Janeta Popovici-Müller
- Molecular Biology
- Cancer Research top 10%
- Genetics top 10%
- Hematology top 10%
- Organic Chemistry
- Co-authors
- Stefan GrößLenny DangScott A. BillerKimberly StraleyKatharine YenGerald W. ShippsValeria R. FantinShinsan M. Su
- Topics
- Acute Myeloid Leukemia Research (5 papers)Cancer, Hypoxia, and Metabolism (5 papers)Glioma Diagnosis and Treatment (4 papers)
- Cited by
- Cancer ResearchGeneticsHematology
- Partner nations
- United StatesChinaIndia
In The Last Decade
Janeta Popovici-Müller
20 papers receiving 567 citations
Peers
Comparison fields: 5 of 72
- Molecular Biology 317
- Cancer Research 208
- Genetics 129
- Hematology 71
- Organic Chemistry 69
Countries citing papers authored by Janeta Popovici-Müller
This map shows the geographic impact of Janeta Popovici-Müller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Janeta Popovici-Müller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Janeta Popovici-Müller more than expected).
Fields of papers citing papers by Janeta Popovici-Müller
This network shows the impact of papers produced by Janeta Popovici-Müller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Janeta Popovici-Müller. The network helps show where Janeta Popovici-Müller may publish in the future.
Co-authorship network of co-authors of Janeta Popovici-Müller
This figure shows the co-authorship network connecting the top 25 collaborators of Janeta Popovici-Müller. A scholar is included among the top collaborators of Janeta Popovici-Müller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Janeta Popovici-Müller. Janeta Popovici-Müller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 17 | |
| 3 | 99 | |
| 4 | 16 | |
| 5 | 72 | |
| 6 | 40 | |
| 7 | 10 | |
| 8 | 3 | |
| 9 | 17 | |
| 10 | 13 | |
| 11 | ML309: A potent inhibitor of R132H mutant IDH1 capable of reducing 2-hydroxyglutarate production in U87 MG glioblastoma cells | 16 |
| 12 | 5 | |
| 13 | 160 | |
| 14 | 9 | |
| 15 | 12 | |
| 16 | 22 | |
| 17 | 37 | |
| 18 | 6 | |
| 19 | 12 | |
| 20 | 5 |
About Janeta Popovici-Müller
Janeta Popovici-Müller is a scholar working on Cancer Research, Hematology and Hepatology, having authored 20 papers that have together received 578 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (5 papers), Cancer, Hypoxia, and Metabolism (5 papers) and Glioma Diagnosis and Treatment (4 papers). The work is most often cited by research in Cancer Research (208 citations), Genetics (129 citations) and Hematology (71 citations). Janeta Popovici-Müller has collaborated with scholars based in United States, China and India. Frequent co-authors include Stefan Größ, Lenny Dang, Scott A. Biller, Kimberly Straley, Katharine Yen, Gerald W. Shipps, Valeria R. Fantin, Shinsan M. Su, Yongqi Deng and Hua Yang. Their work appears in journals such as Journal of Biological Chemistry, Nature Communications and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.