I. Schedel
- Immunology top 5%
- Epidemiology top 10%
- Radiology, Nuclear Medicine and Imaging top 10%
- Molecular Biology
- Oncology
- Co-authors
- H. DeicherD. PeestH. KühnleS NiesertUlrike BodeMichael GlaubitzKarl Ulrich PetryHeinrich G. Köchel
- Topics
- Monoclonal and Polyclonal Antibodies Research (13 papers)Multiple Myeloma Research and Treatments (9 papers)Chronic Lymphocytic Leukemia Research (7 papers)
- Cited by
- ImmunologyHematologyVirology
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
I. Schedel
49 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 89
- Immunology 460
- Epidemiology 358
- Radiology, Nuclear Medicine and Imaging 226
- Molecular Biology 217
- Oncology 207
Countries citing papers authored by I. Schedel
This map shows the geographic impact of I. Schedel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. Schedel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. Schedel more than expected).
Fields of papers citing papers by I. Schedel
This network shows the impact of papers produced by I. Schedel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. Schedel. The network helps show where I. Schedel may publish in the future.
Co-authorship network of co-authors of I. Schedel
This figure shows the co-authorship network connecting the top 25 collaborators of I. Schedel. A scholar is included among the top collaborators of I. Schedel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with I. Schedel. I. Schedel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 14 | |
| 3 | 14 | |
| 4 | Posttravel fever attack. | 1 |
| 5 | 2 | |
| 6 | 3 | |
| 7 | 12 | |
| 8 | 48 | |
| 9 | 12 | |
| 10 | 197 | |
| 11 | 65 | |
| 12 | 69 | |
| 13 | 33 | |
| 14 | 6 | |
| 15 | 11 | |
| 16 | 4 | |
| 17 | 31 | |
| 18 | 5 | |
| 19 | Regulation of the in vitro monoclonal immunoglobulin production in cultures of peripheral blood mononuclear cells and bone marrow cells from myeloma patients mediated by T cell dependent mitogens. | 10 |
| 20 | 2 |
About I. Schedel
I. Schedel is a scholar working on Hematology, Virology and Immunology, having authored 50 papers that have together received 1.1k indexed citations. Recurring topics across this work include Monoclonal and Polyclonal Antibodies Research (13 papers), Multiple Myeloma Research and Treatments (9 papers) and Chronic Lymphocytic Leukemia Research (7 papers). The work is most often cited by research in Immunology (460 citations), Hematology (132 citations) and Virology (50 citations). I. Schedel has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include H. Deicher, D. Peest, H. Kühnle, S Niesert, Ulrike Bode, Michael Glaubitz, Karl Ulrich Petry, Heinrich G. Köchel, Hans‐Joachim Schmoll and E. Kupsch. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Journal of Immunology and PEDIATRICS.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.