H.R. Widmer

436 total citations
9 papers, 363 citations indexed

About

H.R. Widmer is a scholar working on Cellular and Molecular Neuroscience, Developmental Neuroscience and Molecular Biology. According to data from OpenAlex, H.R. Widmer has authored 9 papers receiving a total of 363 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cellular and Molecular Neuroscience, 5 papers in Developmental Neuroscience and 4 papers in Molecular Biology. Recurrent topics in H.R. Widmer's work include Nerve injury and regeneration (7 papers), Neurogenesis and neuroplasticity mechanisms (5 papers) and RNA Interference and Gene Delivery (2 papers). H.R. Widmer is often cited by papers focused on Nerve injury and regeneration (7 papers), Neurogenesis and neuroplasticity mechanisms (5 papers) and RNA Interference and Gene Delivery (2 papers). H.R. Widmer collaborates with scholars based in Switzerland, Denmark and Germany. H.R. Widmer's co-authors include Franz Hefti, Rolf W. Seiler, Christian Spenger, Morten Meyer, J. Sautter, Jack Tseng, Anne D. Zurn, Patrick Aebischer, Günter U. Höglinger and Alberto Pérez-Bouza and has published in prestigious journals such as PLoS ONE, Brain Research and Neuroscience.

In The Last Decade

H.R. Widmer

9 papers receiving 359 citations

Peers

H.R. Widmer

CMN

DN

MB

NEURO

CN

Kotaro Shimoda United States

Tien Manh Tran United States

Ad J. Dekker United States

Thomas J. Mahalik United States

Chumasov Ei Russia

Isao Date Japan

Jo Ann McConnell United States

Farida G. Kaddis United States

Thomas Andreska Germany

Takeshi Hashitani Japan

Kotaro Shimoda United States

H.R. Widmer

244 ×0.9

CMN

114 ×0.8

DN

175 ×1.4

MB

63 ×0.9

NEURO

44 ×0.7

CN

Citations per year, relative to H.R. Widmer H.R. Widmer (= 1×) peers Kotaro Shimoda

Countries citing papers authored by H.R. Widmer

Since Specialization

Citations

This map shows the geographic impact of H.R. Widmer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H.R. Widmer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H.R. Widmer more than expected).

Fields of papers citing papers by H.R. Widmer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H.R. Widmer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H.R. Widmer. The network helps show where H.R. Widmer may publish in the future.

Co-authorship network of co-authors of H.R. Widmer

This figure shows the co-authorship network connecting the top 25 collaborators of H.R. Widmer. A scholar is included among the top collaborators of H.R. Widmer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H.R. Widmer. H.R. Widmer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Fadda, Angela, Andrew Hemphill, H.R. Widmer, et al.. (2016). Primary Postnatal Dorsal Root Ganglion Culture from Conventionally Slaughtered Calves. PLoS ONE. 11(12). e0168228–e0168228. 9 indexed citations

2.

Andres, Robert H., Uwe Schlattner, Alberto Pérez-Bouza, et al.. (2005). Effects of creatine treatment on the survival of dopaminergic neurons in cultured fetal ventral mesencephalic tissue. Neuroscience. 133(3). 701–713. 62 indexed citations

3.

Bauer, M., Morten Meyer, Christian Heßlinger, et al.. (2002). Glial cell line‐derived neurotrophic factor up‐regulates GTP‐cyclohydrolase I activity and tetrahydrobiopterin levels in primary dopaminergic neurones. Journal of Neurochemistry. 82(5). 1300–1310. 21 indexed citations

4.

Meyer, Morten, Esperanza R. Matarredona, Roland Seiler, Jens Zimmer, & H.R. Widmer. (2001). Additive effect of glial cell line-derived neurotrophic factor and neurotrophin-4/5 on rat fetal nigral explant cultures. Neuroscience. 108(2). 273–284. 36 indexed citations

5.

Höglinger, Günter U., H.R. Widmer, Christian Spenger, et al.. (2001). Influence of Time in Culture and BDNF Pretreatment on Survival and Function of Grafted Embryonic Rat Ventral Mesencephalon in the 6-OHDA Rat Model of Parkinson's Disease. Experimental Neurology. 167(1). 148–157. 24 indexed citations

6.

Sautter, J., Jack Tseng, Patrick Aebischer, et al.. (1998). Implants of Polymer-Encapsulated Genetically Modified Cells Releasing Glial Cell Line-Derived Neurotrophic Factor Improve Survival, Growth, and Function of Fetal Dopaminergic Grafts. Experimental Neurology. 149(1). 230–236. 93 indexed citations

7.

Höglinger, Günter U., Jürgen Sautter, Morten Meyer, et al.. (1998). Rat fetal ventral mesencephalon grown as solid tissue cultures: influence of culture time and BDNF treatment on dopamine neuron survival and function. Brain Research. 813(2). 313–322. 45 indexed citations

8.

Widmer, H.R. & Franz Hefti. (1994). Stimulation of GABAergic neuron differentiation by NT-4/5 in cultures of rat cerebral cortex. Developmental Brain Research. 80(1-2). 279–284. 54 indexed citations

9.

Widmer, H.R., et al.. (1993). TGFα stimulation of phosphatidylinositol hydrolysis in mesencephalic cultures requires neuron-glia interactions. Neuroreport. 4(4). 407–410. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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