Ikuko Maejima

2.1k total citations · 2 hit papers
9 papers, 1.6k citations indexed

About

Ikuko Maejima is a scholar working on Epidemiology, Cell Biology and Molecular Biology. According to data from OpenAlex, Ikuko Maejima has authored 9 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Epidemiology, 5 papers in Cell Biology and 3 papers in Molecular Biology. Recurrent topics in Ikuko Maejima's work include Autophagy in Disease and Therapy (5 papers), Cellular transport and secretion (4 papers) and Retinal Development and Disorders (2 papers). Ikuko Maejima is often cited by papers focused on Autophagy in Disease and Therapy (5 papers), Cellular transport and secretion (4 papers) and Retinal Development and Disorders (2 papers). Ikuko Maejima collaborates with scholars based in Japan, Argentina and Brazil. Ikuko Maejima's co-authors include Tamotsu Yoshimori, Tatsuya Saitoh, Takeshi Noda, Hiroko Omori, Keisuke Tabata, Toshiaki Isobe, Kanae Shirahama‐Noda, Kohichi Matsunaga, Tohru Ichimura and Shizuo Akira and has published in prestigious journals such as SHILAP Revista de lepidopterología, The EMBO Journal and Nature Cell Biology.

In The Last Decade

Ikuko Maejima

9 papers receiving 1.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages

2009 945 citations Kohichi Matsunaga, Tatsuya Saitoh et al. Nature Cell Biology profile →
Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury

2013 467 citations Ikuko Maejima, Atsushi Takahashi et al. The EMBO Journal profile →

Peers

Ikuko Maejima

EPIDE

MB

CB

PHYSI

Chieko Kishi‐Itakura United Kingdom

Alf Håkon Lystad Norway

Thanh Ngoc Nguyen Australia

Alison Forrester Italy

Alexis Rozenknop Germany

Tsuyoshi Kawabata Japan

Péter Lőrincz Hungary

Elvira Stefania Cannizzo Italy

Monica Fengsrud Norway

Chieko Kishi‐Itakura United Kingdom

Ikuko Maejima

1.5k ×1.2

EPIDE

751 ×1.4

MB

640 ×1.5

CB

324 ×1.0

PHYSI

269 ×1.3

PHYSI

Citations per year, relative to Ikuko Maejima Ikuko Maejima (= 1×) peers Chieko Kishi‐Itakura

Countries citing papers authored by Ikuko Maejima

Since Specialization

Citations

This map shows the geographic impact of Ikuko Maejima's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ikuko Maejima with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ikuko Maejima more than expected).

Fields of papers citing papers by Ikuko Maejima

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ikuko Maejima. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ikuko Maejima. The network helps show where Ikuko Maejima may publish in the future.

Co-authorship network of co-authors of Ikuko Maejima

This figure shows the co-authorship network connecting the top 25 collaborators of Ikuko Maejima. A scholar is included among the top collaborators of Ikuko Maejima based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ikuko Maejima. Ikuko Maejima is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Maejima, Ikuko & Ken Sato. (2024). New aspects of a small GTPase RAB35 in brain development and function. Neural Regeneration Research. 20(7). 1971–1980. 2 indexed citations

2.

Maejima, Ikuko, Taichi Hara, Satoshi Tsukamoto, et al.. (2023). RAB35 is required for murine hippocampal development and functions by regulating neuronal cell distribution. Communications Biology. 6(1). 440–440. 6 indexed citations

3.

Martins, Waleska K., Kiran Pandey, Ikuko Maejima, et al.. (2021). Autophagy-targeted therapy to modulate age-related diseases: Success, pitfalls, and new directions. SHILAP Revista de lepidopterología. 2. 100033–100033. 11 indexed citations

4.

Hara, Taichi, Ikuko Maejima, Hisae Kobayashi, et al.. (2018). Rer1-mediated quality control system is required for neural stem cell maintenance during cerebral cortex development. PLoS Genetics. 14(9). e1007647–e1007647. 12 indexed citations

5.

Takahashi, Atsushi, Yoshitsugu Takabatake, Tomonori Kimura, et al.. (2017). Autophagy Inhibits the Accumulation of Advanced Glycation End Products by Promoting Lysosomal Biogenesis and Function in the Kidney Proximal Tubules. Diabetes. 66(5). 1359–1372. 101 indexed citations

6.

Hasegawa, Junya, et al.. (2014). Selective autophagy: Lysophagy. Methods. 75. 128–132. 51 indexed citations

7.

Yamasaki, Akinori, Taichi Hara, Ikuko Maejima, et al.. (2014). Rer1p regulates the ER retention of immature rhodopsin and modulates its intracellular trafficking. Scientific Reports. 4(1). 5973–5973. 17 indexed citations

8.

Maejima, Ikuko, Atsushi Takahashi, Hiroko Omori, et al.. (2013). Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury. The EMBO Journal. 32(17). 2336–2347. 467 indexed citations breakdown →

9.

Matsunaga, Kohichi, Tatsuya Saitoh, Keisuke Tabata, et al.. (2009). Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages. Nature Cell Biology. 11(4). 385–396. 945 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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