J. Holtman

528 citations
19 papers · 487 · h-index 8

Impact in

Papers in

J. Holtman

19 papers receiving 470 citations

Peers

J. Holtman
Comparison fields: 5 of 50
  • Endocrine and Autonomic Systems 362
  • Cellular and Molecular Neuroscience 161
  • Social Psychology 165
  • Pharmacy 38
  • Cognitive Neuroscience 142
Replace A. Haji with:
A. Haji Japan
Miriam Haller Germany
Marie‐Pierre Morin‐Surun France
R.L. Martin-Body New Zealand
Michal G. Fortuna United States
Philip J. Gatti United States
Steven W. Shaver Canada
M. E. Schlaefke Germany
F. Laczi Hungary
R. Nissen Canada
J. Holtman relative to A. Haji Japan A. Haji's profile →
Citations per field
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A. Haji · 1×
Citations per year

Countries citing papers authored by J. Holtman

Since Specialization
Citations

This map shows the geographic impact of J. Holtman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Holtman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Holtman more than expected).

Fields of papers citing papers by J. Holtman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Holtman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Holtman. The network helps show where J. Holtman may publish in the future.

Co-authors

The 22 scholars most cited alongside J. Holtman, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with J. Holtman Line = papers co-authored together J. Holtman links everyone, so they are left out of the graph.

All Works

19 of 19 papers shown
#Work
1 1986122
2 199097
3 198487
4 198881
5 199030
6 198517
7 198915
8 19837
9 19847
10 19895
11 20103
12 19913
13 20073
14 19922
15 20082
16 20092
17 20062
18 20091
19 20111

About J. Holtman

J. Holtman is a scholar working on Endocrine and Autonomic Systems, Molecular Biology, Physiology, Cellular and Molecular Neuroscience and Pulmonary and Respiratory Medicine, having authored 19 papers that have together received 487 indexed citations. Recurring topics across this work include Neuroscience of respiration and sleep (9 papers), Pain Mechanisms and Treatments (5 papers), Pharmacological Receptor Mechanisms and Effects (5 papers), Nicotinic Acetylcholine Receptors Study (3 papers), Neuroscience and Neuropharmacology Research (3 papers), Receptor Mechanisms and Signaling (3 papers), Respiratory and Cough-Related Research (3 papers) and Neuropeptides and Animal Physiology (2 papers). The work is most often cited by research in Endocrine and Autonomic Systems (362 citations), Cellular and Molecular Neuroscience (161 citations), Social Psychology (165 citations), Pharmacy (38 citations) and Cognitive Neuroscience (142 citations). J. Holtman has collaborated with scholars based in United States and Sweden. Frequent co-authors include Thomas E. Dick, A. J. Berger, Dexter F. Speck, Kathryn A. King, R A Gillis, Visser Tj, Tomas Hökfelt, A. Claudio Cuello, L.R. Skirboll and J Richter. Their work appears in journals such as Journal of Pain, Journal of Pharmacology and Experimental Therapeutics, Neuroscience, Journal of Neuroscience and Neuropharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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