Ikuko Miyazaki

5.6k total citations
131 papers, 4.7k citations indexed

About

Ikuko Miyazaki is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, Ikuko Miyazaki has authored 131 papers receiving a total of 4.7k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Cellular and Molecular Neuroscience, 35 papers in Neurology and 30 papers in Molecular Biology. Recurrent topics in Ikuko Miyazaki's work include Parkinson's Disease Mechanisms and Treatments (32 papers), Neuroscience and Neuropharmacology Research (26 papers) and Neuroinflammation and Neurodegeneration Mechanisms (16 papers). Ikuko Miyazaki is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (32 papers), Neuroscience and Neuropharmacology Research (26 papers) and Neuroinflammation and Neurodegeneration Mechanisms (16 papers). Ikuko Miyazaki collaborates with scholars based in Japan, United States and Bulgaria. Ikuko Miyazaki's co-authors include Masato Asanuma, Norio Ogawa, Ko Miyoshi, Francisco J. Diaz‐Corrales, Youichirou Higashi, Ken-ichi Tanaka, Shinki Murakami, Mika Takeshima, Yoshihisa Kitamura and Taizo Kita and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and PLoS ONE.

In The Last Decade

Ikuko Miyazaki

126 papers receiving 4.6k citations

Peers

Countries citing papers authored by Ikuko Miyazaki

Since Specialization

Citations

This map shows the geographic impact of Ikuko Miyazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ikuko Miyazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ikuko Miyazaki more than expected).

Fields of papers citing papers by Ikuko Miyazaki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ikuko Miyazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ikuko Miyazaki. The network helps show where Ikuko Miyazaki may publish in the future.

Co-authorship network of co-authors of Ikuko Miyazaki

This figure shows the co-authorship network connecting the top 25 collaborators of Ikuko Miyazaki. A scholar is included among the top collaborators of Ikuko Miyazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ikuko Miyazaki. Ikuko Miyazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor 2025 ·NeuroSci ·Ikuko Miyazaki, (unknown), Tomohisa Nagoshi, (unknown), (unknown), (unknown), (unknown), Kazuhiko Tomimoto, Kaori Masai, (unknown), Masato Asanuma	0
2	Differentially Expressed Nedd4-binding Protein Ndfip1 Protects Neurons Against Methamphetamine-induced Neurotoxicity 2025 ·Neurotoxicity Research ·(unknown), Ikuko Miyazaki, Jean Lud Cadet	1
3	Phosphorylated SARM1 is involved in the pathological process of rotenone-induced neurodegeneration 2023 ·The Journal of Biochemistry ·Hitoshi Murata, (unknown), Toshiki Ochi, Nahoko Tomonobu, Kenichi Yamamoto, Rie Kinoshita, Ikuko Miyazaki, Masahiro Nishibori, Masato Asanuma, Masakiyo Sakaguchi	5
4	Multifunctional Metallothioneins as a Target for Neuroprotection in Parkinson’s Disease 2023 ·Antioxidants ·Ikuko Miyazaki, Masato Asanuma	15
5	Glutathione and Related Molecules in Parkinsonism 2021 ·International Journal of Molecular Sciences ·Masato Asanuma, Ikuko Miyazaki	51
6	Cerebellar Blood Flow and Gene Expression in Crossed Cerebellar Diaschisis after Transient Middle Cerebral Artery Occlusion in Rats 2020 ·International Journal of Molecular Sciences ·(unknown), Tomohito Hishikawa, Masafumi Hiramatsu, (unknown), Kyohei Kin, (unknown), Satoshi Murai, Kenji Sugiu, Takao Yasuhara, Ikuko Miyazaki, Masato Asanuma, Isao Date	9
7	The Rotenone Models Reproducing Central and Peripheral Features of Parkinson’s Disease 2020 ·NeuroSci ·Ikuko Miyazaki, Masato Asanuma	28
8	Chronic Systemic Exposure to Low-Dose Rotenone Induced Central and Peripheral Neuropathology and Motor Deficits in Mice: Reproducible Animal Model of Parkinson’s Disease 2020 ·International Journal of Molecular Sciences ·Ikuko Miyazaki, (unknown), (unknown), (unknown), (unknown), (unknown), Masato Asanuma	76
9	Neuron-Astrocyte Interactions in Parkinson’s Disease 2020 ·Cells ·Ikuko Miyazaki, Masato Asanuma	118
10	Effects of Enteric Environmental Modification by Coffee Components on Neurodegeneration in Rotenone-Treated Mice 2019 ·Cells ·Ikuko Miyazaki, (unknown), Kouichi Wada, (unknown), Yoshihisa Kitamura, Masato Asanuma	39
11	Lack of Dopaminergic Inputs Elongates the Primary Cilia of Striatal Neurons 2014 ·PLoS ONE ·Ko Miyoshi, (unknown), Shinki Murakami, Mika Takeshima, Natsuko Kumamoto, (unknown), Ikuko Miyazaki, Shinsuke Matsuzaki, Toshikuni Sasaoka, Taiichi Katayama, Masato Asanuma	39
12	Transplantation of Melanocytes Obtained from the Skin Ameliorates Apomorphine-Induced Abnormal Behavior in Rodent Hemi-Parkinsonian Models 2013 ·PLoS ONE ·Masato Asanuma, Ikuko Miyazaki, Francisco J. Diaz‐Corrales, Youichirou Higashi, Masayoshi Namba, Norio Ogawa	5
13	Biological roles for neuronal primary cilia 2008 ·Psychiatry and Clinical Neurosciences ·Masato Asanuma, Ikuko Miyazaki, Ko Miyoshi	1
14	Specific Gene Expression and Possible Involvement of Inflammation in Methamphetamine‐Induced Neurotoxicity 2004 ·Annals of the New York Academy of Sciences ·Masato Asanuma, Ikuko Miyazaki, Youichirou Higashi, Takeshi Tsuji, Norio Ogawa	50
15	Direct evidence for expression of dopamine receptors in astrocytes from basal ganglia 2004 ·Brain Research ·Ikuko Miyazaki, Masato Asanuma, Francisco J. Diaz‐Corrales, Ko Miyoshi, Norio Ogawa	115
16	Methamphetamine-induced increase in striatal p53 DNA-binding activity is attenuated in Cu,Zn-superoxide dismutase transgenic mice 2002 ·Neuroscience Letters ·Masato Asanuma, Ikuko Miyazaki, Youichirou Higashi, Jean Lud Cadet, Norio Ogawa	17
17	Apoptosis-inducing neurotoxicity of dopamine and its metabolites via reactive quinone generation in neuroblastoma cells 2002 ·Biochimica et Biophysica Acta (BBA) - General Subjects ·Md. Emdadul Haque, Masato Asanuma, Youichirou Higashi, Ikuko Miyazaki, Ken-ichi Tanaka, Norio Ogawa	136
18	GPI1046 prevents dopaminergic dysfunction by activating glutathione system in the mouse striatum 2002 ·Neuroscience Letters ·Ken-ichi Tanaka, (unknown), Ikuko Miyazaki, Naoko Fujita, Norio Ogawa	23
19	Antioxidants protect against dopamine-induced metallothionein-III (GIF) mRNA expression in mouse glial cell line (VR-2g) 2000 ·Brain Research ·Chiharu Sogawa, Ikuko Miyazaki, Norio Sogawa, Masato Asanuma, Norio Ogawa, Hiroaki Furuta	8
20	ANIRACETAM AMELIORATES IMPAIRED PRE- AND POST-SYNAPTIC CHOLINERGIC INDICES IN GERBIL HIPPOCAMPUS INDUCED BY TRANSIENT FOREBRAIN ISCHEMIA 1997 ·Yoichi Kondo, Masato Asanuma, Hideaki Kabuto, Emi Iwata, Fumio Kondo, Ikuko Miyazaki, Norio Ogawa	4

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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