J. M. Saavedra

1.1k total citations
16 papers, 931 citations indexed

About

J. M. Saavedra is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, J. M. Saavedra has authored 16 papers receiving a total of 931 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cellular and Molecular Neuroscience, 6 papers in Molecular Biology and 5 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in J. M. Saavedra's work include Neurotransmitter Receptor Influence on Behavior (5 papers), Receptor Mechanisms and Signaling (4 papers) and Stress Responses and Cortisol (3 papers). J. M. Saavedra is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (5 papers), Receptor Mechanisms and Signaling (4 papers) and Stress Responses and Cortisol (3 papers). J. M. Saavedra collaborates with scholars based in United States, Czechia and Italy. J. M. Saavedra's co-authors include H. Grobecker, J Axelrod, Miklós Palkovits, Koichiro Tsutsumi, M J Brownstein, John S. Kizer, D.M. Jacobowitz, M. Brownstein, László Záborszky and Andrew V. Schally and has published in prestigious journals such as Nature, Circulation Research and Brain Research.

In The Last Decade

J. M. Saavedra

16 papers receiving 886 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
J. M. Saavedra United States 12 414 302 198 193 150 16 931
L. Cass Terry United States 22 482 1.2× 260 0.9× 266 1.3× 272 1.4× 393 2.6× 36 1.2k
Judith A. Poat United Kingdom 21 655 1.6× 605 2.0× 198 1.0× 116 0.6× 70 0.5× 57 1.1k
Maria Martire Italy 23 858 2.1× 792 2.6× 135 0.7× 113 0.6× 47 0.3× 62 1.4k
R.B. Rastogi Canada 21 457 1.1× 243 0.8× 118 0.6× 109 0.6× 254 1.7× 56 1.0k
T. Kubo Japan 24 542 1.3× 406 1.3× 281 1.4× 476 2.5× 54 0.4× 67 1.3k
Guibao Gu United States 13 365 0.9× 280 0.9× 249 1.3× 260 1.3× 179 1.2× 17 1.0k
Anne‐Marie Galzin France 20 607 1.5× 411 1.4× 161 0.8× 115 0.6× 111 0.7× 46 1.1k
Deborah L. Armstrong United States 18 506 1.2× 518 1.7× 140 0.7× 222 1.2× 81 0.5× 37 1.1k
Haruo Shibuya Japan 17 675 1.6× 489 1.6× 167 0.8× 204 1.1× 63 0.4× 32 1.1k
H. D. Taube Germany 11 966 2.3× 947 3.1× 311 1.6× 82 0.4× 52 0.3× 13 1.4k

Countries citing papers authored by J. M. Saavedra

Since Specialization
Citations

This map shows the geographic impact of J. M. Saavedra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. M. Saavedra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. M. Saavedra more than expected).

Fields of papers citing papers by J. M. Saavedra

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. M. Saavedra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. M. Saavedra. The network helps show where J. M. Saavedra may publish in the future.

Co-authorship network of co-authors of J. M. Saavedra

This figure shows the co-authorship network connecting the top 25 collaborators of J. M. Saavedra. A scholar is included among the top collaborators of J. M. Saavedra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. M. Saavedra. J. M. Saavedra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Tsutsumi, Koichiro & J. M. Saavedra. (1992). Heterogeneity of angiotensin II AT2 receptors in the rat brain.. Molecular Pharmacology. 41(2). 290–297. 104 indexed citations
2.
Shigematsu, Kazuto, M. Niwa, Masaki Kurihara, Eero Ċastrén, & J. M. Saavedra. (1987). Alterations in substance P binding in brain nuclei of spontaneously hypertensive rats. American Journal of Physiology-Heart and Circulatory Physiology. 252(2). H301–H306. 8 indexed citations
3.
Niwa, M., Kazuto Shigematsu, Laura M. Plunkett, & J. M. Saavedra. (1985). High-affinity substance P binding sites in rat sympathetic ganglia. American Journal of Physiology-Heart and Circulatory Physiology. 249(3). H694–H697. 19 indexed citations
4.
Saavedra, J. M., Catherine Rougeot, & F. Dray. (1983). Selective Decrease in Immunoreactive Somatostatin in Rat Intermediate Pituitary Lobe after Stalk Section. Neuroendocrinology. 37(2). 164–165. 7 indexed citations
5.
Bo, A. Del, et al.. (1983). Fastigial stimulation in rats releases adrenomedullary catecholamines. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 244(6). R801–R809. 14 indexed citations
6.
Saavedra, J. M., Carmine Rocca, Richard McCarty, et al.. (1983). Increased adrenal catecholamines in salt-sensitive genetically hypertensive Dahl rats. American Journal of Physiology-Heart and Circulatory Physiology. 245(5). H762–H766. 27 indexed citations
7.
Brosnihan, K. Bridget, Carlos M. Ferrario, J. M. Saavedra, & Robert C. Speth. (1981). Catecholamines and serotonin in the area postrema of normal and sodium-depleted dogs.. Hypertension. 3(6_pt_2). II–151. 3 indexed citations
8.
Saavedra, J. M.. (1981). Spontaneously (genetic) hypertensive rats: Naloxone-reversible and propranolol-reversible decrease in pain sensitivity. Cellular and Molecular Life Sciences. 37(9). 1002–1003. 31 indexed citations
9.
Saavedra, J. M., H. Grobecker, & J Axelrod. (1978). Changes in central catecholaminergic neurons in the spontaneously (genetic) hypertensive rat.. Circulation Research. 42(4). 529–534. 213 indexed citations
10.
Saavedra, J. M.. (1977). Distribution of serotonin and synthesizing enzymes in discrete areas of the brain.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 36(8). 2134–41. 95 indexed citations
11.
Palkovits, Miklós, J. M. Saavedra, D.M. Jacobowitz, et al.. (1977). Serotonergic innervation of the forebrain: Effect of lesions on serotonin and tryptophan hydroxylase levels. Brain Research. 130(1). 121–134. 145 indexed citations
12.
Palkovits, Miklós, M J Brownstein, John S. Kizer, J. M. Saavedra, & Irwin J. Kopin. (1976). Effect of Stress on Serotonin Concentration and Tryptophan Hydroxylase Activity of Brain Nuclei. Neuroendocrinology. 22(4). 298–304. 44 indexed citations
13.
Kizer, John S., Miklós Palkovits, Justin A. Zivin, et al.. (1974). The Effect of Endocrinological Manipulations on Tyrosine Hydroxylase and Dopamine-β-Hydroxylase Activities in Individual Hypothalamic Nuclei of the Adult Male Rat11. Endocrinology. 95(3). 799–812. 84 indexed citations
14.
Palkovits, Miklós, Akira Arimura, M J Brownstein, Andrew V. Schally, & J. M. Saavedra. (1974). Luteinizing Hormone-Releasing Hormone (LH-RH) Content of the Hypothalamic Nuclei in Rat. Endocrinology. 95(2). 554–558. 106 indexed citations
15.
Saavedra, J. M., B Heller, & E. Fischer. (1970). Antagonistic Effects of Tryptamine and β-Phenylethylamine on the Behaviour of Rodents. Nature. 226(5248). 868–868. 11 indexed citations
16.
Heller, B, J. M. Saavedra, & E. Fischer. (1968). Influence of adrenergic blocking agents upon morphine and catecholamine analgesic effect. Cellular and Molecular Life Sciences. 24(8). 804–805. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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