Humberto E. Trejo Bittar

3.9k total citations · 2 hit papers
60 papers, 2.5k citations indexed

About

Humberto E. Trejo Bittar is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Surgery. According to data from OpenAlex, Humberto E. Trejo Bittar has authored 60 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Pulmonary and Respiratory Medicine, 16 papers in Molecular Biology and 13 papers in Surgery. Recurrent topics in Humberto E. Trejo Bittar's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (10 papers), Neonatal Respiratory Health Research (10 papers) and Ion Transport and Channel Regulation (8 papers). Humberto E. Trejo Bittar is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (10 papers), Neonatal Respiratory Health Research (10 papers) and Ion Transport and Channel Regulation (8 papers). Humberto E. Trejo Bittar collaborates with scholars based in United States, Venezuela and Uruguay. Humberto E. Trejo Bittar's co-authors include Jacob I. Sznajder, Tracy Tabib, Robert Lafyatis, John Sembrat, Eleanor Valenzi, Mauricio Rojas, Laura A. Dada, Christina Morse, Emilia Lecuona and Arturo Briva and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Experimental Medicine and SHILAP Revista de lepidopterología.

In The Last Decade

Humberto E. Trejo Bittar

59 papers receiving 2.5k citations

Hit Papers

Proliferating SPP1/MERTK-expressing macrophages in idiopa... 2019 2026 2021 2023 2019 2019 100 200 300 400
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Proliferating SPP1/MERTK-expressing macrophages in idiopathic pulmonary fibrosis
    2019 461 citations Tracy Tabib, John Sembrat et al. profile →
  2. Single-cell analysis reveals fibroblast heterogeneity and myofibroblasts in systemic sclerosis-associated interstitial lung disease
    2019 218 citations Eleanor Valenzi, Tracy Tabib et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Humberto E. Trejo Bittar United States 23 1.2k 894 415 332 316 60 2.5k
Louise Hecker United States 21 1.3k 1.0× 866 1.0× 446 1.1× 480 1.4× 348 1.1× 40 2.9k
Nasreen Khalil Canada 30 2.1k 1.7× 941 1.1× 352 0.8× 585 1.8× 290 0.9× 65 3.4k
Steven K. Huang United States 29 1.4k 1.1× 788 0.9× 259 0.6× 300 0.9× 265 0.8× 74 2.6k
Chrystelle Garat United States 19 1.2k 0.9× 1.3k 1.4× 385 0.9× 270 0.8× 297 0.9× 27 2.9k
Mitchell A. Olman United States 35 1.7k 1.4× 870 1.0× 308 0.7× 411 1.2× 322 1.0× 68 3.5k
John Sembrat United States 23 1.6k 1.3× 1.0k 1.1× 636 1.5× 363 1.1× 270 0.9× 68 3.0k
Daniel J. Kass United States 26 1.7k 1.4× 693 0.8× 377 0.9× 375 1.1× 168 0.5× 77 2.6k
Ari Zaiman United States 30 1.9k 1.5× 993 1.1× 419 1.0× 433 1.3× 379 1.2× 41 3.4k
Masamichi Ishizaki Japan 29 756 0.6× 824 0.9× 306 0.7× 227 0.7× 360 1.1× 85 2.9k
Changfu Yao United States 22 1.3k 1.0× 803 0.9× 234 0.6× 175 0.5× 350 1.1× 47 2.2k

Countries citing papers authored by Humberto E. Trejo Bittar

Since Specialization
Citations

This map shows the geographic impact of Humberto E. Trejo Bittar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Humberto E. Trejo Bittar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Humberto E. Trejo Bittar more than expected).

Fields of papers citing papers by Humberto E. Trejo Bittar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Humberto E. Trejo Bittar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Humberto E. Trejo Bittar. The network helps show where Humberto E. Trejo Bittar may publish in the future.

Co-authorship network of co-authors of Humberto E. Trejo Bittar

This figure shows the co-authorship network connecting the top 25 collaborators of Humberto E. Trejo Bittar. A scholar is included among the top collaborators of Humberto E. Trejo Bittar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Humberto E. Trejo Bittar. Humberto E. Trejo Bittar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Snyder, Mark E., Kaveh Moghbeli, Andrew Craig, et al.. (2022). Modulation of tissue resident memory T cells by glucocorticoids after acute cellular rejection in lung transplantation. The Journal of Experimental Medicine. 219(4). 25 indexed citations
2.
Camiolo, Matthew, Xiuxia Zhou, Qi Wei, et al.. (2021). Machine learning implicates the IL-18 signaling axis in severe asthma. JCI Insight. 6(21). 16 indexed citations
3.
Rosenzweig, Margaret, Adrian V. Lee, Steffi Oesterreich, et al.. (2021). The Development and Implementation of an Autopsy/ Tissue Donation for Breast Cancer Research. The New Bioethics. 27(4). 349–361.
4.
Saygın, Didem, Tracy Tabib, Humberto E. Trejo Bittar, et al.. (2020). Transcriptional profiling of lung cell populations in idiopathic pulmonary arterial hypertension. Pulmonary Circulation. 10(1). 1–15. 67 indexed citations
5.
Mady, Leila J., Abhijit Roy, Ali Aral, et al.. (2020). In-vivo efficacy of biodegradable ultrahigh ductility Mg-Li-Zn alloy tracheal stents for pediatric airway obstruction. Communications Biology. 3(1). 787–787. 19 indexed citations
6.
Yoon, Joo Heung, Mehdi Nouraie, Xiaoping Chen, et al.. (2018). Characteristics of lung cancer among patients with idiopathic pulmonary fibrosis and interstitial lung disease – analysis of institutional and population data. Respiratory Research. 19(1). 195–195. 57 indexed citations
7.
8.
Mehrad, Mitra, Somak Roy, Humberto E. Trejo Bittar, & Sanja Đačić. (2017). Next-Generation Sequencing Approach to Non–Small Cell Lung Carcinoma Yields More Actionable Alterations. Archives of Pathology & Laboratory Medicine. 142(3). 353–357. 21 indexed citations
9.
Mehrad, Mitra, Humberto E. Trejo Bittar, & Samuel A. Yousem. (2017). Sex steroid receptor expression in idiopathic pulmonary fibrosis. Human Pathology. 66. 200–205. 9 indexed citations
10.
Tan, Jiangning, John Tedrow, Brenda Juan-Guardela, et al.. (2016). Expression of RXFP1 Is Decreased in Idiopathic Pulmonary Fibrosis: Implications for Relaxin-based Therapies. American Journal of Respiratory and Critical Care Medicine. 194(11). 1392–1402. 45 indexed citations
11.
Fuhrman, Carl R., et al.. (2016). Pulmonary Alveolar Microlithiasis:AIRP Best Cases in Radiologic-Pathologic Correlation. Radiographics. 36(5). 1334–1338. 10 indexed citations
12.
Bittar, Humberto E. Trejo & Liron Pantanowitz. (2015). Merkel Cell Polyomavirus is Not Detected in Lung Adenocarcinomas by Immunohistochemistry. Applied immunohistochemistry & molecular morphology. 24(6). 427–430. 4 indexed citations
13.
Lecuona, Emilia, Haiying Sun, Jiwang Chen, et al.. (2013). Protein Kinase A-Iα Regulates Na,K-ATPase Endocytosis in Alveolar Epithelial Cells Exposed to High CO2 Concentrations. American Journal of Respiratory Cell and Molecular Biology. 48(5). 626–634. 35 indexed citations
14.
Kamp, David W., Gang Liu, Paul Cheresh, et al.. (2013). Asbestos-Induced Alveolar Epithelial Cell Apoptosis. The Role of Endoplasmic Reticulum Stress Response. American Journal of Respiratory Cell and Molecular Biology. 49(6). 892–901. 51 indexed citations
15.
Kamp, David W., et al.. (2012). Asbestos-Induced Alveolar Epithelial Cell Apoptosis: Mitochondria-ER Crosstalk. A4277–A4277. 1 indexed citations
16.
Welch, Lynn C., Emilia Lecuona, Arturo Briva, et al.. (2010). Extracellular signal‐regulated kinase (ERK) participates in the hypercapnia‐induced Na,K‐ATPase downregulation. FEBS Letters. 584(18). 3985–3989. 42 indexed citations
17.
Zhou, Guofei, Laura A. Dada, Minghua Wu, et al.. (2009). Hypoxia-induced alveolar epithelial-mesenchymal transition requires mitochondrial ROS and hypoxia-inducible factor 1. American Journal of Physiology-Lung Cellular and Molecular Physiology. 297(6). L1120–L1130. 193 indexed citations
18.
Bittar, Humberto E. Trejo, et al.. (2009). Role of kinesin light chain‐2 of kinesin‐1 in the traffic of Na,K‐ATPase‐containing vesicles in alveolar epithelial cells. The FASEB Journal. 24(2). 374–382. 15 indexed citations
19.
Comellas, Alejandro P., Arturo Briva, Laura A. Dada, et al.. (2008). Endothelin-1 Impairs Alveolar Epithelial Function via Endothelial ETB Receptor. American Journal of Respiratory and Critical Care Medicine. 179(2). 113–122. 34 indexed citations
20.
Vadász, István, Laura A. Dada, Arturo Briva, et al.. (2008). AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis. Journal of Clinical Investigation. 118(2). 752–62. 142 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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