Irma Heid
Impact in
- Immunology top 10%
- Immunotherapy and Immune Responses
- Immune Cell Function and Interaction
- Genetics top 10%
- Virus-based gene therapy research
- Glioma Diagnosis and Treatment
Papers in
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- TGF-β signaling in diseases 2
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- Herpesvirus Infections and Treatments 6
- Cytomegalovirus and herpesvirus research 1
- Co-authors
- Stefan Bodmer (3 shared papers)A. Fontana (3 shared papers)Karl Frei (1 shared paper)Nicolas de Tribolet (1 shared paper)Christine Siepl (1 shared paper)Cornel Fraefel (7 shared papers)Mathias Ackermann (5 shared papers)Daniel L. Glauser (3 shared papers)
- Journals
- Journal of Virology (4 papers)Molecular Therapy (1 paper)The Journal of Immunology (1 paper)Biochemical and Biophysical Research Communications (1 paper)Journal of Neuroimmunology (1 paper)
- Partner nations
- SwitzerlandUnited StatesSlovakia
In The Last Decade
Irma Heid
10 papers receiving 666 citations
Peers
Comparison fields: 5 of 61
- Immunology 240
- Genetics 93
- Immunology and Allergy 47
- Oncology 191
- Genetics 171
Countries citing papers authored by Irma Heid
This map shows the geographic impact of Irma Heid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Irma Heid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Irma Heid more than expected).
Fields of papers citing papers by Irma Heid
This network shows the impact of papers produced by Irma Heid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Irma Heid. The network helps show where Irma Heid may publish in the future.
Co-authors
The 24 scholars most cited alongside Irma Heid, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1989 | 334 | |
| 2 | 2005 | 86 | |
| 3 | 2005 | 57 | |
| 4 | 2002 | 51 | |
| 5 | 1990 | 49 | |
| 6 | 1991 | 36 | |
| 7 | 2003 | 34 | |
| 8 | 2004 | 18 | |
| 9 | 2005 | 15 | |
| 10 | 2004 | 3 |
About Irma Heid
Irma Heid is a scholar working on Molecular Biology, Epidemiology, Genetics, Immunology and Infectious Diseases, having authored 10 papers that have together received 683 indexed citations. Recurring topics across this work include Virus-based gene therapy research (6 papers), Herpesvirus Infections and Treatments (6 papers), TGF-β signaling in diseases (2 papers), interferon and immune responses (1 paper), Cytomegalovirus and herpesvirus research (1 paper), Alzheimer's disease research and treatments (1 paper), Poxvirus research and outbreaks (1 paper) and Toxin Mechanisms and Immunotoxins (1 paper). The work is most often cited by research in Immunology (240 citations), Genetics (93 citations), Immunology and Allergy (47 citations), Oncology (191 citations) and Genetics (171 citations). Irma Heid has collaborated with scholars based in Switzerland, United States and Slovakia. Frequent co-authors include Stefan Bodmer, A. Fontana, Karl Frei, Nicolas de Tribolet, Christine Siepl, Cornel Fraefel, Mathias Ackermann, Daniel L. Glauser, Okay Saydam and Peter J. Wild. Their work appears in journals such as Journal of Virology, Molecular Therapy, The Journal of Immunology, Biochemical and Biophysical Research Communications and Journal of Neuroimmunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.