James F. Catterall
- Molecular Biology top 5%
- Endocrinology, Diabetes and Metabolism top 1%
- Genetics top 5%
- Reproductive Medicine top 2%
- Cardiology and Cardiovascular Medicine top 10%
- Co-authors
- C. Wayne BardinDianne O. HardyBert W. O’MalleyMatthew P. HardyAnna MeseguerSavio L.C. WooAchilles DugaiczykYueming Ding
- Topics
- Hormonal and reproductive studies (16 papers)Sexual Differentiation and Disorders (12 papers)Hormonal Regulation and Hypertension (11 papers)
- Partner nations
- United StatesIndiaUnited Kingdom
In The Last Decade
James F. Catterall
56 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 115
- Molecular Biology 1.3k
- Endocrinology, Diabetes and Metabolism 854
- Genetics 704
- Reproductive Medicine 371
- Cardiology and Cardiovascular Medicine 218
Countries citing papers authored by James F. Catterall
This map shows the geographic impact of James F. Catterall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James F. Catterall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James F. Catterall more than expected).
Fields of papers citing papers by James F. Catterall
This network shows the impact of papers produced by James F. Catterall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James F. Catterall. The network helps show where James F. Catterall may publish in the future.
Co-authorship network of co-authors of James F. Catterall
This figure shows the co-authorship network connecting the top 25 collaborators of James F. Catterall. A scholar is included among the top collaborators of James F. Catterall based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James F. Catterall. James F. Catterall is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 110 | |
| 3 | 6 | |
| 4 | 122 | |
| 5 | 5 | |
| 6 | 33 | |
| 7 | 27 | |
| 8 | 134 | |
| 9 | 19 | |
| 10 | 6 | |
| 11 | 48 | |
| 12 | 15 | |
| 13 | 20 | |
| 14 | 4 | |
| 15 | 10 | |
| 16 | 4 | |
| 17 | 35 | |
| 18 | 28 | |
| 19 | 7 | |
| 20 | 43 |
About James F. Catterall
James F. Catterall is a scholar working on Endocrinology, Diabetes and Metabolism, Reproductive Medicine and Genetics, having authored 56 papers that have together received 2.5k indexed citations. Recurring topics across this work include Hormonal and reproductive studies (16 papers), Sexual Differentiation and Disorders (12 papers) and Hormonal Regulation and Hypertension (11 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (854 citations), Reproductive Medicine (371 citations) and Genetics (704 citations). James F. Catterall has collaborated with scholars based in United States, India and United Kingdom. Frequent co-authors include C. Wayne Bardin, Dianne O. Hardy, Bert W. O’Malley, Matthew P. Hardy, Anna Meseguer, Savio L.C. Woo, Achilles Dugaiczyk, Yueming Ding, Ren‐Shan Ge and Robin L. Davisson. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.