Hiroko Omori

8.4k total citations · 6 hit papers
36 papers, 6.7k citations indexed

About

Hiroko Omori is a scholar working on Epidemiology, Public Health, Environmental and Occupational Health and Cell Biology. According to data from OpenAlex, Hiroko Omori has authored 36 papers receiving a total of 6.7k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Epidemiology, 10 papers in Public Health, Environmental and Occupational Health and 8 papers in Cell Biology. Recurrent topics in Hiroko Omori's work include Autophagy in Disease and Therapy (15 papers), Streptococcal Infections and Treatments (7 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Hiroko Omori is often cited by papers focused on Autophagy in Disease and Therapy (15 papers), Streptococcal Infections and Treatments (7 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Hiroko Omori collaborates with scholars based in Japan, United States and Taiwan. Hiroko Omori's co-authors include Tamotsu Yoshimori, Takeshi Noda, Naonobu Fujita, Tatsuya Saitoh, Shizuo Akira, Takashi Satoh, Naoki Yamamoto, Takashi Itoh, Mitsunori Fukuda and Osamu Takeuchi and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Hiroko Omori

34 papers receiving 6.6k citations

Hit Papers

Loss of the autophagy protein Atg16L1 enhances endotoxin-... 2008 2026 2014 2020 2008 2009 2008 2009 2012 500 1000 1.5k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1β production
    2008 1.7k citations Tatsuya Saitoh, Naonobu Fujita et al. profile →
  2. Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages
    2009 945 citations Kohichi Matsunaga, Tatsuya Saitoh et al. Nature Cell Biology profile →
  3. The Atg16L Complex Specifies the Site of LC3 Lipidation for Membrane Biogenesis in Autophagy
    2008 847 citations Naonobu Fujita, Takashi Itoh et al. Molecular Biology of the Cell profile →
  4. Atg9a controls dsDNA-driven dynamic translocation of STING and the innate immune response
    2009 675 citations Tatsuya Saitoh, Naonobu Fujita et al. Proceedings of the National Academy of Sciences profile →
  5. Neutrophil Extracellular Traps Mediate a Host Defense Response to Human Immunodeficiency Virus-1
    2012 557 citations Tatsuya Saitoh, Jun Komano et al. Cell Host & Microbe profile →
  6. Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury
    2013 467 citations Ikuko Maejima, Atsushi Takahashi et al. The EMBO Journal profile →

Peers

Hiroko Omori
Felix Randow United Kingdom
Rhea Sumpter United States
Jennifer Martinez United States
Mónica A. Delgado United States
Honglin Luo Canada
Lynda M. Stuart United States
Myoung Ho Jang South Korea
Seungmin Hwang United States
R. Chris Bleackley Canada
Frances E. Lund United States
Felix Randow United Kingdom
Hiroko Omori
Citations per year, relative to Hiroko Omori Hiroko Omori (= 1×) peers Felix Randow

Countries citing papers authored by Hiroko Omori

Since Specialization
Citations

This map shows the geographic impact of Hiroko Omori's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroko Omori with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroko Omori more than expected).

Fields of papers citing papers by Hiroko Omori

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroko Omori. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroko Omori. The network helps show where Hiroko Omori may publish in the future.

Co-authorship network of co-authors of Hiroko Omori

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroko Omori. A scholar is included among the top collaborators of Hiroko Omori based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroko Omori. Hiroko Omori is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Omori, Hiroko, Kyosuke Asakawa, Emi K. Nishimura, et al.. (2025). ER Stress Ire1‐Xbp1s Pathway Maintains Youthful Epidermal Basal Layer Through the Regulation of Cell Proliferation. Aging Cell. 24(12). e70258–e70258.
2.
Sakakibara, Shuhei, Hiroko Omori, Daisuke Okuzaki, et al.. (2023). Opposing roles of RUBCN isoforms in autophagy and memory B cell generation. Science Signaling. 16(803). eade3599–eade3599. 3 indexed citations
3.
Minami, Shohei, Naomi Matsumoto, Hiroko Omori, et al.. (2023). Effective SARS-CoV-2 replication of monolayers of intestinal epithelial cells differentiated from human induced pluripotent stem cells. Scientific Reports. 13(1). 11610–11610. 2 indexed citations
4.
Tsuzukibashi, Osamu, Taira Kobayashi, Yuji Takahashi, et al.. (2021). One-Step Multiplex PCR for Simultaneous Detection and Identification of Eight Medically Important <i>Candida</i> Species. Open Journal of Stomatology. 11(1). 14–24. 6 indexed citations
5.
Omori, Hiroko, Maho Hamasaki, Tomohisa Hatta, et al.. (2020). ERdj8 governs the size of autophagosomes during the formation process. The Journal of Cell Biology. 219(8). 21 indexed citations
6.
Omori, Hiroko, Maho Hamasaki, Tomohisa Hatta, et al.. (2020). Correction: ERdj8 governs the size of autophagosomes during the formation process. The Journal of Cell Biology. 220(9). 1 indexed citations
7.
Maeda, Yusuke, Natsuko Kishishita, Uamporn Siripanyaphinyo, et al.. (2019). The use of green fluorescent protein-tagged virus-like particles as a tracer in the early phase of chikungunya infection. Virus Research. 272. 197732–197732. 4 indexed citations
8.
Ouji, Yukiteru, Masaharu Sakagami, Hiroko Omori, et al.. (2017). Efficient induction of inner ear hair cell-like cells from mouse ES cells using combination of Math1 transfection and conditioned medium from ST2 stromal cells. Stem Cell Research. 23. 50–56. 8 indexed citations
9.
Sakai, Yusuke, et al.. (2017). Two-amino acids change in the nsp4 of SARS coronavirus abolishes viral replication. Virology. 510. 165–174. 96 indexed citations
10.
Tabata, Keisuke, Masashi Arakawa, Atsuki Nara, et al.. (2016). Unique Requirement for ESCRT Factors in Flavivirus Particle Formation on the Endoplasmic Reticulum. Cell Reports. 16(9). 2339–2347. 78 indexed citations
11.
Rahayu, Retno Pudji, et al.. (2016). Localization of latency-associated nuclear antigen (LANA) on mitotic chromosomes. Virology. 496. 51–58. 5 indexed citations
12.
Maejima, Ikuko, Atsushi Takahashi, Hiroko Omori, et al.. (2013). Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury. The EMBO Journal. 32(17). 2336–2347. 467 indexed citations breakdown →
13.
Matsumoto, Yusuke, Yohei Hayashi, Hiroko Omori, et al.. (2012). Bornavirus Closely Associates and Segregates with Host Chromosomes to Ensure Persistent Intranuclear Infection. Cell Host & Microbe. 11(5). 492–503. 82 indexed citations
14.
Saitoh, Tatsuya, Jun Komano, Yasunori Saitoh, et al.. (2012). Neutrophil Extracellular Traps Mediate a Host Defense Response to Human Immunodeficiency Virus-1. Cell Host & Microbe. 12(1). 109–116. 557 indexed citations breakdown →
15.
Kageyama, Shun, Hiroko Omori, Tatsuya Saitoh, et al.. (2011). The LC3 recruitment mechanism is separate from Atg9L1-dependent membrane formation in the autophagic response againstSalmonella. Molecular Biology of the Cell. 22(13). 2290–2300. 144 indexed citations
16.
Saitoh, Tatsuya, Naonobu Fujita, Takuya Hayashi, et al.. (2009). Atg9a controls dsDNA-driven dynamic translocation of STING and the innate immune response. Proceedings of the National Academy of Sciences. 106(49). 20842–20846. 675 indexed citations breakdown →
17.
Matsunaga, Kohichi, Tatsuya Saitoh, Keisuke Tabata, et al.. (2009). Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages. Nature Cell Biology. 11(4). 385–396. 945 indexed citations breakdown →
18.
Fujita, Naonobu, Takashi Itoh, Hiroko Omori, et al.. (2008). The Atg16L Complex Specifies the Site of LC3 Lipidation for Membrane Biogenesis in Autophagy. Molecular Biology of the Cell. 19(5). 2092–2100. 847 indexed citations breakdown →
19.
Uematsu, Satoshi, Tsuneyasu Kaisho, Takashi Tanaka, et al.. (2007). The C/EBPβ Isoform 34-kDa LAP Is Responsible for NF-IL-6-Mediated Gene Induction in Activated Macrophages, but Is Not Essential for Intracellular Bacteria Killing. The Journal of Immunology. 179(8). 5378–5386. 52 indexed citations
20.
Kato, Takahiro, et al.. (2007). Maturation of fimbria precursor protein by exogenous gingipains inPorphyromonas gingivalisgingipain-null mutant. FEMS Microbiology Letters. 273(1). 96–102. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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