Hiroshi Mizukami
- Cell Biology top 2%
- Molecular Biology
- Physiology top 5%
- Pediatrics, Perinatology and Child Health top 10%
- Genetics top 10%
- Co-authors
- Robert A. GardinerTeddy G. TraylorVijay S. SharmaAgnes OstafinFei LiuRobert S. EliotA.E. Romero-HerreraJames M. Salhany
- Topics
- Hemoglobin structure and function (23 papers)Erythrocyte Function and Pathophysiology (11 papers)Protein Structure and Dynamics (8 papers)
- Cited by
- Cell BiologyPhysiologyBiophysics
- Partner nations
- United StatesJapanGermany
In The Last Decade
Hiroshi Mizukami
46 papers receiving 879 citations
Peers
Comparison fields: 5 of 97
- Cell Biology 534
- Molecular Biology 384
- Physiology 367
- Pediatrics, Perinatology and Child Health 126
- Genetics 92
Countries citing papers authored by Hiroshi Mizukami
This map shows the geographic impact of Hiroshi Mizukami's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroshi Mizukami with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroshi Mizukami more than expected).
Fields of papers citing papers by Hiroshi Mizukami
This network shows the impact of papers produced by Hiroshi Mizukami. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroshi Mizukami. The network helps show where Hiroshi Mizukami may publish in the future.
Co-authorship network of co-authors of Hiroshi Mizukami
This figure shows the co-authorship network connecting the top 25 collaborators of Hiroshi Mizukami. A scholar is included among the top collaborators of Hiroshi Mizukami based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroshi Mizukami. Hiroshi Mizukami is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 10 | |
| 3 | 1 | |
| 4 | 1 | |
| 5 | 50 | |
| 6 | 49 | |
| 7 | 18 | |
| 8 | 8 | |
| 9 | 16 | |
| 10 | 13 | |
| 11 | 10 | |
| 12 | 14 | |
| 13 | 4 | |
| 14 | 203 | |
| 15 | 1 | |
| 16 | Band-8 protein of human erythrocyte membrane: another Ca++ binding protein? | 3 |
| 17 | 37 | |
| 18 | Kinetics of sickling of intact erythrocytes. | 1 |
| 19 | 13 | |
| 20 | 4 |
About Hiroshi Mizukami
Hiroshi Mizukami is a scholar working on Cell Biology, Bioengineering and Genetics, having authored 46 papers that have together received 922 indexed citations. Recurring topics across this work include Hemoglobin structure and function (23 papers), Erythrocyte Function and Pathophysiology (11 papers) and Protein Structure and Dynamics (8 papers). The work is most often cited by research in Cell Biology (534 citations), Physiology (367 citations) and Biophysics (60 citations). Hiroshi Mizukami has collaborated with scholars based in United States, Japan and Germany. Frequent co-authors include Robert A. Gardiner, Teddy G. Traylor, Vijay S. Sharma, Agnes Ostafin, Fei Liu, Robert S. Eliot, A.E. Romero-Herrera, James M. Salhany, Oscar H. Kapp and Serge N. Vinogradov. Their work appears in journals such as Journal of Biological Chemistry, Circulation and Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.