Hiroyuki Imuta
- Molecular Biology
- Cardiology and Cardiovascular Medicine
- Complementary and alternative medicine top 5%
- Physiology
- Pulmonary and Respiratory Medicine
- Co-authors
- Toshiaki NakajimaYasushi KawakamiKuniaki IwasawaHitoshi OonumaTaeho JoMasao OmataFumiki KatsuokaTakeo Arai
- Topics
- Ion channel regulation and function (6 papers)Cardiac electrophysiology and arrhythmias (4 papers)Nitric Oxide and Endothelin Effects (3 papers)
- Cited by
- Complementary and alternative medicineClinical BiochemistryCardiology and Cardiovascular Medicine
- Partner nations
- JapanUnited States
In The Last Decade
Hiroyuki Imuta
15 papers receiving 417 citations
Peers
Comparison fields: 5 of 79
- Molecular Biology 193
- Cardiology and Cardiovascular Medicine 138
- Complementary and alternative medicine 72
- Physiology 72
- Pulmonary and Respiratory Medicine 56
Countries citing papers authored by Hiroyuki Imuta
This map shows the geographic impact of Hiroyuki Imuta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroyuki Imuta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroyuki Imuta more than expected).
Fields of papers citing papers by Hiroyuki Imuta
This network shows the impact of papers produced by Hiroyuki Imuta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroyuki Imuta. The network helps show where Hiroyuki Imuta may publish in the future.
Co-authorship network of co-authors of Hiroyuki Imuta
This figure shows the co-authorship network connecting the top 25 collaborators of Hiroyuki Imuta. A scholar is included among the top collaborators of Hiroyuki Imuta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroyuki Imuta. Hiroyuki Imuta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 3 | |
| 3 | 80 | |
| 4 | 43 | |
| 5 | 13 | |
| 6 | 18 | |
| 7 | 5 | |
| 8 | 10 | |
| 9 | 16 | |
| 10 | 45 | |
| 11 | 13 | |
| 12 | 16 | |
| 13 | 37 | |
| 14 | 35 | |
| 15 | 84 |
About Hiroyuki Imuta
Hiroyuki Imuta is a scholar working on Biophysics, Biochemistry and Cardiology and Cardiovascular Medicine, having authored 15 papers that have together received 424 indexed citations. Recurring topics across this work include Ion channel regulation and function (6 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Nitric Oxide and Endothelin Effects (3 papers). The work is most often cited by research in Complementary and alternative medicine (72 citations), Clinical Biochemistry (55 citations) and Cardiology and Cardiovascular Medicine (138 citations). Hiroyuki Imuta has collaborated with scholars based in Japan and United States. Frequent co-authors include Toshiaki Nakajima, Yasushi Kawakami, Kuniaki Iwasawa, Hitoshi Oonuma, Taeho Jo, Masao Omata, Fumiki Katsuoka, Takeo Arai, Kamejiro Yamashita and Masachika Fujiwara. Their work appears in journals such as Circulation, Biochemical and Biophysical Research Communications and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.