Ingo Rustenbeck

1.6k total citations
93 papers, 1.3k citations indexed

About

Ingo Rustenbeck is a scholar working on Surgery, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Ingo Rustenbeck has authored 93 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 63 papers in Surgery, 51 papers in Molecular Biology and 27 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Ingo Rustenbeck's work include Pancreatic function and diabetes (63 papers), Diabetes Management and Research (19 papers) and Metabolism, Diabetes, and Cancer (17 papers). Ingo Rustenbeck is often cited by papers focused on Pancreatic function and diabetes (63 papers), Diabetes Management and Research (19 papers) and Metabolism, Diabetes, and Cancer (17 papers). Ingo Rustenbeck collaborates with scholars based in Germany, Czechia and Egypt. Ingo Rustenbeck's co-authors include Sigurd Lenzen, Thomas Grimmsmann, U. Panten, Michael Willenborg, Anne Jörns, Torben Schulze, Bernd J. Zünkler, Alexander Matthies, A. Hasselblatt and Peter Ratzka and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and Diabetes.

In The Last Decade

Ingo Rustenbeck

91 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ingo Rustenbeck Germany 21 743 643 306 198 185 93 1.3k
Peter Krippeit‐Drews Germany 23 772 1.0× 883 1.4× 442 1.4× 350 1.8× 174 0.9× 61 1.8k
Irene L. Ennis Argentina 30 1.4k 1.9× 197 0.3× 262 0.9× 327 1.7× 91 0.5× 76 2.2k
Masaya Sakamoto Japan 20 612 0.8× 258 0.4× 519 1.7× 221 1.1× 114 0.6× 55 1.8k
Mourad Ferdaoussi Canada 22 714 1.0× 710 1.1× 605 2.0× 328 1.7× 138 0.7× 43 1.7k
Paulus Wohlfart Germany 23 665 0.9× 190 0.3× 377 1.2× 577 2.9× 75 0.4× 49 1.9k
Jacqueline Hoerter France 24 997 1.3× 214 0.3× 163 0.5× 343 1.7× 56 0.3× 48 1.7k
K. Geisen Germany 16 456 0.6× 218 0.3× 426 1.4× 206 1.0× 81 0.4× 37 1.3k
Cécile Cottet‐Rousselle France 18 684 0.9× 193 0.3× 121 0.4× 233 1.2× 44 0.2× 36 1.2k
Réka Skoumal Hungary 16 427 0.6× 291 0.5× 131 0.4× 112 0.6× 290 1.6× 28 1.1k
Hiroya Hidaka Japan 20 410 0.6× 245 0.4× 143 0.5× 392 2.0× 67 0.4× 51 977

Countries citing papers authored by Ingo Rustenbeck

Since Specialization
Citations

This map shows the geographic impact of Ingo Rustenbeck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ingo Rustenbeck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ingo Rustenbeck more than expected).

Fields of papers citing papers by Ingo Rustenbeck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ingo Rustenbeck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ingo Rustenbeck. The network helps show where Ingo Rustenbeck may publish in the future.

Co-authorship network of co-authors of Ingo Rustenbeck

This figure shows the co-authorship network connecting the top 25 collaborators of Ingo Rustenbeck. A scholar is included among the top collaborators of Ingo Rustenbeck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ingo Rustenbeck. Ingo Rustenbeck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rustenbeck, Ingo, et al.. (2025). The Amount of Releasable Insulin Depends on Continuous Oxidative Phosphorylation. Function. 6(5). 1 indexed citations
2.
Rustenbeck, Ingo, et al.. (2023). PyCreas: a tool for quantification of localization and distribution of endocrine cell types in the islets of Langerhans. Frontiers in Endocrinology. 14. 1250023–1250023. 2 indexed citations
3.
Schulze, Torben, et al.. (2023). Short-Term Inhibition of Translation by Cycloheximide Concurrently Affects Mitochondrial Function and Insulin Secretion in Islets from Female Mice. International Journal of Molecular Sciences. 24(20). 15464–15464. 1 indexed citations
4.
Schulze, Torben, et al.. (2021). A Parallel Perifusion Slide From Glass for the Functional and Morphological Analysis of Pancreatic Islets. Frontiers in Bioengineering and Biotechnology. 9. 615639–615639. 5 indexed citations
5.
Schulze, Torben, et al.. (2017). A 3D microfluidic perfusion system made from glass for multiparametric analysis of stimulus-secretioncoupling in pancreatic islets. Biomedical Microdevices. 19(3). 47–47. 38 indexed citations
6.
Panten, U., et al.. (2016). Acute metabolic amplification of insulin secretion in mouse islets: Role of cytosolic acetyl-CoA. Metabolism. 65(9). 1225–1229. 12 indexed citations
7.
8.
Lorenz, Dirk A., et al.. (2013). Observer-Independent Quantification of Insulin Granule Exocytosis and Pre-Exocytotic Mobility by TIRF Microscopy. Microscopy and Microanalysis. 20(1). 206–218. 6 indexed citations
9.
Jörns, Anne, et al.. (2013). Effect of fluoroquinolones on mitochondrial function in pancreatic beta cells. European Journal of Pharmaceutical Sciences. 52. 206–214. 15 indexed citations
10.
11.
Willenborg, Michael, et al.. (2010). The signalling role of action potential depolarization in insulin secretion. Biochemical Pharmacology. 80(1). 104–112. 7 indexed citations
12.
Willenborg, Michael, U. Panten, & Ingo Rustenbeck. (2009). Triggering and amplification of insulin secretion by dimethyl α-ketoglutarate, a membrane permeable α-ketoglutarate analogue. European Journal of Pharmacology. 607(1-3). 41–46. 13 indexed citations
13.
Zünkler, Bernd J., et al.. (2006). Effects of fluoroquinolones on HERG channels and on pancreatic β-cell ATP-sensitive K+ channels. Toxicology. 228(2-3). 239–248. 24 indexed citations
14.
Rustenbeck, Ingo & Sigurd Lenzen. (2004). Effects of lysophosphatidylcholine and arachidonic acid on the regulation of intracellular Ca2+ transport. Naunyn-Schmiedeberg s Archives of Pharmacology. 339-339(1-2). 37–41. 1 indexed citations
15.
Zünkler, Bernd J., et al.. (2003). Specificity of Nonadrenergic Imidazoline Binding Sites in Insulin‐Secreting Cells and Relation to the Block of ATP‐Sensitive K1 Channels. Annals of the New York Academy of Sciences. 1009(1). 371–377. 11 indexed citations
16.
Rustenbeck, Ingo. (2002). Desensitization of insulin secretion. Biochemical Pharmacology. 63(11). 1921–1935. 54 indexed citations
17.
Rustenbeck, Ingo, et al.. (2001). Desensitization of insulin secretory response to imidazolines, tolbutamide, and quinine. Biochemical Pharmacology. 62(12). 1685–1694. 11 indexed citations
18.
Rustenbeck, Ingo, et al.. (1998). Polyamine modulation of mitochondrial calcium transport. Biochemical Pharmacology. 56(8). 977–985. 37 indexed citations
19.
Rustenbeck, Ingo, et al.. (1998). Polyamine modulation of mitochondrial calcium transport. Biochemical Pharmacology. 56(8). 987–995. 24 indexed citations
20.
Grimmsmann, Thomas & Ingo Rustenbeck. (1998). Direct effects of diazoxide on mitochondria in pancreatic B‐cells and on isolated liver mitochondria. British Journal of Pharmacology. 123(5). 781–788. 110 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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