Ílknur Özen

1.1k total citations
20 papers, 846 citations indexed

About

Ílknur Özen is a scholar working on Neurology, Neurology and Molecular Biology. According to data from OpenAlex, Ílknur Özen has authored 20 papers receiving a total of 846 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Neurology, 8 papers in Neurology and 6 papers in Molecular Biology. Recurrent topics in Ílknur Özen's work include Barrier Structure and Function Studies (8 papers), Traumatic Brain Injury and Neurovascular Disturbances (7 papers) and Neurogenesis and neuroplasticity mechanisms (6 papers). Ílknur Özen is often cited by papers focused on Barrier Structure and Function Studies (8 papers), Traumatic Brain Injury and Neurovascular Disturbances (7 papers) and Neurogenesis and neuroplasticity mechanisms (6 papers). Ílknur Özen collaborates with scholars based in Sweden, United States and United Kingdom. Ílknur Özen's co-authors include Gesine Paul, Thomas Padel, Abderahim Gaceb, Marco Barbariga, Guillem Genové, Tomas Deierborg, Kenichi Miharada, Elisabet Englund, Jordi Boix and Michaela Roth and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Stroke.

In The Last Decade

Ílknur Özen

19 papers receiving 836 citations

Peers

Ílknur Özen
Anna C. Liang United States
Dritan Agalliu United States
Patrick Ventura United States
Yvette Zarb Switzerland
Khayrun Nahar Switzerland
Wayne Tsang United States
Ílknur Özen
Citations per year, relative to Ílknur Özen Ílknur Özen (= 1×) peers Ana Rita Pombo Antunes

Countries citing papers authored by Ílknur Özen

Since Specialization
Citations

This map shows the geographic impact of Ílknur Özen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ílknur Özen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ílknur Özen more than expected).

Fields of papers citing papers by Ílknur Özen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ílknur Özen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ílknur Özen. The network helps show where Ílknur Özen may publish in the future.

Co-authorship network of co-authors of Ílknur Özen

This figure shows the co-authorship network connecting the top 25 collaborators of Ílknur Özen. A scholar is included among the top collaborators of Ílknur Özen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ílknur Özen. Ílknur Özen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ruscher, Karsten, Sami Abu Hamdeh, Fredrik Clausen, et al.. (2025). Persistent increase of Nogo-A-positive cells and dynamic reduction in oligodendroglia lineage cells in white matter regions following experimental and clinical traumatic brain injury. Journal of Neuropathology & Experimental Neurology. 84(5). 423–435.
2.
Özen, Ílknur, Sami Abu Hamdeh, Karsten Ruscher, & Niklas Marklund. (2025). Traumatic brain injury causes early aggregation of beta-amyloid peptides and NOTCH3 reduction in vascular smooth muscle cells of leptomeningeal arteries. Acta Neuropathologica. 149(1). 10–10. 4 indexed citations
3.
Özen, Ílknur, Fredrik Clausen, Johanna Flygt, Niklas Marklund, & Gesine Paul. (2024). Neutralization of Interleukin 1-beta is associated with preservation of thalamic capillaries after experimental traumatic brain injury. Frontiers in Neurology. 15. 1378203–1378203. 2 indexed citations
4.
Clausen, Fredrik, et al.. (2024). Impaired oligodendrogenesis in the white matter of aged mice following diffuse traumatic brain injury. Glia. 72(4). 728–747. 8 indexed citations
6.
Özen, Ílknur, et al.. (2022). Diffuse Traumatic Injury in the Mouse Disrupts Axon-Myelin Integrity in the Cerebellum. Journal of Neurotrauma. 39(5-6). 411–422. 12 indexed citations
7.
Özen, Ílknur, Hongcheng Mai, Karsten Ruscher, et al.. (2022). Purkinje cell vulnerability induced by diffuse traumatic brain injury is linked to disruption of long-range neuronal circuits. Acta Neuropathologica Communications. 10(1). 129–129. 6 indexed citations
8.
Durrant, Claire S., Karsten Ruscher, Olivia Sheppard, Michael P. Coleman, & Ílknur Özen. (2020). Beta secretase 1-dependent amyloid precursor protein processing promotes excessive vascular sprouting through NOTCH3 signalling. Cell Death and Disease. 11(2). 98–98. 28 indexed citations
9.
Özen, Ílknur, et al.. (2020). Interleukin-1 Beta Neutralization Attenuates Traumatic Brain Injury-Induced Microglia Activation and Neuronal Changes in the Globus Pallidus. International Journal of Molecular Sciences. 21(2). 387–387. 37 indexed citations
10.
Roth, Michaela, Abderahim Gaceb, Thomas Padel, et al.. (2019). Regulator of G‐protein signaling 5 regulates the shift from perivascular to parenchymal pericytes in the chronic phase after stroke. The FASEB Journal. 33(8). 8990–8998. 26 indexed citations
11.
Gaceb, Abderahim, Marco Barbariga, Ílknur Özen, & Gesine Paul. (2018). The pericyte secretome: Potential impact on regeneration. Biochimie. 155. 16–25. 46 indexed citations
12.
Özen, Ílknur, Michaela Roth, Marco Barbariga, et al.. (2018). Loss of Regulator of G-Protein Signaling 5 Leads to Neurovascular Protection in Stroke. Stroke. 49(9). 2182–2190. 48 indexed citations
13.
Carlsson, R., Ílknur Özen, Marco Barbariga, et al.. (2018). STAT3 precedes HIF1α transcriptional responses to oxygen and oxygen and glucose deprivation in human brain pericytes. PLoS ONE. 13(3). e0194146–e0194146. 22 indexed citations
14.
Gaceb, Abderahim, Ílknur Özen, Thomas Padel, Marco Barbariga, & Gesine Paul. (2017). Pericytes secrete pro-regenerative molecules in response to platelet-derived growth factor-BB. Journal of Cerebral Blood Flow & Metabolism. 38(1). 45–57. 93 indexed citations
15.
Padel, Thomas, Ílknur Özen, Jordi Boix, et al.. (2016). Platelet-derived growth factor-BB has neurorestorative effects and modulates the pericyte response in a partial 6-hydroxydopamine lesion mouse model of Parkinson's disease. Neurobiology of Disease. 94. 95–105. 47 indexed citations
16.
Svensson, Andreas, Ílknur Özen, Guillem Genové, Gesine Paul, & Johan Bengzon. (2015). Endogenous Brain Pericytes Are Widely Activated and Contribute to Mouse Glioma Microvasculature. PLoS ONE. 10(4). e0123553–e0123553. 43 indexed citations
17.
Özen, Ílknur, Tomas Deierborg, Kenichi Miharada, et al.. (2014). Brain pericytes acquire a microglial phenotype after stroke. Acta Neuropathologica. 128(3). 381–396. 159 indexed citations
18.
Paul, Gesine, Ílknur Özen, Nicolaj S. Christophersen, et al.. (2012). The Adult Human Brain Harbors Multipotent Perivascular Mesenchymal Stem Cells. PLoS ONE. 7(4). e35577–e35577. 161 indexed citations
19.
Özen, Ílknur, Jordi Boix, & Gesine Paul. (2012). Perivascular mesenchymal stem cells in the adult human brain: a future target for neuroregeneration?. SHILAP Revista de lepidopterología. 1(1). 30–30. 42 indexed citations
20.
Özen, Ílknur, et al.. (2001). N-Cadherin Is Involved in Axon-Oligodendrocyte Contact and Myelination. Molecular and Cellular Neuroscience. 17(6). 1084–1093. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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