Magali Demoor

1.5k total citations
35 papers, 1.2k citations indexed

About

Magali Demoor is a scholar working on Rheumatology, Genetics and Urology. According to data from OpenAlex, Magali Demoor has authored 35 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Rheumatology, 13 papers in Genetics and 13 papers in Urology. Recurrent topics in Magali Demoor's work include Osteoarthritis Treatment and Mechanisms (29 papers), Mesenchymal stem cell research (13 papers) and Periodontal Regeneration and Treatments (13 papers). Magali Demoor is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (29 papers), Mesenchymal stem cell research (13 papers) and Periodontal Regeneration and Treatments (13 papers). Magali Demoor collaborates with scholars based in France, United States and Canada. Magali Demoor's co-authors include Philippe Galéra, Florence Legendre, Karim Boumédiene, Magalie Hervieu, Fabrice Audigié, David Ollitrault, Frédéric Mallein‐Gérin, Tangni Gómez-Leduc, Jean‐Marie Denoix and Mouloud Bouyoucef and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Scientific Reports.

In The Last Decade

Magali Demoor

35 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Magali Demoor France	22	739	297	292	249	244	35	1.2k
Roberto Narcisi Netherlands	18	551 0.7×	332 1.1×	378 1.3×	249 1.0×	211 0.9×	47	1.0k
Michael B. Mueller Germany	13	673 0.9×	264 0.9×	330 1.1×	372 1.5×	292 1.2×	15	1.1k
Andy Cremers Netherlands	16	658 0.9×	359 1.2×	140 0.5×	265 1.1×	149 0.6×	43	1.1k
Simon R. Tew United Kingdom	23	1.1k 1.5×	475 1.6×	393 1.3×	509 2.0×	301 1.2×	48	1.7k
Anthony M. DeLise United States	8	595 0.8×	410 1.4×	167 0.6×	163 0.7×	136 0.6×	17	1.1k
Wendy Koevoet Netherlands	16	667 0.9×	218 0.7×	336 1.2×	433 1.7×	318 1.3×	23	1.1k
Carlotta Perucca Orfei Italy	22	384 0.5×	540 1.8×	365 1.3×	364 1.5×	152 0.6×	59	1.3k
Haiou Pan Japan	18	428 0.6×	616 2.1×	548 1.9×	380 1.5×	267 1.1×	22	1.6k
Yiying Qi China	20	258 0.3×	304 1.0×	216 0.7×	305 1.2×	159 0.7×	42	1.0k
Ling Wu China	23	522 0.7×	490 1.6×	678 2.3×	396 1.6×	294 1.2×	35	1.4k

Countries citing papers authored by Magali Demoor

Since Specialization

Citations

This map shows the geographic impact of Magali Demoor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Magali Demoor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Magali Demoor more than expected).

Fields of papers citing papers by Magali Demoor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Magali Demoor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Magali Demoor. The network helps show where Magali Demoor may publish in the future.

Co-authorship network of co-authors of Magali Demoor

This figure shows the co-authorship network connecting the top 25 collaborators of Magali Demoor. A scholar is included among the top collaborators of Magali Demoor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Magali Demoor. Magali Demoor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Jacquet, Sandrine, Frédéric Cassé, Florence Legendre, et al.. (2024). Double-blinded, randomized tolerance study of a biologically enhanced Nanogel with endothelin-1 and bradykinin receptor antagonist peptides via intra-articular injection for osteoarthritis treatment in horses. BMC Veterinary Research. 20(1). 547–547. 1 indexed citations

Audigié, Fabrice, et al.. (2023). New trends for osteoarthritis: Biomaterials, models and modeling. Drug Discovery Today. 28(3). 103488–103488. 27 indexed citations

Velot, Émilie, Elizabeth R. Balmayor, Lélia Bertoni, et al.. (2023). Women’s contribution to stem cell research for osteoarthritis: an opinion paper. Frontiers in Cell and Developmental Biology. 11. 1209047–1209047. 2 indexed citations

Cassé, Frédéric, Florence Legendre, Pierré Sirois, et al.. (2022). Functionalized Nanogels with Endothelin-1 and Bradykinin Receptor Antagonist Peptides Decrease Inflammatory and Cartilage Degradation Markers of Osteoarthritis in a Horse Organoid Model of Cartilage. International Journal of Molecular Sciences. 23(16). 8949–8949. 14 indexed citations

Benmoussa, Abderrahim, Émilie Brotin, J. Lafont, et al.. (2021). Tumor Suppressive Role of miR-342-5p in Human Chondrosarcoma Cells and 3D Organoids. International Journal of Molecular Sciences. 22(11). 5590–5590. 18 indexed citations

Bertoni, Lélia, Florence Legendre, Jean‐Marie Denoix, et al.. (2020). An experimentally induced osteoarthritis model in horses performed on both metacarpophalangeal and metatarsophalangeal joints: Technical, clinical, imaging, biochemical, macroscopic and microscopic characterization. PLoS ONE. 15(6). e0235251–e0235251. 15 indexed citations

Demoor, Magali, et al.. (2019). Comparison of the Chondrogenic Potential of Mesenchymal Stem Cells Derived from Bone Marrow and Umbilical Cord Blood Intended for Cartilage Tissue Engineering. Stem Cell Reviews and Reports. 16(1). 126–143. 35 indexed citations

Demoor, Magali, et al.. (2018). Differences in the intrinsic chondrogenic potential of equine umbilical cord matrix and cord blood mesenchymal stromal/stem cells for cartilage regeneration. Scientific Reports. 8(1). 13799–13799. 22 indexed citations

Legendre, Florence, David Ollitrault, Tangni Gómez-Leduc, et al.. (2017). Enhanced chondrogenesis of bone marrow-derived stem cells by using a combinatory cell therapy strategy with BMP-2/TGF-β1, hypoxia, and COL1A1/HtrA1 siRNAs. Scientific Reports. 7(1). 3406–3406. 38 indexed citations

10.

Gómez-Leduc, Tangni, Magalie Hervieu, Florence Legendre, et al.. (2016). Chondrogenic commitment of human umbilical cord blood-derived mesenchymal stem cells in collagen matrices for cartilage engineering. Scientific Reports. 6(1). 32786–32786. 38 indexed citations

11.

Ollitrault, David, Florence Legendre, Mélanie Briand, et al.. (2014). BMP-2, Hypoxia, and COL1A1 / HtrA1 siRNAs Favor Neo-Cartilage Hyaline Matrix Formation in Chondrocytes. Tissue Engineering Part C Methods. 21(2). 133–147. 36 indexed citations

12.

Gómez-Leduc, Tangni, Magalie Hervieu, David Ollitrault, et al.. (2014). Oxygen is a critical parameter for chondrogenic differentiation of human umbilical cord blood mesenchymal stem cell in 3D-cultures. Osteoarthritis and Cartilage. 22. S486–S486. 1 indexed citations

13.

Ollitrault, David, Nicolas Bigot, Noureddine Boujrad, et al.. (2014). Up-regulation of type II collagen gene by 17β-estradiol in articular chondrocytes involves Sp1/3, Sox-9, and estrogen receptor α. Journal of Molecular Medicine. 92(11). 1179–1200. 32 indexed citations

14.

Legendre, Florence, David Ollitrault, Magalie Hervieu, et al.. (2012). Enhanced Hyaline Cartilage Matrix Synthesis in Collagen Sponge Scaffolds by Using siRNA to Stabilize Chondrocytes Phenotype Cultured with Bone Morphogenetic Protein-2 Under Hypoxia. Tissue Engineering Part C Methods. 19(7). 550–567. 44 indexed citations

15.

Renard, Emmanuelle, Christos Chadjichristos, Magdalini Kypriotou, et al.. (2012). Sox9/Sox6 and Sp1 are involved in the insulin-like growth factor-I-mediated upregulation of human type II collagen gene expression in articular chondrocytes. Journal of Molecular Medicine. 90(6). 649–666. 33 indexed citations

16.

Ollitrault, David, Florence Legendre, Tangni Gómez-Leduc, et al.. (2012). Differentiation of human adult mesenchymal stem cells in chondrocytes for cartilage engineering. Osteoarthritis and Cartilage. 20. S278–S278. 1 indexed citations

17.

Bigot, Nicolas, Gallic Beauchef, Magalie Hervieu, et al.. (2012). NF-κB Accumulation Associated with COL1A1 Transactivators Defects during Chronological Aging Represses Type I Collagen Expression through a –112/–61-bp Region of the COL1A1 Promoter in Human Skin Fibroblasts. Journal of Investigative Dermatology. 132(10). 2360–2367. 44 indexed citations

18.

Aubert‐Foucher, E., Magali Demoor, Muriel Piperno, et al.. (2010). Chronic exposure of bone morphogenetic protein‐2 favors chondrogenic expression in human articular chondrocytes amplified in monolayer cultures. Journal of Cellular Biochemistry. 111(6). 1642–1651. 44 indexed citations

19.

Duval, Elise, et al.. (2009). Hypoxia‐inducible factor 1α inhibits the fibroblast‐like markers type I and type III collagen during hypoxia‐induced chondrocyte redifferentiation: Hypoxia not only induces type II collagen and aggrecan, but it also inhibits type I and type III collagen in the hypoxia‐inducible factor 1α–dependent redifferentiation of chondrocytes. Arthritis & Rheumatism. 60(10). 3038–3048. 135 indexed citations

20.

Kypriotou, Magdalini, Magali Demoor, Christos Chadjichristos, et al.. (2003). SOX9 Exerts a Bifunctional Effect on Type II Collagen Gene (COL2A1) Expression in Chondrocytes Depending on the Differentiation State. DNA and Cell Biology. 22(2). 119–129. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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