Jay S. Schneider

11.9k total citations · 1 hit paper
210 papers, 9.3k citations indexed

About

Jay S. Schneider is a scholar working on Neurology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Jay S. Schneider has authored 210 papers receiving a total of 9.3k indexed citations (citations by other indexed papers that have themselves been cited), including 104 papers in Neurology, 95 papers in Cellular and Molecular Neuroscience and 44 papers in Molecular Biology. Recurrent topics in Jay S. Schneider's work include Parkinson's Disease Mechanisms and Treatments (90 papers), Neurological disorders and treatments (67 papers) and Neuroscience and Neuropharmacology Research (60 papers). Jay S. Schneider is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (90 papers), Neurological disorders and treatments (67 papers) and Neuroscience and Neuropharmacology Research (60 papers). Jay S. Schneider collaborates with scholars based in United States, Norway and Canada. Jay S. Schneider's co-authors include T.I. Lidsky, Tomás R. Guilarte, Charles H. Markham, David W. Anderson, Sarah K. Kidd, Emmanuel Decamp, David P. Roeltgen, Wei Zheng, Michael Aschner and Arthur Yuwiler and has published in prestigious journals such as Science, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Jay S. Schneider

207 papers receiving 9.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Lead neurotoxicity in children: basic mechanisms and clinical correlates

2002 856 citations Jay S. Schneider et al. profile →

Peers

Countries citing papers authored by Jay S. Schneider

Since Specialization

Citations

This map shows the geographic impact of Jay S. Schneider's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jay S. Schneider with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jay S. Schneider more than expected).

Fields of papers citing papers by Jay S. Schneider

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jay S. Schneider. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jay S. Schneider. The network helps show where Jay S. Schneider may publish in the future.

Co-authorship network of co-authors of Jay S. Schneider

This figure shows the co-authorship network connecting the top 25 collaborators of Jay S. Schneider. A scholar is included among the top collaborators of Jay S. Schneider based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jay S. Schneider. Jay S. Schneider is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Influences of quality of maternal care and environmental enrichment on associative memory function in rats with early life lead exposure 2024 ·Brain and Behavior ·Jay S. Schneider, (unknown), Shamaila Zafar, (unknown), Blanca E. Himes	1
2	GM1 Ganglioside as a Disease-Modifying Therapeutic for Parkinson’s Disease: A Multi-Functional Glycosphingolipid That Targets Multiple Parkinson’s Disease-Relevant Pathogenic Mechanisms 2023 ·International Journal of Molecular Sciences ·Jay S. Schneider	5
3	Neurotoxicity and Outcomes from Developmental Lead Exposure: Persistent or Permanent? 2023 ·Environmental Health Perspectives ·Jay S. Schneider	20
4	Altered expression of glycobiology-related genes in Parkinson’s disease brain 2022 ·Frontiers in Molecular Neuroscience ·Jay S. Schneider, (unknown)	9
5	A critical role for GM1 ganglioside in the pathophysiology and potential treatment of Parkinson’s disease 2021 ·Glycoconjugate Journal ·Jay S. Schneider	11
6	Low-level lead exposure impairs fronto-executive functions: A call to update the DSM–5 with lead poisoning as a neurodevelopmental disorder. 2020 ·Psychology & Neuroscience ·Lorenz S. Neuwirth, (unknown), Jay S. Schneider, Morri Markowitz	12
7	The attention set-shifting test is sensitive for revealing sex-based impairments in executive functions following developmental lead exposure in rats 2019 ·Behavioural Brain Research ·Lorenz S. Neuwirth, (unknown), David W. Anderson, Jay S. Schneider	10
8	Altered expression of genes involved in ganglioside biosynthesis in substantia nigra neurons in Parkinson’s disease 2018 ·PLoS ONE ·Jay S. Schneider	65
9	Post-translational histone modifications and their interaction with sex influence normal brain development and elaboration of neuropsychiatric disorders 2018 ·Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease ·(unknown), (unknown), Jay S. Schneider	25
10	Different Behavioral Experiences Produce Distinctive Parallel Changes in, and Correlate With, Frontal Cortex and Hippocampal Global Post-translational Histone Levels 2018 ·Frontiers in Integrative Neuroscience ·Marissa Sobolewski, (unknown), Jay S. Schneider, Deborah A. Cory‐Slechta	7
11	Cognitive function in 1736 participants in NINDS Exploratory Trials in PD Long-term Study-1 2016 ·Parkinsonism & Related Disorders ·Anne‐Marie Wills, Jordan J. Elm, Rong Ye, Kelvin L. Chou, Sotirios A. Parashos, Robert A. Hauser, Iván Bódis-Wollner, Vanessa K. Hinson, Chadwick W. Christine, Jay S. Schneider	9
12	Caffeine and Progression of Parkinson Disease 2015 ·Clinical Neuropharmacology ·David K. Simon, Cai Wu, Barbara C. Tilley, Anne‐Marie Wills, Michael J. Aminoff, Jacquelyn Bainbridge, Robert A. Hauser, Jay S. Schneider, Saloni Sharma, Carlos Singer, Caroline M. Tanner, Daniel Truong, (unknown)	20
13	Gangliosides and Glycolipids in Neurodegenerative Disorders 2014 ·Advances in neurobiology ·Jay S. Schneider	18
14	Genetic Diversity Influences the Response of the Brain to Developmental Lead Exposure 2014 ·Toxicological Sciences ·Jay S. Schneider, Keyur Talsania, (unknown), David W. Anderson	15
15	Behavioral toxicology of cognition: Extrapolation from experimental animal models to humans 2012 ·Neurotoxicology and Teratology ·Merle G. Paule, Leonard Green, Joel Myerson, Maria C. Alvarado, Jocelyne Bachevalier, Jay S. Schneider, Susan L. Schantz	14
16	GM1 ganglioside in Parkinson's disease: Results of a five year open study 2010 ·Journal of the Neurological Sciences ·Jay S. Schneider, (unknown), Constantine Daskalakis, Franca Cambi	118
17	Sphingolipids as signaling modulators in the nervous system 1998 ·New York Academy of Sciences eBooks ·Robert W. Ledeen, Sen‐itiroh Hakomori, Allan J. Yates, Jay S. Schneider, Robert K. Yu	28
18	Dihydrexidine, a full D1 dopamine receptor agonist, induces rotational asymmetry in hemiparkinsonian monkeys 1995 ·Pharmacology Biochemistry and Behavior ·Bonnie Johnson, (unknown), Jay S. Schneider	8
19	Repeated Exposure to MPTP Does Not Produce a Permanent Movement Disorder in Cats Recovered from MPTP-induced Parkinsonism 1995 ·PubMed ·(unknown), Jay S. Schneider	5
20	Astrocytic Responses to the Dopaminergic Neurotoxin 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) in Cat and Mouse Brain 1988 ·Journal of Neuropathology & Experimental Neurology ·Jay S. Schneider, Frank Denaro	48

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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