Daniel J. Kass

4.8k total citations · 1 hit paper
77 papers, 2.6k citations indexed

About

Daniel J. Kass is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Epidemiology. According to data from OpenAlex, Daniel J. Kass has authored 77 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 60 papers in Pulmonary and Respiratory Medicine, 16 papers in Molecular Biology and 10 papers in Epidemiology. Recurrent topics in Daniel J. Kass's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (55 papers), Occupational and environmental lung diseases (9 papers) and Sarcoidosis and Beryllium Toxicity Research (8 papers). Daniel J. Kass is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (55 papers), Occupational and environmental lung diseases (9 papers) and Sarcoidosis and Beryllium Toxicity Research (8 papers). Daniel J. Kass collaborates with scholars based in United States, Canada and China. Daniel J. Kass's co-authors include Kevin F. Gibson, Naftali Kaminski, John Sembrat, Jiangning Tan, Robert Lafyatis, Mauricio Rojas, Humberto E. Trejo Bittar, Tracy Tabib, Steven R. Duncan and Mehdi Nouraie and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Daniel J. Kass

71 papers receiving 2.6k citations

Hit Papers

Proliferating SPP1/MERTK-expressing macrophages in idiopa... 2019 2026 2021 2023 2019 100 200 300 400
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Proliferating SPP1/MERTK-expressing macrophages in idiopathic pulmonary fibrosis
    2019 461 citations Christina Morse, John Sembrat et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniel J. Kass United States 26 1.7k 693 377 375 257 77 2.6k
Humberto E. Trejo Bittar United States 23 1.2k 0.7× 894 1.3× 415 1.1× 332 0.9× 214 0.8× 60 2.5k
Shuntaro Nagai Japan 28 1.3k 0.8× 642 0.9× 418 1.1× 654 1.7× 174 0.7× 95 2.6k
Carina Becerril Mexico 22 1.7k 1.0× 620 0.9× 226 0.6× 365 1.0× 132 0.5× 42 2.5k
Hugues Bégueret France 29 1.3k 0.8× 569 0.8× 317 0.8× 769 2.1× 139 0.5× 78 2.4k
Steven K. Huang United States 29 1.4k 0.8× 788 1.1× 259 0.7× 300 0.8× 139 0.5× 74 2.6k
Raphaël Borie France 35 1.9k 1.1× 791 1.1× 781 2.1× 688 1.8× 491 1.9× 182 3.7k
Αrgyris Τzouvelekis Greece 29 2.1k 1.2× 659 1.0× 171 0.5× 658 1.8× 263 1.0× 94 2.9k
Yoav H. Messinger United States 32 1.2k 0.7× 844 1.2× 221 0.6× 213 0.6× 297 1.2× 106 3.5k
Gary W. Mierau United States 27 911 0.5× 810 1.2× 194 0.5× 441 1.2× 386 1.5× 78 2.5k
Brigham C. Willis United States 13 1.5k 0.9× 705 1.0× 133 0.4× 201 0.5× 105 0.4× 30 2.4k

Countries citing papers authored by Daniel J. Kass

Since Specialization
Citations

This map shows the geographic impact of Daniel J. Kass's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel J. Kass with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel J. Kass more than expected).

Fields of papers citing papers by Daniel J. Kass

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel J. Kass. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel J. Kass. The network helps show where Daniel J. Kass may publish in the future.

Co-authorship network of co-authors of Daniel J. Kass

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel J. Kass. A scholar is included among the top collaborators of Daniel J. Kass based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel J. Kass. Daniel J. Kass is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Padilla, Cristina M., Robert Lafyatis, Kevin F. Gibson, et al.. (2025). Monocyte LOXHD1 and RHOB Expression Predictive of Progressive Systemic Sclerosis–Associated Interstitial Lung Disease. Arthritis Care & Research.
2.
Assayag, Deborah, Xiaoping Chen, Jolene H. Fisher, et al.. (2025). Particulate Matter Associations with Lung Function in Pulmonary Sarcoidosis. American Journal of Respiratory and Critical Care Medicine. 211(10). 1846–1854. 1 indexed citations
3.
Aggarwal, Rohit, Chester V. Oddis, Siamak Moghadam‐Kia, et al.. (2024). Design of a randomised controlled hybrid trial of nintedanib in patients with progressive myositis-associated interstitial lung disease. BMC Pulmonary Medicine. 24(1). 544–544. 4 indexed citations
4.
Clark, Kristopher, Mehdi Nouraie, Kathleen O. Lindell, et al.. (2024). A Ramped Treadmill Protocol Exercise Test Identifies Higher Ambulatory Oxygen Needs in Idiopathic Pulmonary Fibrosis and Chronic Obstructive Pulmonary Disease. Annals of the American Thoracic Society. 22(4). 541–548.
5.
Bulik, Melissa, Travis Lear, Yanwen Chen, et al.. (2022). Fibronectin-EDA accumulates via reduced ubiquitination downstream of Toll-like receptor 9 activation in SSc-ILD fibroblasts. American Journal of Physiology-Lung Cellular and Molecular Physiology. 323(4). L484–L494. 5 indexed citations
6.
Goobie, G.C., Mehdi Nouraie, Yingze Zhang, et al.. (2020). Air Pollution and Interstitial Lung Diseases: Defining Epigenomic Effects. American Journal of Respiratory and Critical Care Medicine. 202(9). 1217–1224. 22 indexed citations
7.
Valenzi, Eleanor, Haopu Yang, John Sembrat, et al.. (2020). Topographic heterogeneity of lung microbiota in end-stage idiopathic pulmonary fibrosis: the Microbiome in Lung Explants-2 (MiLEs-2) study. Thorax. 76(3). 239–247. 10 indexed citations
8.
Li, Xiaoyun, G.C. Goobie, Alyssa D. Gregory, Daniel J. Kass, & Yingze Zhang. (2020). Toll-Interacting Protein in Pulmonary Diseases. Abiding by the Goldilocks Principle. American Journal of Respiratory Cell and Molecular Biology. 64(5). 536–546. 20 indexed citations
9.
Xue, Jianmin, et al.. (2020). CD70 Activation Decreases Pulmonary Fibroblast Production of Extracellular Matrix Proteins. American Journal of Respiratory Cell and Molecular Biology. 63(2). 255–265. 3 indexed citations
10.
Iasella, Carlo J., Spencer Winters, Jaehee Cho, et al.. (2019). Idiopathic pulmonary fibrosis lung transplant recipients are at increased risk for EBV-associated posttransplant lymphoproliferative disorder and worse survival. American Journal of Transplantation. 20(5). 1439–1446. 9 indexed citations
11.
Li, Fu Jun, Ranu Surolia, Huashi Li, et al.. (2017). Autoimmunity to Vimentin Is Associated with Outcomes of Patients with Idiopathic Pulmonary Fibrosis. The Journal of Immunology. 199(5). 1596–1605. 65 indexed citations
12.
13.
Tan, Jiangning, John Tedrow, Brenda Juan-Guardela, et al.. (2016). Expression of RXFP1 Is Decreased in Idiopathic Pulmonary Fibrosis: Implications for Relaxin-based Therapies. American Journal of Respiratory and Critical Care Medicine. 194(11). 1392–1402. 45 indexed citations
14.
Christmann, Romy, Percival D. Sampaio‐Barros, Carlos Roberto Ribeiro de Carvalho, et al.. (2016). miR-155 in the progression of lung fibrosis in systemic sclerosis. Arthritis Research & Therapy. 18(1). 155–155. 85 indexed citations
15.
Ling, Nan, Jiangning Tan, Dan Wang, et al.. (2016). Ubiquitin carboxyl-terminal hydrolase-L5 promotes TGFβ-1 signaling by de-ubiquitinating and stabilizing Smad2/Smad3 in pulmonary fibrosis. Scientific Reports. 6(1). 33116–33116. 40 indexed citations
16.
Yu, Guoying, Jose D. Herazo‐Maya, Tomoko Nukui, et al.. (2014). Matrix Metalloproteinase-19 Promotes Metastatic Behavior In Vitro and is Associated with Increased Mortality in Non–Small Cell Lung Cancer. American Journal of Respiratory and Critical Care Medicine. 190(7). 780–790. 52 indexed citations
17.
Vuga, Louis J., John Tedrow, Kusum Pandit, et al.. (2014). C-X-C Motif Chemokine 13 (CXCL13) is a Prognostic Biomarker of Idiopathic Pulmonary Fibrosis. American Journal of Respiratory and Critical Care Medicine. 189(8). 966–974. 142 indexed citations
18.
Xue, Jianmin, Arpit Bhargava, Eva Csizmadia, et al.. (2012). Patients with Idiopathic Pulmonary Fibrosis with Antibodies to Heat Shock Protein 70 Have Poor Prognoses. American Journal of Respiratory and Critical Care Medicine. 187(7). 768–775. 134 indexed citations
19.
Yu, Guoying, Anil V. Parwani, Daniel J. Kass, et al.. (2012). Matrix Metalloproteinase-19 Is a Key Regulator of Lung Fibrosis in Mice and Humans. American Journal of Respiratory and Critical Care Medicine. 186(8). 752–762. 82 indexed citations
20.
Kass, Daniel J. & Naftali Kaminski. (2011). Evolving Genomic Approaches to Idiopathic Pulmonary Fibrosis: Moving Beyond Genes. Clinical and Translational Science. 4(5). 372–379. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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