Hydar Ali

6.7k total citations
100 papers, 5.4k citations indexed

About

Hydar Ali is a scholar working on Immunology, Molecular Biology and Physiology. According to data from OpenAlex, Hydar Ali has authored 100 papers receiving a total of 5.4k indexed citations (citations by other indexed papers that have themselves been cited), including 62 papers in Immunology, 58 papers in Molecular Biology and 21 papers in Physiology. Recurrent topics in Hydar Ali's work include Mast cells and histamine (51 papers), Receptor Mechanisms and Signaling (37 papers) and Asthma and respiratory diseases (21 papers). Hydar Ali is often cited by papers focused on Mast cells and histamine (51 papers), Receptor Mechanisms and Signaling (37 papers) and Asthma and respiratory diseases (21 papers). Hydar Ali collaborates with scholars based in United States, Thailand and United Kingdom. Hydar Ali's co-authors include Bodduluri Haribabu, Ralph Snyderman, Ricardo M. Richardson, Hariharan Subramanian, Michael A. Beaven, Kshitij Gupta, Gary L. Stiles, Vickram Ramkumar, Saptarshi Roy and Eric D. Tomhave and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and PLoS ONE.

In The Last Decade

Hydar Ali

99 papers receiving 5.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hydar Ali United States 42 3.0k 2.4k 1.3k 1.0k 783 100 5.4k
Jean-Pierre Kinet United States 37 4.7k 1.6× 3.0k 1.2× 1.5k 1.2× 1.6k 1.6× 471 0.6× 50 9.6k
Yoshihide Kanaoka United States 49 2.0k 0.7× 2.5k 1.0× 2.4k 1.9× 736 0.7× 577 0.7× 93 6.8k
Alasdair M. Gilfillan United States 46 4.7k 1.6× 2.0k 0.8× 1.9k 1.5× 1.7k 1.6× 279 0.4× 111 6.7k
Karina Reiß Germany 42 1.3k 0.4× 3.2k 1.3× 1.3k 1.0× 1.4k 1.4× 2.0k 2.6× 71 7.3k
Toshiaki Kawakami United States 61 5.7k 1.9× 3.2k 1.3× 1.8k 1.4× 1.9k 1.9× 868 1.1× 165 9.6k
Giorgio Berton Italy 42 3.1k 1.0× 2.1k 0.9× 1.0k 0.8× 1.8k 1.7× 615 0.8× 109 6.3k
Ricardo M. Richardson United States 37 1.9k 0.6× 2.0k 0.8× 431 0.3× 663 0.6× 1.4k 1.8× 72 4.2k
Wanghua Gong United States 46 2.9k 1.0× 3.3k 1.4× 788 0.6× 309 0.3× 1.6k 2.1× 103 6.5k
Craig Gerard United States 20 2.3k 0.8× 1.7k 0.7× 574 0.5× 344 0.3× 1.0k 1.3× 26 4.2k
Hansruedi Loetscher Switzerland 34 2.2k 0.7× 2.4k 1.0× 1.2k 0.9× 385 0.4× 700 0.9× 47 5.6k

Countries citing papers authored by Hydar Ali

Since Specialization
Citations

This map shows the geographic impact of Hydar Ali's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hydar Ali with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hydar Ali more than expected).

Fields of papers citing papers by Hydar Ali

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hydar Ali. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hydar Ali. The network helps show where Hydar Ali may publish in the future.

Co-authorship network of co-authors of Hydar Ali

This figure shows the co-authorship network connecting the top 25 collaborators of Hydar Ali. A scholar is included among the top collaborators of Hydar Ali based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hydar Ali. Hydar Ali is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Roy, Saptarshi, et al.. (2024). MRGPRX2 facilitates IgE-mediated systemic anaphylaxis in a newly established knock-in mouse model. Journal of Allergy and Clinical Immunology. 155(3). 974–987.e1. 4 indexed citations
2.
Ali, Hydar, et al.. (2024). USE OF ANTIBIOTICS AND THE FARMERS' AWARENESS LEVEL OF ANTIBIOTICS RESISTANCE OF BROILER FARMS. Biological and Clinical Sciences Research Journal. 2024(1). 856–856. 2 indexed citations
3.
Ali, Hydar, et al.. (2024). Mast cell MrgprB2 in neuroimmune interaction in IgE-mediated airway inflammation and its modulation by β-arrestin2. Frontiers in Immunology. 15. 1470016–1470016. 5 indexed citations
4.
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Ayudhya, Chalatip Chompunud Na, et al.. (2021). Murepavadin, a Small Molecule Host Defense Peptide Mimetic, Activates Mast Cells via MRGPRX2 and MrgprB2. Frontiers in Immunology. 12. 689410–689410. 20 indexed citations
7.
Roy, Saptarshi, et al.. (2019). Angiogenic Host Defense Peptide AG-30/5C and Bradykinin B2 Receptor Antagonist Icatibant Are G Protein Biased Agonists for MRGPRX2 in Mast Cells. The Journal of Immunology. 202(4). 1229–1238. 39 indexed citations
8.
Subramanian, Hariharan, et al.. (2013). β-Defensins Activate Human Mast Cells via Mas-Related Gene X2. The Journal of Immunology. 191(1). 345–352. 122 indexed citations
9.
Guo, Qiang, Hariharan Subramanian, Kshitij Gupta, & Hydar Ali. (2011). Regulation of C3a Receptor Signaling in Human Mast Cells by G Protein Coupled Receptor Kinases. PLoS ONE. 6(7). e22559–e22559. 48 indexed citations
10.
Vibhuti, Arpana, Kshitij Gupta, Hariharan Subramanian, Qiang Guo, & Hydar Ali. (2011). Distinct and Shared Roles of β-Arrestin-1 and β-Arrestin-2 on the Regulation of C3a Receptor Signaling in Human Mast Cells. PLoS ONE. 6(5). e19585–e19585. 50 indexed citations
11.
Shenker, Bruce J., et al.. (2010). Inhibition of mast cell degranulation by a chimeric toxin containing a novel phosphatidylinositol-3,4,5-triphosphate phosphatase. Molecular Immunology. 48(1-3). 203–210. 18 indexed citations
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Ahamed, Jasimuddin, et al.. (2004). C3a Enhances Nerve Growth Factor-Induced NFAT Activation and Chemokine Production in a Human Mast Cell Line, HMC-1. The Journal of Immunology. 172(11). 6961–6968. 40 indexed citations
14.
Ahamed, Jasimuddin, Bodduluri Haribabu, & Hydar Ali. (2001). Cutting Edge: Differential Regulation of Chemoattractant Receptor-Induced Degranulation and Chemokine Production by Receptor Phosphorylation. The Journal of Immunology. 167(7). 3559–3563. 37 indexed citations
15.
Ali, Hydar, et al.. (1997). EFFECT OF SOME POLLUTANTS AS STRESSORS ON SOME BEHAVIOURAL PATTERNS AND PERFORMANCE OF BROILER CHICKENS |. Veterinary Medical Journal Giza. 45(3). 307–314. 2 indexed citations
16.
Haribabu, Bodduluri, Ricardo M. Richardson, Ian Fisher, et al.. (1997). Regulation of Human Chemokine Receptors CXCR4. Journal of Biological Chemistry. 272(45). 28726–28731. 249 indexed citations
17.
Richardson, Ricardo M., Bodduluri Haribabu, Hydar Ali, & Ralph Snyderman. (1996). Cross-desensitization Among Receptors for Platelet Activating Factor and Peptide Chemoattractants. Journal of Biological Chemistry. 271(45). 28717–28724. 39 indexed citations
18.
Ali, Hydar, Christa E. Müller, John W. Daly, & Michael A. Beaven. (1991). Methylxanthines block antigen-induced responses in RBL-2H3 cells independently of adenosine receptors or cyclic AMP: evidence for inhibition of antigen binding to IgE.. Journal of Pharmacology and Experimental Therapeutics. 258(3). 954–962. 18 indexed citations
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Maeyama, Kazutaka, R J Hohman, Hydar Ali, J R Cunha-Melo, & Michael A. Beaven. (1988). Assessment of IgE-receptor function through measurement of hydrolysis of membrane inositol phospholipids. New insights on the phenomena of biphasic antigen concentration-response curves and desensitization.. The Journal of Immunology. 140(11). 3919–3927. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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