Anders Ericsson‐Dahlstrand
- Neurology top 5%
- Behavioral Neuroscience top 5%
- Stress Responses and Cortisol 2
- Biological Psychiatry top 10%
- Immunology top 10%
- Immune Response and Inflammation 4
- Immunotherapy and Immune Responses 3
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- Receptor Mechanisms and Signaling 5
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- Inflammatory mediators and NSAID effects 5
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- Neuropeptides and Animal Physiology 5
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- Chemokine receptors and signaling 4
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- Multiple Sclerosis Research Studies 3
Anders Ericsson‐Dahlstrand
17 papers receiving 987 citations
Peers
Comparison fields: 5 of 86
- Neurology 279
- Behavioral Neuroscience 105
- Biological Psychiatry 61
- Endocrine and Autonomic Systems 155
- Immunology 292
Countries citing papers authored by Anders Ericsson‐Dahlstrand
This map shows the geographic impact of Anders Ericsson‐Dahlstrand's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anders Ericsson‐Dahlstrand with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anders Ericsson‐Dahlstrand more than expected).
Fields of papers citing papers by Anders Ericsson‐Dahlstrand
This network shows the impact of papers produced by Anders Ericsson‐Dahlstrand. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anders Ericsson‐Dahlstrand. The network helps show where Anders Ericsson‐Dahlstrand may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Anders Ericsson‐Dahlstrand, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 11 | |
| 2 | 2014 | 74 | |
| 3 | 2007 | 66 | |
| 4 | 2005 | 110 | |
| 5 | 2004 | 10 | |
| 6 | 2003 | 29 | |
| 7 | 2003 | 37 | |
| 8 | 2002 | 120 | |
| 9 | 2002 | 49 | |
| 10 | 2002 | 2 | |
| 11 | 2001 | 27 | |
| 12 | 2001 | 269 | |
| 13 | 2001 | 18 | |
| 14 | 2000 | 143 | |
| 15 | 2000 | 9 | |
| 16 | 2000 | 17 | |
| 17 | 1997 | 15 |
About Anders Ericsson‐Dahlstrand
Anders Ericsson‐Dahlstrand is a scholar working on Behavioral Neuroscience, Biological Psychiatry, Immunology, Endocrine and Autonomic Systems and Pharmacology, having authored 17 papers that have together received 1.0k indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (5 papers), Inflammatory mediators and NSAID effects (5 papers), Neuropeptides and Animal Physiology (5 papers), Immune Response and Inflammation (4 papers), Chemokine receptors and signaling (4 papers), Immunotherapy and Immune Responses (3 papers), Multiple Sclerosis Research Studies (3 papers) and Stress Responses and Cortisol (2 papers). The work is most often cited by research in Neurology (279 citations), Behavioral Neuroscience (105 citations), Biological Psychiatry (61 citations), Endocrine and Autonomic Systems (155 citations) and Immunology (292 citations). Anders Ericsson‐Dahlstrand has collaborated with scholars based in Sweden, United Kingdom and Austria. Frequent co-authors include Monica Ek, David Engblom, Anders Blomqvist, Per‐Johan Jakobsson, Sipra Saha, Paul E. Sawchenko, Carlos Arias, Dan Sunnemark, Anna‐Lena Berg and Erik Wallström. Their work appears in journals such as The Journal of Comparative Neurology, Journal of Neuroinflammation, Neuroreport, Journal of Neuroimmunology and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.