J. T. Reeves

4.4k total citations
64 papers, 3.4k citations indexed

About

J. T. Reeves is a scholar working on Genetics, Endocrine and Autonomic Systems and Pulmonary and Respiratory Medicine. According to data from OpenAlex, J. T. Reeves has authored 64 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Genetics, 27 papers in Endocrine and Autonomic Systems and 26 papers in Pulmonary and Respiratory Medicine. Recurrent topics in J. T. Reeves's work include High Altitude and Hypoxia (32 papers), Neuroscience of respiration and sleep (27 papers) and Nitric Oxide and Endothelin Effects (10 papers). J. T. Reeves is often cited by papers focused on High Altitude and Hypoxia (32 papers), Neuroscience of respiration and sleep (27 papers) and Nitric Oxide and Endothelin Effects (10 papers). J. T. Reeves collaborates with scholars based in United States, China and Canada. J. T. Reeves's co-authors include Adam Tucker, R. F. Grover, Lorna G. Moore, A. Cymerman, Ivan F. McMurtry, R.C. Murphy, R. E. McCullough, N. F. Voelkel, P M Henson and Scott Worthen and has published in prestigious journals such as Science, The Journal of Physiology and Journal of Applied Physiology.

In The Last Decade

J. T. Reeves

64 papers receiving 3.2k citations

Peers

J. T. Reeves
Robert F. Grover United States
O Oelz Switzerland
R. F. Grover United States
James S. Milledge United Kingdom
Cláudio Sartori Switzerland
A. P. Fishman United States
Norberto C. González United States
Silvia Ulrich Switzerland
Robert F. Grover United States
J. T. Reeves
Citations per year, relative to J. T. Reeves J. T. Reeves (= 1×) peers Robert F. Grover

Countries citing papers authored by J. T. Reeves

Since Specialization
Citations

This map shows the geographic impact of J. T. Reeves's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. T. Reeves with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. T. Reeves more than expected).

Fields of papers citing papers by J. T. Reeves

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. T. Reeves. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. T. Reeves. The network helps show where J. T. Reeves may publish in the future.

Co-authorship network of co-authors of J. T. Reeves

This figure shows the co-authorship network connecting the top 25 collaborators of J. T. Reeves. A scholar is included among the top collaborators of J. T. Reeves based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. T. Reeves. J. T. Reeves is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mazzeo, Robert S., George A. Brooks, G. E. Butterfield, et al.. (1994). Beta-adrenergic blockade does not prevent the lactate response to exercise after acclimatization to high altitude. Journal of Applied Physiology. 76(2). 610–615. 32 indexed citations
2.
Sun, Shanquan, Tarshi Droma, Tao Ji, et al.. (1992). Internal carotid arterial flow velocity during exercise in Tibetan and Han residents of Lhasa (3,658 m). Journal of Applied Physiology. 73(6). 2638–2642. 47 indexed citations
3.
Sun, S., Tarshi Droma, R. E. McCullough, et al.. (1990). Decreased Ventilation and Hypoxic Ventilatory Responsiveness Are Not Reversed by Naloxone in Lhasa Residents with Chronic Mountain Sickness. American Review of Respiratory Disease. 142(6_pt_1). 1294–1300. 20 indexed citations
4.
Hiatt, William R., Szu‐Wei Huang, Judith G. Regensteiner, et al.. (1989). Venous occlusion plethysmography reduces arterial diameter and flow velocity. Journal of Applied Physiology. 66(5). 2239–2244. 44 indexed citations
5.
Burghuber, Otto C., Robert J. Strife, Joseph A. Zirrolli, et al.. (1985). Leukotriene inhibitors attenuate rat lung injury induced by hydrogen peroxide.. PubMed. 131(5). 778–85. 47 indexed citations
6.
Stenmark, Kurt R., Melvin L. Morganroth, Linda K. Remigio, et al.. (1985). Alveolar inflammation and arachidonate metabolism in monocrotaline-induced pulmonary hypertension. American Journal of Physiology-Heart and Circulatory Physiology. 248(6). H859–H866. 72 indexed citations
7.
Voelkel, N. F., John G. Simpson, Scott Worthen, et al.. (1983). Platelet-activating factor causes pulmonary vasoconstriction and edema via platelet-independent leukotriene formation.. PubMed. 12. 179–83. 11 indexed citations
8.
Sugita, Takashi, Kurt R. Stenmark, Wiltz W. Wagner, et al.. (1983). Abnormal Alveolar Cells in Monocrotaline Induced Pulmonary Hypertension. Experimental Lung Research. 5(3). 201–215. 30 indexed citations
9.
Reeves, J. T., et al.. (1983). Low exercise pulmonary resistance is not dependent on vasodilator prostaglandins. Journal of Applied Physiology. 55(2). 558–561. 16 indexed citations
10.
Grondelle, Albertus van, Roy V. Ditchey, B. M. Groves, W. W. Wagner, & J. T. Reeves. (1983). Thermodilution method overestimates low cardiac output in humans. American Journal of Physiology-Heart and Circulatory Physiology. 245(4). H690–H692. 114 indexed citations
11.
Gerber, J G, N. F. Voelkel, Alan S. Nies, Ivan F. McMurtry, & J. T. Reeves. (1980). Moderation of hypoxic vasoconstriction by infused arachidonic acid: role of PGI2. Journal of Applied Physiology. 49(1). 107–112. 79 indexed citations
12.
Will, D. H., Ivan F. McMurtry, J. T. Reeves, & R. F. Grover. (1978). Cold-induced pulmonary hypertension in cattle. Journal of Applied Physiology. 45(3). 469–473. 37 indexed citations
13.
Doekel, Robert C., et al.. (1978). Potentiation of hypoxic pulmonary vasoconstriction by ethyl alcohol in dogs. Journal of Applied Physiology. 44(1). 76–80. 27 indexed citations
14.
McMurtry, Ivan F., et al.. (1978). Lungs from chronically hypoxic rats have decreased pressor response to acute hypoxia. American Journal of Physiology-Heart and Circulatory Physiology. 235(1). H104–H109. 99 indexed citations
15.
Reeves, J. T., et al.. (1975). Preservation of hypoxic pulmonary pressor response in canine pneumococcal pneumonia.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 112(6). 817–22. 5 indexed citations
16.
Weir, Ε. Kenneth, J. T. Reeves, & R. F. Grover. (1974). Meclofenamate and indomethacin augment the pulmonary pressor response to hypoxia and exogenous prostaglandin F2α (PGF2α). 17(3). 6 indexed citations
17.
Weir, Ε. Kenneth, J. T. Reeves, & R. F. Grover. (1974). Inhibition of prostaglandin synthetase prevents blockade of hypoxic pulmonary hypertension by endotoxin. Clinical research. 22(4). 2 indexed citations
18.
Reeves, J. T., et al.. (1972). Growth of the fetal calf and its arterial pressure, blood gases, and hematologic data. Journal of Applied Physiology. 32(2). 240–244. 23 indexed citations
19.
Dawes, G. S., et al.. (1969). Hypoxaemia and aortic chemoreceptor function in foetal lambs. The Journal of Physiology. 201(1). 105–116. 70 indexed citations
20.
Grover, R. F., et al.. (1967). Muscular exercise in young men native to 3,100 m altitude. Journal of Applied Physiology. 22(3). 555–564. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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