Imma Castaldo
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- Genetic Neurodegenerative Diseases 22
- Hereditary Neurological Disorders 3
- Neurology top 5%
- Neurological diseases and metabolism 3
- Neurological disorders and treatments 3
- Molecular Biology top 10%
- Mitochondrial Function and Pathology 15
- DNA Repair Mechanisms 3
- Ubiquitin and proteasome pathways 3
- Prion Diseases and Protein Misfolding 1
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- Neurological diseases and metabolism 3
- Neurological disorders and treatments 3
- Clinical Biochemistry top 10%
- Co-authors
- Alessandro FillaAntonella MonticelliGiuseppe De MicheleSergio CocozzaOlga CalabreseMichele PinelliFabio AcquavivaEnrico V. Avvedimento
- Partner nations
- ItalyUnited StatesSweden
In The Last Decade
Imma Castaldo
27 papers receiving 949 citations
Peers
Comparison fields: 5 of 61
- Cellular and Molecular Neuroscience 723
- Neurology 307
- Molecular Biology 820
- Neurology 55
- Clinical Biochemistry 44
Countries citing papers authored by Imma Castaldo
This map shows the geographic impact of Imma Castaldo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Imma Castaldo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Imma Castaldo more than expected).
Fields of papers citing papers by Imma Castaldo
This network shows the impact of papers produced by Imma Castaldo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Imma Castaldo. The network helps show where Imma Castaldo may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Imma Castaldo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 5 | |
| 2 | 2012 | 26 | |
| 3 | 2011 | 8 | |
| 4 | 2008 | 82 | |
| 5 | 2008 | 50 | |
| 6 | 2008 | 25 | |
| 7 | 2005 | 131 | |
| 8 | 2005 | 6 | |
| 9 | 2005 | 15 | |
| 10 | 2004 | 43 | |
| 11 | 2002 | 20 | |
| 12 | 2001 | 11 | |
| 13 | 2000 | 49 | |
| 14 | 1999 | 48 | |
| 15 | 1998 | 14 | |
| 16 | 1997 | 25 | |
| 17 | 1996 | 28 | |
| 18 | 1995 | 67 | |
| 19 | 1995 | 38 | |
| 20 | 1994 | 4 |
About Imma Castaldo
Imma Castaldo is a scholar working on Cellular and Molecular Neuroscience, Neurology and Neurology, having authored 27 papers that have together received 973 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (22 papers), Mitochondrial Function and Pathology (15 papers), Neurological diseases and metabolism (3 papers), DNA Repair Mechanisms (3 papers), Neurological disorders and treatments (3 papers), Hereditary Neurological Disorders (3 papers), Ubiquitin and proteasome pathways (3 papers) and Prion Diseases and Protein Misfolding (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (723 citations), Neurology (307 citations) and Molecular Biology (820 citations). Imma Castaldo has collaborated with scholars based in Italy, United States and Sweden. Frequent co-authors include Alessandro Filla, Antonella Monticelli, Giuseppe De Michele, Sergio Cocozza, Olga Calabrese, Michele Pinelli, Alessandro Filla, Fabio Acquaviva, Enrico V. Avvedimento and G Campanella. Their work appears in journals such as Neurology, Diabetes Care and Annals of Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.