Hiroshi Nara
- Molecular Biology top 10%
- Oncology top 10%
- Hematology top 5%
- Cancer Research
- Organic Chemistry
- Co-authors
- Nobuo ChoTakayuki HattoriMikihiko NaitoNobumichi OhokaNorihito ShibataOsamu UjikawaKenichiro ShimokawaKatsunori Nagai
- Topics
- Protein Degradation and Inhibitors (8 papers)Ubiquitin and proteasome pathways (5 papers)Peptidase Inhibition and Analysis (4 papers)
- Cited by
- HematologyOncologyMolecular Biology
- Partner nations
- JapanUnited StatesChina
In The Last Decade
Hiroshi Nara
25 papers receiving 997 citations
Peers
Comparison fields: 5 of 72
- Molecular Biology 868
- Oncology 378
- Hematology 292
- Cancer Research 100
- Organic Chemistry 90
Countries citing papers authored by Hiroshi Nara
This map shows the geographic impact of Hiroshi Nara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroshi Nara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroshi Nara more than expected).
Fields of papers citing papers by Hiroshi Nara
This network shows the impact of papers produced by Hiroshi Nara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroshi Nara. The network helps show where Hiroshi Nara may publish in the future.
Co-authorship network of co-authors of Hiroshi Nara
This figure shows the co-authorship network connecting the top 25 collaborators of Hiroshi Nara. A scholar is included among the top collaborators of Hiroshi Nara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroshi Nara. Hiroshi Nara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 21 | |
| 2 | 84 | |
| 3 | 46 | |
| 4 | 193 | |
| 5 | 120 | |
| 6 | 91 | |
| 7 | 90 | |
| 8 | 27 | |
| 9 | 42 | |
| 10 | 37 | |
| 11 | 5 | |
| 12 | [Toxicity study of cefmatilen hydrochloride hydrate (S-1090) (8)--Nephrotoxicity study in rabbits by single oral administration]. | 4 |
| 13 | 5 | |
| 14 | 33 | |
| 15 | 47 | |
| 16 | [A phase II study of SM-5887 for advanced gastric cancer]. | 1 |
| 17 | 1 | |
| 18 | 7 | |
| 19 | [Clinical evaluation of cefotiam in internal medicine]. | 4 |
| 20 | 4 |
About Hiroshi Nara
Hiroshi Nara is a scholar working on Cancer Research, Oncology and Hematology, having authored 26 papers that have together received 1.0k indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (8 papers), Ubiquitin and proteasome pathways (5 papers) and Peptidase Inhibition and Analysis (4 papers). The work is most often cited by research in Hematology (292 citations), Oncology (378 citations) and Molecular Biology (868 citations). Hiroshi Nara has collaborated with scholars based in Japan, United States and China. Frequent co-authors include Nobuo Cho, Takayuki Hattori, Mikihiko Naito, Nobumichi Ohoka, Norihito Shibata, Osamu Ujikawa, Kenichiro Shimokawa, Katsunori Nagai, Yasuhiro Imaeda and Tomoya Sameshima. Their work appears in journals such as Journal of Biological Chemistry, Scientific Reports and Journal of Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.