Ioana Inta

770 total citations
17 papers, 645 citations indexed

About

Ioana Inta is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Ioana Inta has authored 17 papers receiving a total of 645 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 4 papers in Neurology and 4 papers in Genetics. Recurrent topics in Ioana Inta's work include Sexual Differentiation and Disorders (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers). Ioana Inta is often cited by papers focused on Sexual Differentiation and Disorders (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers). Ioana Inta collaborates with scholars based in Germany, United States and Italy. Ioana Inta's co-authors include Markus Schwaninger, Oliver Herrmann, Mathias Zink, Alexandre Urani, Peter Gass, Carles Sanchis‐Segura, Sabine Chourbaji, Christiane Brandwein, Ilenia Sarnico and Marina Pizzi and has published in prestigious journals such as Journal of Neuroscience, Journal of Neurochemistry and Behavioural Brain Research.

In The Last Decade

Ioana Inta

17 papers receiving 634 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ioana Inta Germany 12 256 183 159 142 132 17 645
Zhao‐Lan Hu China 15 228 0.9× 134 0.7× 136 0.9× 162 1.1× 64 0.5× 29 789
Krzysztof Kolmus Belgium 8 215 0.8× 171 0.9× 99 0.6× 71 0.5× 58 0.4× 11 670
Anneleen Spooren Belgium 8 202 0.8× 208 1.1× 90 0.6× 62 0.4× 61 0.5× 8 662
Ammar Kutiyanawalla United States 12 221 0.9× 82 0.4× 69 0.4× 75 0.5× 59 0.4× 13 570
Lisa I. Greene United States 6 325 1.3× 96 0.5× 161 1.0× 49 0.3× 113 0.9× 6 746
Eva M. Medina‐Rodríguez United States 14 240 0.9× 95 0.5× 237 1.5× 147 1.0× 27 0.2× 20 580
Ariel Caviedes Chile 6 280 1.1× 66 0.4× 99 0.6× 60 0.4× 126 1.0× 9 491
Nicole Hanley Ireland 4 259 1.0× 335 1.8× 87 0.5× 34 0.2× 86 0.7× 7 694
Anders Ericsson‐Dahlstrand Sweden 13 206 0.8× 279 1.5× 61 0.4× 105 0.7× 35 0.3× 17 1.0k
Nobutaka Doe Japan 15 190 0.7× 172 0.9× 51 0.3× 38 0.3× 38 0.3× 28 503

Countries citing papers authored by Ioana Inta

Since Specialization
Citations

This map shows the geographic impact of Ioana Inta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ioana Inta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ioana Inta more than expected).

Fields of papers citing papers by Ioana Inta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ioana Inta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ioana Inta. The network helps show where Ioana Inta may publish in the future.

Co-authorship network of co-authors of Ioana Inta

This figure shows the co-authorship network connecting the top 25 collaborators of Ioana Inta. A scholar is included among the top collaborators of Ioana Inta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ioana Inta. Ioana Inta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
2.
Inta, Ioana, et al.. (2019). Typical characteristics of children with congenital adrenal hyperplasia due to 11β-hydroxylase deficiency: a single-centre experience and review of the literature. Journal of Pediatric Endocrinology and Metabolism. 32(3). 259–267. 9 indexed citations
3.
Choukair, Daniela, et al.. (2018). Clinical Features and Response to Treatment of Prolactinomas in Children and Adolescents: A Retrospective Single-Centre Analysis and Review of the Literature. Hormone Research in Paediatrics. 89(3). 157–165. 21 indexed citations
4.
Inta, Ioana, Georg F. Hoffmann, & Markus Bettendorf. (2017). Pediatric use of tetracyclines: focus on neurodevelopmental effects. Pediatric Research. 82(5). 725–726. 4 indexed citations
5.
Inta, Ioana, Natascha Pfeiffer, Rolf Sprengel, et al.. (2016). Puberty marks major changes in the hippocampal and cortical c-Fos activation pattern induced by NMDA receptor antagonists. Neuropharmacology. 112(Pt A). 181–187. 4 indexed citations
6.
Inta, Ioana, Markus Bettendorf, & P. Gass. (2016). Conserved Hypothalamic c-Fos Activation Pattern Induced by the mGlu5 Receptor Antagonist MPEP during Peri-pubertal Development in Mice. Pharmacopsychiatry. 49(4). 142–145. 1 indexed citations
7.
Vogt, Miriam A., Anne Stephanie Mallien, Natascha Pfeiffer, et al.. (2015). Minocycline does not evoke anxiolytic and antidepressant-like effects in C57BL/6 mice. Behavioural Brain Research. 301. 96–101. 28 indexed citations
8.
Inta, Ioana, et al.. (2015). Minocycline exacerbates apoptotic neurodegeneration induced by the NMDA receptor antagonist MK-801 in the early postnatal mouse brain. European Archives of Psychiatry and Clinical Neuroscience. 266(7). 673–677. 13 indexed citations
9.
Inta, Ioana, et al.. (2014). Analyses of Gonadoblastoma Y (GBY)-locus and of Y Centromere in Turner Syndrome Patients. Experimental and Clinical Endocrinology & Diabetes. 123(1). 61–65. 16 indexed citations
10.
Bettendorf, Markus, Ioana Inta, H. G. Doerr, et al.. (2013). Height Gain in Ullrich-Turner Syndrome after Early and Late Growth Hormone Treatment Start: Results from a Large Retrospective German Study and Potential Basis for an Individualized Treatment Approach. Hormone Research in Paediatrics. 80(5). 356–362. 12 indexed citations
11.
Inta, Ioana, Daniela Weber, Roland Veltkamp, et al.. (2009). Correlation of soluble gp130 serum concentrations with arterial blood pressure. Journal of Hypertension. 27(3). 527–534. 11 indexed citations
12.
Sarnico, Ilenia, Annamaria Lanzillotta, F. Boroni, et al.. (2008). NF‐κB p50/RelA and c‐Rel‐containing dimers: opposite regulators of neuron vulnerability to ischaemia. Journal of Neurochemistry. 108(2). 475–485. 90 indexed citations
13.
Inta, Ioana, Katrin Frauenknecht, Patricia Kohlhof, et al.. (2008). Induction of the cytokine TWEAK and its receptor Fn14 in ischemic stroke. Journal of the Neurological Sciences. 275(1-2). 117–120. 51 indexed citations
14.
Jüttler, Eric, Ioana Inta, Oliver Herrmann, et al.. (2007). Neuronal NF-κB influences thermoregulation and survival in a sepsis model. Journal of Neuroimmunology. 189(1-2). 41–49. 13 indexed citations
15.
Chourbaji, Sabine, Alexandre Urani, Ioana Inta, et al.. (2006). IL-6 knockout mice exhibit resistance to stress-induced development of depression-like behaviors. Neurobiology of Disease. 23(3). 587–594. 195 indexed citations
16.
Inta, Ioana, Stephan Paxian, Ira Maegele, et al.. (2006). Bim and Noxa Are Candidates to Mediate the Deleterious Effect of the NF-κB Subunit RelA in Cerebral Ischemia. Journal of Neuroscience. 26(50). 12896–12903. 126 indexed citations
17.
Schwaninger, Markus, Ioana Inta, & Oliver Herrmann. (2006). NF-κB signalling in cerebral ischaemia. Biochemical Society Transactions. 34(6). 1291–1294. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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