Jamal B. Williams

620 total citations
16 papers, 393 citations indexed

About

Jamal B. Williams is a scholar working on Molecular Biology, Genetics and Physiology. According to data from OpenAlex, Jamal B. Williams has authored 16 papers receiving a total of 393 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Genetics and 6 papers in Physiology. Recurrent topics in Jamal B. Williams's work include Genetics and Neurodevelopmental Disorders (6 papers), Alzheimer's disease research and treatments (5 papers) and Epigenetics and DNA Methylation (4 papers). Jamal B. Williams is often cited by papers focused on Genetics and Neurodevelopmental Disorders (6 papers), Alzheimer's disease research and treatments (5 papers) and Epigenetics and DNA Methylation (4 papers). Jamal B. Williams collaborates with scholars based in United States, Pakistan and China. Jamal B. Williams's co-authors include Zhen Yan, Qing Cao, I. Martha Skerrett, Wei Wang, Fengwei Yang, Zijun Wang, Tao Tan, Kaijie Ma, Benjamin Rein and Ping Zhong and has published in prestigious journals such as Nature Communications, PLoS ONE and Brain.

In The Last Decade

Jamal B. Williams

14 papers receiving 390 citations

Peers

Jamal B. Williams
Qing Cao United States
Thekla J. Hemstedt United States
Jessica M. Fawcett‐Patel United States
Shaghayegh Navabpour United States
Fengwei Yang United States
Bisrat T. Woldemichael Switzerland
André O. White United States
Freddy Zhang United States
Sarah C. Borrie Belgium
Wen‐Chin Huang United States
Qing Cao United States
Jamal B. Williams
Citations per year, relative to Jamal B. Williams Jamal B. Williams (= 1×) peers Qing Cao

Countries citing papers authored by Jamal B. Williams

Since Specialization
Citations

This map shows the geographic impact of Jamal B. Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jamal B. Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jamal B. Williams more than expected).

Fields of papers citing papers by Jamal B. Williams

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jamal B. Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jamal B. Williams. The network helps show where Jamal B. Williams may publish in the future.

Co-authorship network of co-authors of Jamal B. Williams

This figure shows the co-authorship network connecting the top 25 collaborators of Jamal B. Williams. A scholar is included among the top collaborators of Jamal B. Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jamal B. Williams. Jamal B. Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
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Williams, Jamal B., et al.. (2024). Large-scale analysis reveals racial disparities in the prevalence of ADHD and conduct disorders. Scientific Reports. 14(1). 3 indexed citations
4.
Cao, Qing, et al.. (2023). Longitudinal characterization of behavioral, morphological and transcriptomic changes in a tauopathy mouse model. Aging. 15(21). 11697–11719. 4 indexed citations
5.
Williams, Jamal B., Qing Cao, Wei Wang, et al.. (2023). Inhibition of histone methyltransferase Smyd3 rescues NMDAR and cognitive deficits in a tauopathy mouse model. Nature Communications. 14(1). 91–91. 16 indexed citations
6.
Mitra, Swarup, Jennifer A. Martin, Jamal B. Williams, et al.. (2022). EGR3 regulates opioid-related nociception and motivation in male rats. Psychopharmacology. 239(11). 3539–3550. 2 indexed citations
7.
Rapanelli, Maximiliano, Jamal B. Williams, Kaijie Ma, et al.. (2022). Targeting histone demethylase LSD1 for treatment of deficits in autism mouse models. Molecular Psychiatry. 27(8). 3355–3366. 24 indexed citations
8.
Williams, Jamal B., Jennifer A. Martin, Ping Zhong, et al.. (2022). A convergent mechanism of high risk factors ADNP and POGZ in neurodevelopmental disorders. Brain. 145(9). 3250–3263. 14 indexed citations
9.
Wang, Wei, Qing Cao, Tao Tan, et al.. (2021). Epigenetic treatment of behavioral and physiological deficits in a tauopathy mouse model. Aging Cell. 20(10). e13456–e13456. 23 indexed citations
10.
Qin, Luye, Jamal B. Williams, Tao Tan, et al.. (2021). Deficiency of autism risk factor ASH1L in prefrontal cortex induces epigenetic aberrations and seizures. Nature Communications. 12(1). 6589–6589. 37 indexed citations
11.
Wang, Zijun, Benjamin Rein, Ping Zhong, et al.. (2021). Autism risk gene KMT5B deficiency in prefrontal cortex induces synaptic dysfunction and social deficits via alterations of DNA repair and gene transcription. Neuropsychopharmacology. 46(9). 1617–1626. 33 indexed citations
12.
Williams, Jamal B., Qing Cao, & Zhen Yan. (2021). Transcriptomic analysis of human brains with Alzheimer’s disease reveals the altered expression of synaptic genes linked to cognitive deficits. Brain Communications. 3(3). fcab123–fcab123. 35 indexed citations
13.
Rein, Benjamin, Tao Tan, Fengwei Yang, et al.. (2020). Reversal of synaptic and behavioral deficits in a 16p11.2 duplication mouse model via restoration of the GABA synapse regulator Npas4. Molecular Psychiatry. 26(6). 1967–1979. 37 indexed citations
14.
Cao, Qing, Wei Wang, Jamal B. Williams, et al.. (2020). Targeting histone K4 trimethylation for treatment of cognitive and synaptic deficits in mouse models of Alzheimer’s disease. Science Advances. 6(50). 74 indexed citations
15.
Tan, Tao, Wei Wang, Jamal B. Williams, et al.. (2018). Stress Exposure in Dopamine D4 Receptor Knockout Mice Induces Schizophrenia-Like Behaviors via Disruption of GABAergic Transmission. Schizophrenia Bulletin. 45(5). 1012–1023. 19 indexed citations
16.
Skerrett, I. Martha & Jamal B. Williams. (2016). A structural and functional comparison of gap junction channels composed of connexins and innexins. Developmental Neurobiology. 77(5). 522–547. 72 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026