Hiroshi Sato

5.3k total citations · 2 hit papers
42 papers, 4.6k citations indexed

About

Hiroshi Sato is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Hiroshi Sato has authored 42 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 9 papers in Oncology and 9 papers in Genetics. Recurrent topics in Hiroshi Sato's work include Immune Cell Function and Interaction (8 papers), Mitochondrial Function and Pathology (5 papers) and Mesenchymal stem cell research (5 papers). Hiroshi Sato is often cited by papers focused on Immune Cell Function and Interaction (8 papers), Mitochondrial Function and Pathology (5 papers) and Mesenchymal stem cell research (5 papers). Hiroshi Sato collaborates with scholars based in Japan and United States. Hiroshi Sato's co-authors include Eisuke Kondo, Haruhiko Koseki, Tetsu Kawano, Masaru Taniguchi, Isao Toura, Yoshikatsu Kaneko, Junqing Cui, Yasuhiko Koezuka, Hitomi Ueno and Ryusuke Nakagawa and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Biochemical and Biophysical Research Communications.

In The Last Decade

Hiroshi Sato

39 papers receiving 4.5k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

CD1d-Restricted and TCR-Mediated Activation of V _α 14 NKT Cells by Glycosylceramides

1997 2.1k citations Hiroshi Sato, Haruhiko Koseki et al. profile →
Requirement for V _α 14 NKT Cells in IL-12-Mediated Rejection of Tumors

1997 1.1k citations Hiroshi Sato, Haruhiko Koseki et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hiroshi Sato Japan	18	3.6k	1.0k	843	345	229	42	4.6k
Junqing Cui Japan	13	3.8k 1.0×	979 1.0×	675 0.8×	294 0.9×	82 0.4×	14	4.4k
Hui‐Chen Hsu United States	34	2.1k 0.6×	649 0.6×	1.4k 1.6×	342 1.0×	234 1.0×	102	3.9k
Jian Hong China	38	1.5k 0.4×	841 0.8×	1.3k 1.6×	453 1.3×	417 1.8×	120	3.9k
Azzam A. Maghazachi Norway	37	2.5k 0.7×	1.0k 1.0×	1.2k 1.4×	289 0.8×	177 0.8×	114	4.2k
Huabao Xiong United States	24	2.3k 0.6×	958 0.9×	927 1.1×	259 0.8×	154 0.7×	61	3.3k
Shunsuke Chikuma Japan	31	2.6k 0.7×	1.8k 1.8×	995 1.2×	307 0.9×	211 0.9×	56	4.3k
David Chantry United States	35	3.0k 0.8×	1.8k 1.8×	1.1k 1.3×	397 1.2×	273 1.2×	74	5.5k
Angela Gismondi Italy	33	2.0k 0.6×	638 0.6×	652 0.8×	195 0.6×	190 0.8×	95	3.2k
Réjean Lapointe Canada	30	2.1k 0.6×	1.2k 1.2×	1.0k 1.2×	372 1.1×	221 1.0×	80	4.0k
Stephan Gasser Singapore	29	3.2k 0.9×	1.4k 1.4×	1.3k 1.6×	436 1.3×	281 1.2×	50	4.4k

Countries citing papers authored by Hiroshi Sato

Since Specialization

Citations

This map shows the geographic impact of Hiroshi Sato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroshi Sato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroshi Sato more than expected).

Fields of papers citing papers by Hiroshi Sato

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroshi Sato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroshi Sato. The network helps show where Hiroshi Sato may publish in the future.

Co-authorship network of co-authors of Hiroshi Sato

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroshi Sato. A scholar is included among the top collaborators of Hiroshi Sato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroshi Sato. Hiroshi Sato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Chiba, Yuta, Hiroshi Sato, Yumiko Nakashima, et al.. (2025). Expression patterns of desmosome family members during tooth development and the role of Desmocollin-3 in cytodifferentiation of stratum intermedium. Archives of Oral Biology. 180. 106404–106404.

Sun, Xiao, Jun Kong, Hiroki Kato, et al.. (2023). TRPV4‐mediated Ca²⁺ deregulation causes mitochondrial dysfunction via the AKT/α‐synuclein pathway in dopaminergic neurons. FASEB BioAdvances. 5(12). 507–520. 6 indexed citations

Sun, Xiao, Hiroki Kato, Jun Kong, et al.. (2022). Mitochondrial Calcium-Triggered Oxidative Stress and Developmental Defects in Dopaminergic Neurons Differentiated from Deciduous Teeth-Derived Dental Pulp Stem Cells with MFF Insufficiency. Antioxidants. 11(7). 1361–1361. 9 indexed citations

Masuda, Keiji, Xu Han, Hiroki Kato, et al.. (2021). Dental Pulp-Derived Mesenchymal Stem Cells for Modeling Genetic Disorders. International Journal of Molecular Sciences. 22(5). 2269–2269. 28 indexed citations

Sun, Xiao, Hiroki Kato, Hiroshi Sato, et al.. (2021). Impaired neurite development and mitochondrial dysfunction associated with calcium accumulation in dopaminergic neurons differentiated from the dental pulp stem cells of a patient with metatropic dysplasia. Biochemistry and Biophysics Reports. 26. 100968–100968. 5 indexed citations

Nguyen, Huong, Hiroki Kato, Hiroshi Sato, et al.. (2019). Positive effect of exogenous brain-derived neurotrophic factor on impaired neurite development and mitochondrial function in dopaminergic neurons derived from dental pulp stem cells from children with attention deficit hyperactivity disorder. Biochemical and Biophysical Research Communications. 513(4). 1048–1054. 15 indexed citations

Pham, Thanh, Hiroki Kato, Haruyoshi Yamaza, et al.. (2018). Altered development of dopaminergic neurons differentiated from stem cells from human exfoliated deciduous teeth of a patient with Down syndrome. BMC Neurology. 18(1). 132–132. 9 indexed citations

Han, Xu, et al.. (2018). Osteoblastic differentiation improved by bezafibrate-induced mitochondrial biogenesis in deciduous tooth-derived pulp stem cells from a child with Leigh syndrome. Biochemistry and Biophysics Reports. 17. 32–37. 10 indexed citations

Zhang, Yu, Hiroki Kato, Hiroshi Sato, et al.. (2018). Folic acid-mediated mitochondrial activation for protection against oxidative stress in human dental pulp stem cells derived from deciduous teeth. Biochemical and Biophysical Research Communications. 508(3). 850–856. 16 indexed citations

10.

Kato, Hiroki, Keiji Masuda, Haruyoshi Yamaza, et al.. (2018). Mitochondrial dysfunction in dopaminergic neurons differentiated from exfoliated deciduous tooth-derived pulp stem cells of a child with Rett syndrome. Biochemical and Biophysical Research Communications. 498(4). 898–904. 16 indexed citations

11.

Nguyen, Huong, Hiroki Kato, Keiji Masuda, et al.. (2018). Impaired neurite development associated with mitochondrial dysfunction in dopaminergic neurons differentiated from exfoliated deciduous tooth-derived pulp stem cells of children with autism spectrum disorder. Biochemistry and Biophysics Reports. 16. 24–31. 27 indexed citations

12.

Kato, Hiroki, Keiji Masuda, Haruyoshi Yamaza, et al.. (2017). Accelerated dentinogenesis by inhibiting the mitochondrial fission factor, dynamin related protein 1. Biochemical and Biophysical Research Communications. 495(2). 1655–1660. 6 indexed citations

13.

Sato, Hiroshi, Hiroki Kato, Haruyoshi Yamaza, et al.. (2017). Engineering of Systematic Elimination of a Targeted Chromosome in Human Cells. BioMed Research International. 2017. 1–5. 9 indexed citations

14.

Kato, Hiroki, Xu Han, Haruyoshi Yamaza, et al.. (2017). Direct effects of mitochondrial dysfunction on poor bone health in Leigh syndrome. Biochemical and Biophysical Research Communications. 493(1). 207–212. 17 indexed citations

15.

Fukuda, Katsuyuki, Hisahiro Yoshida, Tôru Satô, et al.. (2003). Mesenchymal expression of Foxl1, a winged helix transcriptional factor, regulates generation and maintenance of gut-associated lymphoid organs. Developmental Biology. 255(2). 278–289. 18 indexed citations

16.

Rumi, M. A. Karim, Hiroshi Sato, Shunji Ishihara, et al.. (2002). Growth inhibition of esophageal squamous carcinoma cells by peroxisome proliferator-activated receptor-γ ligands. Journal of Laboratory and Clinical Medicine. 140(1). 17–26. 25 indexed citations

17.

Makino, Yasuhiko, Donald N. Cook, Oliver Smithies, et al.. (2002). Impaired T Cell Function in RANTES-Deficient Mice. Clinical Immunology. 102(3). 302–309. 103 indexed citations

18.

Sato, Hiroshi, Shunji Ishihara, Kousaku Kawashima, et al.. (2000). Expression of peroxisome proliferator-activated receptor (PPAR)γ in gastric cancer and inhibitory effects of PPARγ agonists. British Journal of Cancer. 83(10). 1394–1400. 191 indexed citations

19.

Aoyagi, Kunihiko, et al.. (1997). A case of multiple gastric carcinoids associated with type A gastritis. Acta gastro-enterologica belgica. 39(3). 659–664. 3 indexed citations

20.

Morisaki, Takashi, Mitsuo Katano, Akashi Ikubo, et al.. (1996). Immunosuppressive cytokines (IL-10, TGF-β) genes expression in human gastric carcinoma tissues. Journal of Surgical Oncology. 63(4). 234–239. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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