Hwan‐Jin Hwang

879 total citations
23 papers, 729 citations indexed

About

Hwan‐Jin Hwang is a scholar working on Molecular Biology, Epidemiology and Physiology. According to data from OpenAlex, Hwan‐Jin Hwang has authored 23 papers receiving a total of 729 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 8 papers in Epidemiology and 7 papers in Physiology. Recurrent topics in Hwan‐Jin Hwang's work include Adipose Tissue and Metabolism (6 papers), Metabolism, Diabetes, and Cancer (5 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). Hwan‐Jin Hwang is often cited by papers focused on Adipose Tissue and Metabolism (6 papers), Metabolism, Diabetes, and Cancer (5 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). Hwan‐Jin Hwang collaborates with scholars based in South Korea and Japan. Hwan‐Jin Hwang's co-authors include Kyung Mook Choi, Sei Hyun Baik, Hye Jin Yoo, Tae Woo Jung, Ho Cheol Hong, Nan Hee Kim, Sin Gon Kim, Ji A Seo, Dong Seop Choi and Hye Jin Yoo and has published in prestigious journals such as PLoS ONE, Diabetologia and Biochemical Pharmacology.

In The Last Decade

Hwan‐Jin Hwang

22 papers receiving 724 citations

Peers

Hwan‐Jin Hwang
Hagoon Jang South Korea
Minna Woo Canada
Ki-Up Lee South Korea
Gautham Yepuri United States
Hagoon Jang South Korea
Hwan‐Jin Hwang
Citations per year, relative to Hwan‐Jin Hwang Hwan‐Jin Hwang (= 1×) peers Hagoon Jang

Countries citing papers authored by Hwan‐Jin Hwang

Since Specialization
Citations

This map shows the geographic impact of Hwan‐Jin Hwang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hwan‐Jin Hwang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hwan‐Jin Hwang more than expected).

Fields of papers citing papers by Hwan‐Jin Hwang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hwan‐Jin Hwang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hwan‐Jin Hwang. The network helps show where Hwan‐Jin Hwang may publish in the future.

Co-authorship network of co-authors of Hwan‐Jin Hwang

This figure shows the co-authorship network connecting the top 25 collaborators of Hwan‐Jin Hwang. A scholar is included among the top collaborators of Hwan‐Jin Hwang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hwan‐Jin Hwang. Hwan‐Jin Hwang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Joo Won, Minjeong Park, Kyung Mook Choi, et al.. (2025). HD6277 Suppresses Muscle Atrophy by Promoting Myogenic Factors and Inhibiting Proteolysis in Aged Mice. Journal of Cachexia Sarcopenia and Muscle. 16(2). e13805–e13805. 1 indexed citations
2.
Kim, Joo Won, Minjeong Park, Kyung Mook Choi, et al.. (2024). GPR40-full agonist AM1638 alleviates palmitate-induced oxidative damage in H9c2 cells via an AMPK-dependent pathway. BMB Reports. 58(3). 133–139.
3.
4.
Park, Minjeong, Eun Roh, Kyung Mook Choi, et al.. (2022). Sestrin2 Regulates Beneficial β3-Adrenergic Receptor-Mediated Effects Observed in Inguinal White Adipose Tissue and Soleus Muscle. Endocrinology and Metabolism. 37(3). 552–557. 2 indexed citations
5.
Roh, Eun, et al.. (2022). GPR40 Agonism Modulates Inflammatory Reactions in Vascular Endothelial Cells. Diabetes & Metabolism Journal. 46(3). 506–511. 16 indexed citations
6.
Roh, Eun, Hwan‐Jin Hwang, Joo Won Kim, et al.. (2020). Ginsenoside Mc1 improves liver steatosis and insulin resistance by attenuating ER stress. Journal of Ethnopharmacology. 259. 112927–112927. 35 indexed citations
7.
Hwang, Hwan‐Jin, Joo Won Kim, Jung A Kim, et al.. (2019). Ginsenoside compound-Mc1 attenuates oxidative stress and apoptosis in cardiomyocytes through an AMP-activated protein kinase–dependent mechanism. Journal of Ginseng Research. 44(4). 664–671. 26 indexed citations
8.
Chung, Hye Soo, Hwan‐Jin Hwang, Soon Young Hwang, et al.. (2018). Association of serum Sestrin2 level with metabolic risk factors in newly diagnosed drug-naïve type 2 diabetes. Diabetes Research and Clinical Practice. 144. 34–41. 28 indexed citations
9.
Hwang, Hwan‐Jin, Tae Woo Jung, Joo Won Kim, et al.. (2018). Protectin DX prevents H2O2-mediated oxidative stress in vascular endothelial cells via an AMPK-dependent mechanism. Cellular Signalling. 53. 14–21. 26 indexed citations
10.
Hwang, Hwan‐Jin, Tae Woo Jung, Hyun Jung Lee, et al.. (2017). Knockdown of sestrin2 increases pro-inflammatory reactions and ER stress in the endothelium via an AMPK dependent mechanism. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1863(6). 1436–1444. 58 indexed citations
11.
Kim, Ji‐Youn, Hwan‐Jin Hwang, Hak‐Jae Chung, et al.. (2016). Identification and Functional Analysis of Pig β-1,4-N-Acetylglucosaminyltransferase A (MGAT4A). Journal of Life Science. 26(3). 275–281. 1 indexed citations
12.
Hwang, Hwan‐Jin, Hye Soo Chung, Tae Woo Jung, et al.. (2015). The dipeptidyl peptidase-IV inhibitor inhibits the expression of vascular adhesion molecules and inflammatory cytokines in HUVECs via Akt- and AMPK-dependent mechanisms. Molecular and Cellular Endocrinology. 405. 25–34. 41 indexed citations
13.
Hwang, Hwan‐Jin, Tae Woo Jung, Baek‐hui Kim, et al.. (2015). A dipeptidyl peptidase-IV inhibitor improves hepatic steatosis and insulin resistance by AMPK-dependent and JNK-dependent inhibition of LECT2 expression. Biochemical Pharmacology. 98(1). 157–166. 59 indexed citations
14.
Jung, Tae Woo, Ho Cheol Hong, Hwan‐Jin Hwang, et al.. (2015). C1q/TNF-Related Protein 9 (CTRP9) attenuates hepatic steatosis via the autophagy-mediated inhibition of endoplasmic reticulum stress. Molecular and Cellular Endocrinology. 417. 131–140. 64 indexed citations
15.
Hwang, Hwan‐Jin, Tae Woo Jung, Ho Cheol Hong, et al.. (2015). LECT2 induces atherosclerotic inflammatory reaction via CD209 receptor-mediated JNK phosphorylation in human endothelial cells. Metabolism. 64(9). 1175–1182. 52 indexed citations
16.
Jung, Tae Woo, Hwan‐Jin Hwang, Ho Cheol Hong, et al.. (2015). BAIBA attenuates insulin resistance and inflammation induced by palmitate or a high fat diet via an AMPK–PPARδ-dependent pathway in mice. Diabetologia. 58(9). 2096–2105. 105 indexed citations
17.
Hwang, Hwan‐Jin, Tae Woo Jung, Ja Young Ryu, et al.. (2014). Dipeptidyl petidase-IV inhibitor (gemigliptin) inhibits tunicamycin-induced endoplasmic reticulum stress, apoptosis and inflammation in H9c2 cardiomyocytes. Molecular and Cellular Endocrinology. 392(1-2). 1–7. 29 indexed citations
18.
Jung, Tae Woo, Hwan‐Jin Hwang, Ho Cheol Hong, et al.. (2014). Resolvin D1 reduces ER stress-induced apoptosis and triglyceride accumulation through JNK pathway in HepG2 cells. Molecular and Cellular Endocrinology. 391(1-2). 30–40. 59 indexed citations
19.
Yoo, Hye Jin, Hwan‐Jin Hwang, Tae Woo Jung, et al.. (2014). Adipose Gene Expression Profiles Related to Metabolic Syndrome Using Microarray Analyses in Two Different Models. Diabetes & Metabolism Journal. 38(5). 356–356. 7 indexed citations
20.
Hwang, Hwan‐Jin, Tae Woo Jung, Ho Cheol Hong, et al.. (2013). Progranulin Protects Vascular Endothelium against Atherosclerotic Inflammatory Reaction via Akt/eNOS and Nuclear Factor-κB Pathways. PLoS ONE. 8(9). e76679–e76679. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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