Hui‐Chen Ku

1.4k total citations · 2 hit papers
20 papers, 1.0k citations indexed

About

Hui‐Chen Ku is a scholar working on Epidemiology, Pathology and Forensic Medicine and Physiology. According to data from OpenAlex, Hui‐Chen Ku has authored 20 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Epidemiology, 7 papers in Pathology and Forensic Medicine and 6 papers in Physiology. Recurrent topics in Hui‐Chen Ku's work include Adipokines, Inflammation, and Metabolic Diseases (10 papers), Tea Polyphenols and Effects (7 papers) and Adipose Tissue and Metabolism (5 papers). Hui‐Chen Ku is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (10 papers), Tea Polyphenols and Effects (7 papers) and Adipose Tissue and Metabolism (5 papers). Hui‐Chen Ku collaborates with scholars based in Taiwan and United States. Hui‐Chen Ku's co-authors include Ching‐Feng Cheng, Heng Lin, Shi-Wei Chao, Hsu‐Wen Chao, Chi-Wei Liu, Hsin-Huei Chang, Yung-Hsi Kao, Yung‐Hsi Kao, Li‐Jane Shih and Pei-Fang Hung and has published in prestigious journals such as International Journal of Molecular Sciences, American Journal of Physiology-Cell Physiology and American Journal of Physiology-Regulatory, Integrative and Comparative Physiology.

In The Last Decade

Hui‐Chen Ku

20 papers receiving 1.0k citations

Hit Papers

PGC-1α as a Pivotal Facto... 2018 2026 2020 2023 2018 2020 100 200 300

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Hui‐Chen Ku 454 311 283 139 113 20 1.0k
Yu‐Lan Yeh 542 1.2× 229 0.7× 190 0.7× 156 1.1× 80 0.7× 56 1.1k
Pei‐Ying Pai 772 1.7× 238 0.8× 329 1.2× 172 1.2× 119 1.1× 68 1.6k
Solaleh Emamgholipour 451 1.0× 397 1.3× 413 1.5× 115 0.8× 112 1.0× 75 1.2k
Bianca Knoch 782 1.7× 360 1.2× 294 1.0× 116 0.8× 49 0.4× 15 1.3k
Silvia Palmerio 505 1.1× 157 0.5× 257 0.9× 128 0.9× 81 0.7× 18 1.1k
James P. Stice 619 1.4× 170 0.5× 123 0.4× 98 0.7× 104 0.9× 20 1.1k
Daniel Tews 472 1.0× 501 1.6× 309 1.1× 187 1.3× 37 0.3× 42 1.1k
Qiren Huang 678 1.5× 220 0.7× 191 0.7× 80 0.6× 229 2.0× 71 1.3k
Maximilian Hatting 464 1.0× 191 0.6× 344 1.2× 114 0.8× 42 0.4× 22 1.0k

Countries citing papers authored by Hui‐Chen Ku

Since Specialization
Citations

This map shows the geographic impact of Hui‐Chen Ku's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hui‐Chen Ku with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hui‐Chen Ku more than expected).

Fields of papers citing papers by Hui‐Chen Ku

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hui‐Chen Ku. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hui‐Chen Ku. The network helps show where Hui‐Chen Ku may publish in the future.

Co-authorship network of co-authors of Hui‐Chen Ku

This figure shows the co-authorship network connecting the top 25 collaborators of Hui‐Chen Ku. A scholar is included among the top collaborators of Hui‐Chen Ku based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hui‐Chen Ku. Hui‐Chen Ku is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Chen, Wenting, Yow‐Chii Kuo, Yi Chen, et al.. (2023). Green Tea Epigallocatechin Gallate Inhibits Preadipocyte Growth via the microRNA‐let‐7a/HMGA2 Signaling Pathway. Molecular Nutrition & Food Research. 67(9). e2200336–e2200336. 5 indexed citations
3.
Chen, Wenting, Yen‐Yue Lin, Yow‐Chii Kuo, et al.. (2022). Green tea epigallocatechin gallate suppresses 3T3-L1 cell growth via microRNA-143/MAPK7 pathways. Experimental Biology and Medicine. 247(18). 1670–1679. 8 indexed citations
4.
Shih, Li‐Jane, et al.. (2021). Investigation of the Molecular Mechanisms by Which Endothelin-3 Stimulates Preadipocyte Growth. Frontiers in Endocrinology. 12. 661828–661828. 1 indexed citations
5.
Ku, Hui‐Chen, et al.. (2021). The ATF3 inducer protects against diet-induced obesity via suppressing adipocyte adipogenesis and promoting lipolysis and browning. Biomedicine & Pharmacotherapy. 145. 112440–112440. 13 indexed citations
6.
Ku, Hui‐Chen, et al.. (2021). The potential of using itaconate as treatment for inflammation-related heart diseases. Tzu Chi Medical Journal. 34(2). 113–118. 4 indexed citations
7.
Cheng, Ching‐Feng, et al.. (2021). Erythrocyte degradation, metabolism, secretion, and communication with immune cells in the blood during sepsis. Tzu Chi Medical Journal. 34(2). 125–133. 7 indexed citations
8.
Ku, Hui‐Chen & Ching‐Feng Cheng. (2020). Master Regulator Activating Transcription Factor 3 (ATF3) in Metabolic Homeostasis and Cancer. Frontiers in Endocrinology. 11. 556–556. 232 indexed citations breakdown →
9.
Chao, Hsu‐Wen, Shi-Wei Chao, Heng Lin, Hui‐Chen Ku, & Ching‐Feng Cheng. (2019). Homeostasis of Glucose and Lipid in Non-Alcoholic Fatty Liver Disease. International Journal of Molecular Sciences. 20(2). 298–298. 134 indexed citations
10.
Cheng, Ching‐Feng, Hui‐Chen Ku, Jing‐Jy Cheng, et al.. (2019). Adipocyte browning and resistance to obesity in mice is induced by expression of ATF3. Communications Biology. 2(1). 389–389. 48 indexed citations
11.
Cheng, Ching‐Feng, Hui‐Chen Ku, & Heng Lin. (2018). PGC-1α as a Pivotal Factor in Lipid and Metabolic Regulation. International Journal of Molecular Sciences. 19(11). 3447–3447. 338 indexed citations breakdown →
13.
Liu, Chi-Wei, Low‐Tone Ho, Liang-Yi Wu, et al.. (2013). The forkhead transcription factor FOXO1 stimulates the expression of the adipocyte resistin gene. General and Comparative Endocrinology. 196. 41–51. 9 indexed citations
15.
Ku, Hui‐Chen, Chia‐Lin Chen, Li‐Jane Shih, et al.. (2012). Green tea (–)‐epigallocatechin gallate inhibits IGFI and IGFIIstimulation of 3T3‐L1 preadipocyte mitogenesis via the 67‐kDa laminin receptor, but not AMP‐activated protein kinase pathway. Molecular Nutrition & Food Research. 56(4). 580–592. 38 indexed citations
16.
Liu, Chi-Wei, Li‐Jane Shih, Low‐Tone Ho, et al.. (2010). Green Tea Epigallocatechin Gallate Inhibits Insulin Stimulation of Adipocyte Glucose Uptake via the 67-Kilodalton Laminin Receptor and AMP-Activated Protein Kinase Pathways. Planta Medica. 76(15). 1694–1698. 24 indexed citations
17.
Ku, Hui‐Chen, et al.. (2009). Green tea (−)-epigallocatechin gallate inhibits insulin stimulation of 3T3-L1 preadipocyte mitogenesis via the 67-kDa laminin receptor pathway. American Journal of Physiology-Cell Physiology. 297(1). C121–C132. 52 indexed citations
18.
Lin, Heng, Chi-Wei Liu, Wei‐Ju Liao, et al.. (2008). Octylphenol stimulates resistin gene expression in 3T3-L1 adipocytes via the estrogen receptor and extracellular signal-regulated kinase pathways. American Journal of Physiology-Cell Physiology. 294(6). C1542–C1551. 29 indexed citations
20.
Ku, Hui‐Chen, et al.. (2006). A preprogalanin cDNA from the turtle pituitary and regulation of its gene expression. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 292(4). R1649–R1656. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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