Jean‐Marie Cloutier

1.4k total citations
16 papers, 1.2k citations indexed

About

Jean‐Marie Cloutier is a scholar working on Rheumatology, Pharmacology and Molecular Biology. According to data from OpenAlex, Jean‐Marie Cloutier has authored 16 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Rheumatology, 7 papers in Pharmacology and 3 papers in Molecular Biology. Recurrent topics in Jean‐Marie Cloutier's work include Osteoarthritis Treatment and Mechanisms (15 papers), Inflammatory mediators and NSAID effects (7 papers) and Rheumatoid Arthritis Research and Therapies (4 papers). Jean‐Marie Cloutier is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (15 papers), Inflammatory mediators and NSAID effects (7 papers) and Rheumatoid Arthritis Research and Therapies (4 papers). Jean‐Marie Cloutier collaborates with scholars based in Canada and United States. Jean‐Marie Cloutier's co-authors include Jean‐Pierre Pelletier, Johanne Martel‐Pelletier, Ginette Tardif, Richard D. Komistek, Douglas A. Dennis, James B. Stiehl, Florina Moldovan, Johanne Martel‐Pelletier, John Hambor and Marie‐Pierre Faure and has published in prestigious journals such as FEBS Letters, Osteoarthritis and Cartilage and The Journal of Arthroplasty.

In The Last Decade

Jean‐Marie Cloutier

16 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jean‐Marie Cloutier Canada 15 721 340 267 242 204 16 1.2k
Boris V. Shlopov United States 8 403 0.6× 252 0.7× 88 0.3× 252 1.0× 234 1.1× 11 832
B.J.E. de Lange-Brokaar Netherlands 9 781 1.1× 250 0.7× 185 0.7× 231 1.0× 93 0.5× 11 1.0k
Christian S. Thudium Denmark 21 694 1.0× 241 0.7× 176 0.7× 379 1.6× 100 0.5× 80 1.2k
George Hilal Lebanon 15 500 0.7× 185 0.5× 172 0.6× 417 1.7× 166 0.8× 29 1.1k
Céline Deroyer Belgium 11 555 0.8× 148 0.4× 76 0.3× 387 1.6× 229 1.1× 23 890
Quanbo Ji China 17 442 0.6× 146 0.4× 159 0.6× 675 2.8× 511 2.5× 38 1.3k
Gyuseok Lee South Korea 9 386 0.5× 163 0.5× 84 0.3× 313 1.3× 181 0.9× 15 803
Judit I. Pulai United States 11 277 0.4× 106 0.3× 150 0.6× 139 0.6× 155 0.8× 16 596
Fei Zhu China 13 212 0.3× 132 0.4× 101 0.4× 261 1.1× 115 0.6× 21 782
Siru Zhou China 18 446 0.6× 98 0.3× 94 0.4× 616 2.5× 237 1.2× 42 1.1k

Countries citing papers authored by Jean‐Marie Cloutier

Since Specialization
Citations

This map shows the geographic impact of Jean‐Marie Cloutier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jean‐Marie Cloutier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jean‐Marie Cloutier more than expected).

Fields of papers citing papers by Jean‐Marie Cloutier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jean‐Marie Cloutier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jean‐Marie Cloutier. The network helps show where Jean‐Marie Cloutier may publish in the future.

Co-authorship network of co-authors of Jean‐Marie Cloutier

This figure shows the co-authorship network connecting the top 25 collaborators of Jean‐Marie Cloutier. A scholar is included among the top collaborators of Jean‐Marie Cloutier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jean‐Marie Cloutier. Jean‐Marie Cloutier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Stiehl, James B., Richard D. Komistek, Jean‐Marie Cloutier, & Douglas A. Dennis. (2000). The cruciate ligaments in total knee arthroplasty. The Journal of Arthroplasty. 15(5). 545–550. 140 indexed citations
2.
Moldovan, Florina, Jean‐Pierre Pelletier, François Mineau, et al.. (2000). Modulation of collagenase 3 in human osteoarthritic cartilage by activation of extracellular transforming growth factor β: Role of furin convertase. Arthritis & Rheumatism. 43(9). 2100–2109. 31 indexed citations
5.
6.
Tardif, Ginette, Jean‐Pierre Pelletier, Martine Dupuis, et al.. (1999). Collagenase 3 production by human osteoarthritic chondrocytes in response to growth factors and cytokines is a function of the physiologic state of the cells. Arthritis & Rheumatism. 42(6). 1147–1158. 72 indexed citations
7.
Moldovan, Florina, Jean‐Pierre Pelletier, John Hambor, Jean‐Marie Cloutier, & Johanne Martel‐Pelletier. (1997). Collagenase‐3 (matrix metalloprotease 13) is preferentially localized in the deep layer of human arthritic cartilage in situ. In vitro mimicking effect by transforming growth factor β. Arthritis & Rheumatism. 40(9). 1653–1661. 149 indexed citations
8.
Martel‐Pelletier, Johanne, Jean‐Marie Cloutier, & Jean‐Pierre Pelletier. (1997). Effects of Aceclofenac and Diclofenac on Synovial Inflammatory Factors in Human Osteoarthritis. Clinical Drug Investigation. 14(3). 226–232. 18 indexed citations
9.
Tardif, Ginette, Pascal Reboul, Jean‐Pierre Pelletier, et al.. (1996). Normal expression of type 1 insulin‐like growth factor receptor by human osteoarthritic chondrocytes with increased expression and synthesis of insulin‐like growth factor binding proteins. Arthritis & Rheumatism. 39(6). 968–978. 72 indexed citations
10.
Doré, Sylvain, Thierry Abribat, Paul Brazeau, et al.. (1995). Increased insulin‐like growth factor 1 production by human osteoarthritic chondrocytes is not dependent on growth hormone action. Arthritis & Rheumatism. 38(3). 413–419. 35 indexed citations
11.
Pelletier, Jean‐Pierre, Robert W. McCollum, John A. DiBattista, et al.. (1993). Regulation of human normal and osteoarthritic chondrocyte interleukin‐1 receptor by antirheumatic drugs. Arthritis & Rheumatism. 36(11). 1517–1527. 35 indexed citations
12.
Zafarullah, Muhammad, et al.. (1993). Distinct expression pattern of early- and late-response genes in normal and osteoarthritic human synovial membranes. Osteoarthritis and Cartilage. 1(3). 151–156. 3 indexed citations
13.
Zafarullah, Muhammad, Johanne Martel‐Pelletier, Jean‐Marie Cloutier, Lashitew Gedamu, & Jean‐Pierre Pelletier. (1992). Expression of c‐fos, c‐jun, jun‐B, metallothionein and metalloproteinase genes in human chondrocyte. FEBS Letters. 306(2-3). 169–172. 33 indexed citations
14.
Haraoui, Boulos, Jean‐Pierre Pelletier, Jean‐Marie Cloutier, Marie‐Pierre Faure, & Johanne Martel‐Pelletier. (1991). Synovial membrane histology and immunopathology in rheumatoid arthritis and osteoarthritis. In vivo effects of antirheumatic drugs. Arthritis & Rheumatism. 34(2). 153–163. 142 indexed citations
15.
Martel‐Pelletier, Johanne, Jean‐Marie Cloutier, David S. Howell, & Jean‐Pierre Pelletier. (1985). Human rheumatoid arthritic cartilage and its neutral proteoglycan‐degrading proteases. The effects of antirheumatic drugs. Arthritis & Rheumatism. 28(4). 405–412. 18 indexed citations
16.
Martel‐Pelletier, Johanne, et al.. (1984). Neutral proteases capable of proteoglycan digesting activity in osteoarthritic and normal human articular cartilage. Arthritis & Rheumatism. 27(3). 305–312. 142 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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