Kaijie Ma

1.7k total citations
21 papers, 1.2k citations indexed

About

Kaijie Ma is a scholar working on Genetics, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Kaijie Ma has authored 21 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Genetics, 11 papers in Molecular Biology and 11 papers in Cognitive Neuroscience. Recurrent topics in Kaijie Ma's work include Genetics and Neurodevelopmental Disorders (12 papers), Autism Spectrum Disorder Research (11 papers) and Neuroscience and Neuropharmacology Research (4 papers). Kaijie Ma is often cited by papers focused on Genetics and Neurodevelopmental Disorders (12 papers), Autism Spectrum Disorder Research (11 papers) and Neuroscience and Neuropharmacology Research (4 papers). Kaijie Ma collaborates with scholars based in United States, China and Canada. Kaijie Ma's co-authors include Zhen Yan, Luye Qin, Emmanuel Matas, Jing Wei, Benjamin Rein, Zijun Wang, Ping Zhong, Zihua Hu, Jia Cheng and Lara J. Duffney and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and The Journal of Experimental Medicine.

In The Last Decade

Kaijie Ma

20 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kaijie Ma United States	17	570	516	476	293	134	21	1.2k
Andre H. Lagrange United States	22	539 0.9×	374 0.7×	130 0.3×	530 1.8×	87 0.6×	50	1.5k
Caroline L. Tinsley United Kingdom	11	855 1.5×	339 0.7×	237 0.5×	588 2.0×	153 1.1×	14	1.5k
Yi Sul Cho South Korea	17	556 1.0×	484 0.9×	463 1.0×	548 1.9×	189 1.4×	29	1.3k
Won Mah South Korea	12	635 1.1×	551 1.1×	463 1.0×	428 1.5×	81 0.6×	16	1.3k
Pradeep Punnakkal India	9	369 0.6×	158 0.3×	316 0.7×	566 1.9×	244 1.8×	15	888
Bing Lang China	20	621 1.1×	209 0.4×	100 0.2×	354 1.2×	124 0.9×	65	1.1k
Jia Cheng United States	21	725 1.3×	292 0.6×	344 0.7×	465 1.6×	205 1.5×	38	1.5k
Annie Vogel Ciernia United States	20	689 1.2×	403 0.8×	436 0.9×	390 1.3×	142 1.1×	34	1.5k
Jennifer Larimore United States	17	398 0.7×	429 0.8×	292 0.6×	298 1.0×	80 0.6×	19	908
Samantha J. Fung Australia	22	693 1.2×	228 0.4×	360 0.8×	847 2.9×	108 0.8×	27	1.9k

Countries citing papers authored by Kaijie Ma

Since Specialization

Citations

This map shows the geographic impact of Kaijie Ma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kaijie Ma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kaijie Ma more than expected).

Fields of papers citing papers by Kaijie Ma

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kaijie Ma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kaijie Ma. The network helps show where Kaijie Ma may publish in the future.

Co-authorship network of co-authors of Kaijie Ma

This figure shows the co-authorship network connecting the top 25 collaborators of Kaijie Ma. A scholar is included among the top collaborators of Kaijie Ma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kaijie Ma. Kaijie Ma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ma, Kaijie, et al.. (2024). Chemogenetic Inhibition of Prefrontal Cortex Ameliorates Autism-Like Social Deficits and Absence-Like Seizures in a Gene-Trap Ash1l Haploinsufficiency Mouse Model. Genes. 15(12). 1619–1619.

Ma, Kaijie, et al.. (2024). A sexually dimorphic signature of activity-dependent BDNF signaling on the intrinsic excitability of pyramidal neurons in the prefrontal cortex. Frontiers in Cellular Neuroscience. 18. 1496930–1496930. 1 indexed citations

Williams, Jamal B., Qing Cao, Wei Wang, et al.. (2023). Inhibition of histone methyltransferase Smyd3 rescues NMDAR and cognitive deficits in a tauopathy mouse model. Nature Communications. 14(1). 91–91. 16 indexed citations

Ma, Kaijie, et al.. (2023). Diminished activity-dependent BDNF signaling differentially causes autism-like behavioral deficits in male and female mice. Frontiers in Psychiatry. 14. 1182472–1182472. 13 indexed citations

Rapanelli, Maximiliano, Jamal B. Williams, Kaijie Ma, et al.. (2022). Targeting histone demethylase LSD1 for treatment of deficits in autism mouse models. Molecular Psychiatry. 27(8). 3355–3366. 24 indexed citations

Qin, Luye, Kaijie Ma, & Zhen Yan. (2021). Rescue of histone hypoacetylation and social deficits by ketogenic diet in a Shank3 mouse model of autism. Neuropsychopharmacology. 47(6). 1271–1279. 24 indexed citations

Qin, Luye, Jamal B. Williams, Tao Tan, et al.. (2021). Deficiency of autism risk factor ASH1L in prefrontal cortex induces epigenetic aberrations and seizures. Nature Communications. 12(1). 6589–6589. 37 indexed citations

Wang, Zijun, Benjamin Rein, Ping Zhong, et al.. (2021). Autism risk gene KMT5B deficiency in prefrontal cortex induces synaptic dysfunction and social deficits via alterations of DNA repair and gene transcription. Neuropsychopharmacology. 46(9). 1617–1626. 33 indexed citations

Rein, Benjamin, Kaijie Ma, & Zhen Yan. (2020). A standardized social preference protocol for measuring social deficits in mouse models of autism. Nature Protocols. 15(10). 3464–3477. 130 indexed citations

10.

Rapanelli, Maximiliano, Tao Tan, Wei Wang, et al.. (2019). Behavioral, circuitry, and molecular aberrations by region-specific deficiency of the high-risk autism gene Cul3. Molecular Psychiatry. 26(5). 1491–1504. 55 indexed citations

11.

Qin, Luye, Kaijie Ma, & Zhen Yan. (2019). Chemogenetic Activation of Prefrontal Cortex in Shank3-Deficient Mice Ameliorates Social Deficits, NMDAR Hypofunction, and Sgk2 Downregulation. iScience. 17. 24–35. 39 indexed citations

12.

Wang, Zijun, Ping Zhong, Kaijie Ma, et al.. (2019). Amelioration of autism-like social deficits by targeting histone methyltransferases EHMT1/2 in Shank3-deficient mice. Molecular Psychiatry. 25(10). 2517–2533. 66 indexed citations

13.

Zheng, Yan, Aiyi Liu, Zijun Wang, et al.. (2019). Inhibition of EHMT1/2 rescues synaptic and cognitive functions for Alzheimer’s disease. Brain. 142(3). 787–807. 113 indexed citations

14.

Qin, Luye, Kaijie Ma, Zijun Wang, et al.. (2018). Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition. Nature Neuroscience. 21(4). 564–575. 172 indexed citations

15.

Tan, Tao, Wei Wang, Jamal B. Williams, et al.. (2018). Stress Exposure in Dopamine D4 Receptor Knockout Mice Induces Schizophrenia-Like Behaviors via Disruption of GABAergic Transmission. Schizophrenia Bulletin. 45(5). 1012–1023. 19 indexed citations

16.

Qin, Luye, Wenhua Liu, Kaijie Ma, et al.. (2016). The ADHD-linked human dopamine D4 receptor variant D4.7 induces over-suppression of NMDA receptor function in prefrontal cortex. Neurobiology of Disease. 95. 194–203. 16 indexed citations

17.

Duffney, Lara J., Ping Zhong, Jing Wei, et al.. (2015). Autism-like Deficits in Shank3-Deficient Mice Are Rescued by Targeting Actin Regulators. Cell Reports. 11(9). 1400–1413. 226 indexed citations

18.

O’Donovan, Kevin J., Kaijie Ma, Hengchang Guo, et al.. (2014). B-RAF kinase drives developmental axon growth and promotes axon regeneration in the injured mature CNS. The Journal of Experimental Medicine. 211(5). 801–814. 77 indexed citations

19.

O’Donovan, Kevin J., Kaijie Ma, Hengchang Guo, et al.. (2014). B-RAF kinase drives developmental axon growth and promotes axon regeneration in the injured mature CNS. The Journal of Cell Biology. 205(2). 2052OIA78–2052OIA78. 1 indexed citations

20.

Zhao, Zhongqiu, Fu‐Quan Huo, Joseph Jeffry, et al.. (2013). Chronic itch development in sensory neurons requires BRAF signaling pathways. Journal of Clinical Investigation. 123(11). 4769–4780. 85 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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