Research Communications. 333(4). 1123–1131. 15 indexed citations

Kypriotou, Magdalini, Magali Demoor, Christos Chadjichristos, et al.. (2003). SOX9 Exerts a Bifunctional Effect on Type II Collagen Gene (COL2A1) Expression in Chondrocytes Depending on the Differentiation State. DNA and Cell Biology. 22(2). 119–129. 74 indexed citations

10.

Legendre, Florence, Jayesh Dudhia, Jean‐Pierre Pujol, & Patrick Bogdanowicz. (2003). JAK/STAT but Not ERK1/ERK2 Pathway Mediates Interleukin (IL)-6/Soluble IL-6R Down-regulation of Type II Collagen, Aggrecan Core, and Link Protein Transcription in Articular Chondrocytes. Journal of Biological Chemistry. 278(5). 2903–2912. 156 indexed citations

11.

Martin, Grégoire, et al.. (2003). Rhein Inhibits Interleukin-1β-Induced Activation of MEK/ERK Pathway and DNA Binding of NF-κB and AP-1 in Chondrocytes Cultured in Hypoxia: A Potential Mechanism for Its Disease-Modifying Effect in Osteoarthritis. Inflammation. 27(4). 233–246. 74 indexed citations

12.

Dupradeau, François‐Yves, et al.. (2002). Synthesis, and functional properties of a modified human insulin A-Chain: implication in a ‘Mini-Insulin’ structure determination. Bioorganic & Medicinal Chemistry. 10(7). 2111–2117. 5 indexed citations

13.

Ghayor, Chafik, Christos Chadjichristos, Leena Ala‐Kokko, et al.. (2001). SP3 Represses the SP1-mediated Transactivation of the HumanCOL2A1 Gene in Primary and De-differentiated Chondrocytes. Journal of Biological Chemistry. 276(40). 36881–36895. 80 indexed citations

14.

Galéra, Philippe, et al.. (2001). A Composite Element Binding the Vitamin D Receptor and the Retinoic X Receptor α Mediates the Transforming Growth Factor-β Inhibition of Decorin Gene Expression in Articular Chondrocytes. Journal of Biological Chemistry. 276(40). 36983–36992. 25 indexed citations

15.

Rédiní, Françoise, et al.. (1998). Expression of decorin and biglycan by rabbit articular chondrocytes.. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1398(2). 179–191. 43 indexed citations

16.

Rédiní, Françoise, et al.. (1997). Differential expression of membrane-anchored proteoglycans in rabbit articular chondrocytes cultured in monolayers and in alginate beads. Effect of transforming growth factor-β1. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1355(1). 20–32. 13 indexed citations

17.

Bogdanowicz, Patrick, et al.. (1996). An inositolphosphate glycan released by TGF-β mimics the proliferative but not the transcriptional effects of the factor and requires functional receptors. Cellular Signalling. 8(7). 503–509. 5 indexed citations

18.

Vivien, Denis, E. Petitfrere, Laurent Martiny, et al.. (1993). IPG (inositolphosphate glycan) as a cellular signal for TGF‐β1 modulation of chondrocyte cell cycle. Journal of Cellular Physiology. 155(3). 437–444. 23 indexed citations

19.

Vivien, Denis, et al.. (1992). TGF‐β–induced G2/M delay in proliferating rabbit articular chondrocytes is associated with an enhancement of replication rate and a cAMP decrease: Possible involvement of pertussis toxin‐sensitive pathway. Journal of Cellular Physiology. 150(2). 291–298. 18 indexed citations

20.

Rédiní, Françoise, et al.. (1991). Characterization of proteoglycans synthesized by rabbit articular chondrocytes in response to transforming growth factor-β (TGF-β). Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1093(2-3). 196–206. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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