Ignazia Tusa

823 total citations
30 papers, 597 citations indexed

About

Ignazia Tusa is a scholar working on Molecular Biology, Hematology and Cancer Research. According to data from OpenAlex, Ignazia Tusa has authored 30 papers receiving a total of 597 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 8 papers in Hematology and 7 papers in Cancer Research. Recurrent topics in Ignazia Tusa's work include Melanoma and MAPK Pathways (11 papers), Chronic Myeloid Leukemia Treatments (8 papers) and PI3K/AKT/mTOR signaling in cancer (5 papers). Ignazia Tusa is often cited by papers focused on Melanoma and MAPK Pathways (11 papers), Chronic Myeloid Leukemia Treatments (8 papers) and PI3K/AKT/mTOR signaling in cancer (5 papers). Ignazia Tusa collaborates with scholars based in Italy, United States and Spain. Ignazia Tusa's co-authors include Elisabetta Rovida, Persio Dello Sbarba, Maria Grazia Cipolleschi, Giulia Cheloni, Graziana Digiacomo, Nathanael S. Gray, Antonella Gozzini, Xianming Deng, Shaoguang Li and Yi Shan and has published in prestigious journals such as Blood, Cancer Research and Oncogene.

In The Last Decade

Ignazia Tusa

29 papers receiving 589 citations

Peers

Ignazia Tusa

ONCOL

HEMAT

IMMUN

Sho Kubota Japan

Alice Cani Italy

Grace R. Anderson United States

Mariana Lazarini Brazil

Jörn Lausen Germany

Andrea Ghelli Luserna di Rorà Italy

Nicole E. McNeil United States

A M Martelli Italy

Rikio Suzuki Japan

Shengyan Xiang United States

Sho Kubota Japan

Ignazia Tusa

369 ×1.1

122 ×1.0

ONCOL

80 ×0.7

97 ×1.0

HEMAT

65 ×0.9

IMMUN

Citations per year, relative to Ignazia Tusa Ignazia Tusa (= 1×) peers Sho Kubota

Countries citing papers authored by Ignazia Tusa

Since Specialization

Citations

This map shows the geographic impact of Ignazia Tusa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ignazia Tusa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ignazia Tusa more than expected).

Fields of papers citing papers by Ignazia Tusa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ignazia Tusa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ignazia Tusa. The network helps show where Ignazia Tusa may publish in the future.

Co-authorship network of co-authors of Ignazia Tusa

This figure shows the co-authorship network connecting the top 25 collaborators of Ignazia Tusa. A scholar is included among the top collaborators of Ignazia Tusa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ignazia Tusa. Ignazia Tusa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Tusa, Ignazia, et al.. (2025). The MEK5/ERK5 pathway promotes the activation of the Hedgehog/GLI signaling in melanoma cells. Cellular Oncology. 48(3). 789–799. 1 indexed citations

Martella, Daniele, Ignazia Tusa, Armando Negri, et al.. (2025). Liquid Crystalline Networks Hamper the Malignancy of Cancer Cells. Advanced Healthcare Materials. 14(7). e2403607–e2403607. 1 indexed citations

Gardini, Lucia, Matteo Lulli, Ignazia Tusa, et al.. (2024). Importin subunit beta‐1 mediates ERK5 nuclear translocation, and its inhibition synergizes with ERK5 kinase inhibitors in reducing cancer cell proliferation. Molecular Oncology. 19(1). 99–113. 3 indexed citations

Tusa, Ignazia, Azucena Esparís‐Ogando, Atanasio Pandiella, et al.. (2024). Latent-Transforming Growth Factor β-Binding Protein 1/Transforming Growth Factor β1 Complex Drives Antitumoral Effects upon ERK5 Targeting in Melanoma. American Journal Of Pathology. 194(8). 1581–1591. 4 indexed citations

Tusa, Ignazia, Francesca Cencetti, Silvia Vannuccini, et al.. (2023). Sphingosine 1‐phosphate elicits a ROS‐mediated proinflammatory response in human endometrial stromal cells via ERK5 activation. The FASEB Journal. 37(8). e23061–e23061. 8 indexed citations

Tusa, Ignazia, et al.. (2023). Pathophysiological Impact of the MEK5/ERK5 Pathway in Oxidative Stress. Cells. 12(8). 1154–1154. 11 indexed citations

Peppicelli, Silvia, Ignazia Tusa, Giulia Cheloni, et al.. (2021). Glutamine Availability Controls BCR/Abl Protein Expression and Functional Phenotype of Chronic Myeloid Leukemia Cells Endowed with Stem/Progenitor Cell Potential. Cancers. 13(17). 4372–4372. 6 indexed citations

Tusa, Ignazia, Sinforosa Gagliardi, Matteo Lulli, et al.. (2021). Inhibition of ERK5 Elicits Cellular Senescence in Melanoma via the Cyclin-Dependent Kinase Inhibitor p21. Cancer Research. 82(3). 447–457. 29 indexed citations

Foglia, Beatrice, Salvatore Sutti, Stefania Cannito, et al.. (2021). Hepatocyte-Specific Deletion of HIF2α Prevents NASH-Related Liver Carcinogenesis by Decreasing Cancer Cell Proliferation. Cellular and Molecular Gastroenterology and Hepatology. 13(2). 459–482. 18 indexed citations

10.

Tusa, Ignazia, et al.. (2020). Beyond Kinase Activity: ERK5 Nucleo-Cytoplasmic Shuttling as a Novel Target for Anticancer Therapy. International Journal of Molecular Sciences. 21(3). 938–938. 29 indexed citations

11.

Tusa, Ignazia, Giulia Cheloni, Antonella Gozzini, et al.. (2018). Targeting the Extracellular Signal-Regulated Kinase 5 Pathway to Suppress Human Chronic Myeloid Leukemia Stem Cells. Stem Cell Reports. 11(4). 929–943. 20 indexed citations

12.

Tusa, Ignazia, Sinforosa Gagliardi, Silvia Pandolfi, et al.. (2018). ERK5 is activated by oncogenic BRAF and promotes melanoma growth. Oncogene. 37(19). 2601–2614. 53 indexed citations

13.

Cheloni, Giulia, Michele Tanturli, Ignazia Tusa, et al.. (2017). Targeting chronic myeloid leukemia stem cells with the hypoxia-inducible factor inhibitor acriflavine. Blood. 130(5). 655–665. 69 indexed citations

14.

Rovida, Elisabetta, Giovanni Di Maira, Ignazia Tusa, et al.. (2014). The mitogen-activated protein kinase ERK5 regulates the development and growth of hepatocellular carcinoma. Gut. 64(9). 1454–1465. 54 indexed citations

15.

Cannito, Stefania, Cristian Turato, Claudia Paternostro, et al.. (2014). Hypoxia up-regulates SERPINB3 through HIF-2α in human liver cancer cells. Oncotarget. 6(4). 2206–2221. 47 indexed citations

16.

Rovida, Elisabetta, Silvia Peppicelli, Francesca Bianchini, et al.. (2014). The metabolically-modulated stem cell niche: a dynamic scenario regulating cancer cell phenotype and resistance to therapy. Cell Cycle. 13(20). 3169–3175. 41 indexed citations

17.

Barbetti, V, Ignazia Tusa, Maria Grazia Cipolleschi, Elisabetta Rovida, & Persio Dello Sbarba. (2013). AML1/ETO sensitizes via TRAIL acute myeloid leukemia cells to the pro-apoptotic effects of hypoxia. Cell Death and Disease. 4(3). e536–e536. 21 indexed citations

18.

Barbetti, V, Andrea Morandi, Ignazia Tusa, et al.. (2013). Chromatin-associated CSF-1R binds to the promoter of proliferation-related genes in breast cancer cells. Oncogene. 33(34). 4359–4364. 28 indexed citations

19.

Ballardin, Michela, Ignazia Tusa, Nunzia Fontana, et al.. (2011). Non-thermal effects of 2.45 GHz microwaves on spindle assembly, mitotic cells and viability of Chinese hamster V-79 cells. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 716(1-2). 1–9. 32 indexed citations

20.

Scarpato, Roberto, et al.. (2009). Spontaneous and bleomycin‐induced chromosome damage in non cancer thyroid patients. European Journal of Clinical Investigation. 39(12). 1091–1097. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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