Hye Jin Jee

773 total citations
22 papers, 585 citations indexed

About

Hye Jin Jee is a scholar working on Molecular Biology, Physiology and Oncology. According to data from OpenAlex, Hye Jin Jee has authored 22 papers receiving a total of 585 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Physiology and 4 papers in Oncology. Recurrent topics in Hye Jin Jee's work include Telomeres, Telomerase, and Senescence (4 papers), DNA Repair Mechanisms (3 papers) and Genomics, phytochemicals, and oxidative stress (3 papers). Hye Jin Jee is often cited by papers focused on Telomeres, Telomerase, and Senescence (4 papers), DNA Repair Mechanisms (3 papers) and Genomics, phytochemicals, and oxidative stress (3 papers). Hye Jin Jee collaborates with scholars based in South Korea, United Kingdom and United States. Hye Jin Jee's co-authors include Jeanho Yun, Hyun‐Ju Kim, Jee‐Hyun Um, Young Yeon Kim, Sun Sik Bae, Minjee Kim, Sang Goo Lee, Young Mi Kim, Hyongjong Koh and Yoe‐Sik Bae and has published in prestigious journals such as The FASEB Journal, Biochemical and Biophysical Research Communications and International Journal of Molecular Sciences.

In The Last Decade

Hye Jin Jee

20 papers receiving 577 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hye Jin Jee South Korea 14 330 93 92 85 68 22 585
Raquel M. Melero-Fernández de Mera Spain 11 319 1.0× 40 0.4× 96 1.0× 70 0.8× 49 0.7× 17 659
Rut Fadó Spain 17 449 1.4× 134 1.4× 174 1.9× 61 0.7× 93 1.4× 23 858
Stéphanie Reix Spain 11 379 1.1× 46 0.5× 111 1.2× 67 0.8× 57 0.8× 11 716
Congli Hu China 12 210 0.6× 57 0.6× 53 0.6× 71 0.8× 45 0.7× 27 537
Mao‐Liang Chen Taiwan 18 388 1.2× 50 0.5× 55 0.6× 83 1.0× 54 0.8× 56 918
Beatrice D’Orsi Ireland 11 332 1.0× 59 0.6× 111 1.2× 37 0.4× 63 0.9× 19 541
Nicoletta C. Surdo United Kingdom 16 619 1.9× 62 0.7× 136 1.5× 54 0.6× 30 0.4× 26 888
Hebe M. Guardiola‐Diaz United States 7 340 1.0× 31 0.3× 110 1.2× 47 0.6× 53 0.8× 9 656
Shiyong Diao United States 11 351 1.1× 51 0.5× 218 2.4× 96 1.1× 57 0.8× 11 604

Countries citing papers authored by Hye Jin Jee

Since Specialization
Citations

This map shows the geographic impact of Hye Jin Jee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hye Jin Jee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hye Jin Jee more than expected).

Fields of papers citing papers by Hye Jin Jee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hye Jin Jee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hye Jin Jee. The network helps show where Hye Jin Jee may publish in the future.

Co-authorship network of co-authors of Hye Jin Jee

This figure shows the co-authorship network connecting the top 25 collaborators of Hye Jin Jee. A scholar is included among the top collaborators of Hye Jin Jee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hye Jin Jee. Hye Jin Jee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lee, Hyun Sook, Hye Jin Jee, & Yi-Sook Jung. (2025). Neuroprotective Effect of β-Lapachone against Glutamate-Induced Injury in HT22 Cells. Biomolecules & Therapeutics. 33(2). 286–296.
2.
3.
Kim, Suyeon, et al.. (2024). Hypnotic effect of AR-001 through adenosine A1 receptor. European Journal of Pharmacology. 981. 176847–176847.
4.
Choi, Sun Ah, et al.. (2023). Sex Differences in the Preventive Effect of Cardiovascular and Metabolic Therapeutics on Dementia. Biomolecules & Therapeutics. 31(6). 583–598. 2 indexed citations
5.
Jee, Hye Jin, et al.. (2021). Inhibitory Effect of Fermented Mentha piperita on MUC5AC in Lung Epithelial Cells. Journal of the Korean Society of Food Science and Nutrition. 50(4). 330–338. 2 indexed citations
6.
Jee, Hye Jin, et al.. (2020). Effect of Caffeine Consumption on the Risk for Neurological and Psychiatric Disorders: Sex Differences in Human. Nutrients. 12(10). 3080–3080. 74 indexed citations
7.
Kim, Young Yeon, Jee‐Hyun Um, Jeong‐Hyun Yoon, et al.. (2019). Assessment of mitophagy in mt‐Keima Drosophila revealed an essential role of the PINK1‐Parkin pathway in mitophagy induction in vivo. The FASEB Journal. 33(9). 9742–9751. 65 indexed citations
8.
Jee, Hye Jin, et al.. (2019). Impact of Sleep Disorder as a Risk Factor for Dementia in Men and Women. Biomolecules & Therapeutics. 28(1). 58–73. 35 indexed citations
9.
Kim, Young Yeon, Hye Jin Jee, Jee‐Hyun Um, et al.. (2017). Cooperation between p21 and Akt is required for p53‐dependent cellular senescence. Aging Cell. 16(5). 1094–1103. 84 indexed citations
10.
Kim, Minjee, Young Yeon Kim, Hye Jin Jee, et al.. (2016). Akt3 knockdown induces mitochondrial dysfunction in human cancer cells. Acta Biochimica et Biophysica Sinica. 48(5). 447–453. 16 indexed citations
11.
12.
Kim, Hyun‐Ju, et al.. (2012). Melatonin suppresses doxorubicin‐induced premature senescence of A549 lung cancer cells by ameliorating mitochondrial dysfunction. Journal of Pineal Research. 53(4). 335–343. 43 indexed citations
13.
Kim, Hyunju, Mee Sook Roh, Hye Jin Jee, et al.. (2012). Loss of Med1/TRAP220 promotes the invasion and metastasis of human non-small-cell lung cancer cells by modulating the expression of metastasis-related genes. Cancer Letters. 321(2). 195–202. 36 indexed citations
14.
Jee, Hye Jin, et al.. (2012). p21WAF1/CIP1 deficiency induces mitochondrial dysfunction in HCT116 colon cancer cells. Biochemical and Biophysical Research Communications. 430(2). 653–658. 13 indexed citations
15.
Kim, Hyunju, Hye Jin Jee, & Jeanho Yun. (2011). DNA damage induces down-regulation of <italic>PEPCK</italic> and <italic>G6P</italic> gene expression through degradation of PGC-1α. Acta Biochimica et Biophysica Sinica. 43(8). 589–594. 16 indexed citations
16.
Jee, Hye Jin, et al.. (2011). Nek6 suppresses the premature senescence of human cancer cells induced by camptothecin and doxorubicin treatment. Biochemical and Biophysical Research Communications. 408(4). 669–673. 23 indexed citations
17.
Kim, Hyun‐Ju, Hye Jin Jee, Minjee Kim, et al.. (2011). Glucose deprivation is associated with Chk1 degradation through the ubiquitin–proteasome pathway and effective checkpoint response to replication blocks. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1813(6). 1230–1238. 11 indexed citations
18.
Jee, Hye Jin, et al.. (2010). Nek6 overexpression antagonizes p53-induced senescence in human cancer cells. Cell Cycle. 9(23). 4703–4710. 55 indexed citations
19.
Jee, Hye Jin, et al.. (2009). UV light induces premature senescence in Akt1-null mouse embryonic fibroblasts by increasing intracellular levels of ROS. Biochemical and Biophysical Research Communications. 383(3). 358–362. 22 indexed citations
20.
Lee, Min‐Young, Hyun‐Ju Kim, Hye Jin Jee, et al.. (2008). Nek6 is involved in G2/M phase cell cycle arrest through DNA damage-induced phosphorylation. Cell Cycle. 7(17). 2705–2709. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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