J. Egeberg

1.4k citations
42 papers · 978 indexed · h-index 16

J. Egeberg

41 papers receiving 836 citations

Peers

J. Egeberg
Comparison fields: 5 of 86
  • Endocrinology, Diabetes and Metabolism 465
  • Genetics 600
  • Surgery 570
  • Immunology 139
  • Ophthalmology 38
Replace Akira Kasuga with:
Akira Kasuga Japan
H. M. Dosch Canada
Giuseppina Emanuela Grieco Italy
J P Mordes United States
Harold D. Chapman United States
Natalie Amirian United States
Mats Lake Sweden
Hitoshi Katsuta Japan
Mitsukazu Gotoh Japan
Mitsuo Tashiro Japan
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Citations per field
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Citations per year

Countries citing papers authored by J. Egeberg

Since Specialization
Citations

This map shows the geographic impact of J. Egeberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Egeberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Egeberg more than expected).

Fields of papers citing papers by J. Egeberg

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Egeberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Egeberg. The network helps show where J. Egeberg may publish in the future.

Co-authorship network

The 25 scholars most cited alongside J. Egeberg, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with J. Egeberg Line = papers co-authored together J. Egeberg links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 19972
2 19898
3 19894
4
Mechanisms of pancreatic beta-cell destruction in type I diabetes.
1989117
5 19888
6 198762
7 198658
8 198314
9 198213
10
Direct streptozotocin toxicity on dispersed mouse islet cells determined by [51]Cr-release.
19805
11 197911
12 19772
13 197713
14 19765
15 19741
16 197467
17 197332
18
The fate of corpuscular antigen in leukocyte migration cultures.
19724
19 196714
20 196424

About J. Egeberg

J. Egeberg is a scholar working on Endocrinology, Diabetes and Metabolism, Structural Biology, Genetics, Geriatrics and Gerontology and Surgery, having authored 42 papers that have together received 978 indexed citations. Recurring topics across this work include Diabetes and associated disorders (18 papers), Pancreatic function and diabetes (16 papers), Diabetes Management and Research (10 papers), Eosinophilic Esophagitis (2 papers), Multiple Myeloma Research and Treatments (2 papers), Pancreatitis Pathology and Treatment (2 papers), Melanoma and MAPK Pathways (2 papers) and Caveolin-1 and cellular processes (2 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (465 citations), Genetics (600 citations), Surgery (570 citations), Immunology (139 citations) and Ophthalmology (38 citations). J. Egeberg has collaborated with scholars based in Denmark, Sweden and United States. Frequent co-authors include Jørn Nerup, Thomas Mandrup‐Poulsen, Ole Andersen, Jacob E. Poulsen, Gunnar Bendixen, H. Kromann, Steffen Helqvist, Klaus Bendtzen, Jens Høiriis Nielsen and Jens Mølvig. Their work appears in journals such as Acta Ophthalmologica, Diabetologia, Allergy, Diabetes and European Journal of Endocrinology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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