Ian C. Forster
- Nephrology top 0.1%
- Molecular Biology top 5%
- Nutrition and Dietetics top 0.5%
- Genetics top 1%
- Pulmonary and Respiratory Medicine top 5%
- Co-authors
- Heini MurerJürg BiberNati HernandoLeila V. VirkkiJ�rg BiberMartin TraebertCarsten A. WagnerGerti Stange
- Topics
- Ion Transport and Channel Regulation (59 papers)Ion channel regulation and function (42 papers)Parathyroid Disorders and Treatments (35 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Neuroscience
- Partner nations
- SwitzerlandAustraliaUnited States
In The Last Decade
Ian C. Forster
122 papers receiving 5.0k citations
Peers
Comparison fields: 5 of 133
- Nephrology 2.5k
- Molecular Biology 2.3k
- Nutrition and Dietetics 1.5k
- Genetics 1.2k
- Pulmonary and Respiratory Medicine 712
Countries citing papers authored by Ian C. Forster
This map shows the geographic impact of Ian C. Forster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ian C. Forster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ian C. Forster more than expected).
Fields of papers citing papers by Ian C. Forster
This network shows the impact of papers produced by Ian C. Forster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ian C. Forster. The network helps show where Ian C. Forster may publish in the future.
Co-authorship network of co-authors of Ian C. Forster
This figure shows the co-authorship network connecting the top 25 collaborators of Ian C. Forster. A scholar is included among the top collaborators of Ian C. Forster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ian C. Forster. Ian C. Forster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 1 | |
| 3 | 8 | |
| 4 | 17 | |
| 5 | 29 | |
| 6 | 15 | |
| 7 | 7 | |
| 8 | 51 | |
| 9 | 18 | |
| 10 | 136 | |
| 11 | 192 | |
| 12 | 36 | |
| 13 | 6 | |
| 14 | 35 | |
| 15 | 36 | |
| 16 | 6 | |
| 17 | 37 | |
| 18 | 14 | |
| 19 | 25 | |
| 20 | 1 |
About Ian C. Forster
Ian C. Forster is a scholar working on Nephrology, Nutrition and Dietetics and Biochemistry, having authored 123 papers that have together received 5.1k indexed citations. Recurring topics across this work include Ion Transport and Channel Regulation (59 papers), Ion channel regulation and function (42 papers) and Parathyroid Disorders and Treatments (35 papers). The work is most often cited by research in Nephrology (2.5k citations), Nutrition and Dietetics (1.5k citations) and Genetics (1.2k citations). Ian C. Forster has collaborated with scholars based in Switzerland, Australia and United States. Frequent co-authors include Heini Murer, Jürg Biber, Nati Hernando, Leila V. Virkki, J�rg Biber, Martin Traebert, Carsten A. Wagner, Gerti Stange, Katja Köhler and Georg Lambert. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.