Hideyuki Ishikawa

446 citations
24 papers · 338 indexed · h-index 9
Topics
Drug-Induced Adverse Reactions (3 papers)Phosphorus and nutrient management (3 papers)Immunodeficiency and Autoimmune Disorders (2 papers)

In The Last Decade

Hideyuki Ishikawa

22 papers receiving 312 citations

Peers

Hideyuki Ishikawa
Comparison fields: 5 of 75
  • Mechanical Engineering 154
  • Materials Chemistry 132
  • Industrial and Manufacturing Engineering 49
  • Aerospace Engineering 48
  • Mechanics of Materials 39
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Citations per field
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Countries citing papers authored by Hideyuki Ishikawa

Since Specialization
Citations

This map shows the geographic impact of Hideyuki Ishikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideyuki Ishikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideyuki Ishikawa more than expected).

Fields of papers citing papers by Hideyuki Ishikawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideyuki Ishikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideyuki Ishikawa. The network helps show where Hideyuki Ishikawa may publish in the future.

Co-authorship network of co-authors of Hideyuki Ishikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Hideyuki Ishikawa. A scholar is included among the top collaborators of Hideyuki Ishikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hideyuki Ishikawa. Hideyuki Ishikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 0
3 21
4 2
5 18
6 3
7 4
8 8
9 9
10 34
11 5
12 6
13 5
14 32
15 1
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[Combined therapies with medroxyprogesterone acetate (MPA) in DMBA-induced rat mammary cancer].
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17 3
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[Effect of medroxyprogesterone acetate (MAP) and vitamin C on DMBA induced rat mammary cancer in relation to estrogen receptor(ER)].
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19 26
20 1

About Hideyuki Ishikawa

Hideyuki Ishikawa is a scholar working on Industrial and Manufacturing Engineering, Dermatology and Fluid Flow and Transfer Processes, having authored 24 papers that have together received 338 indexed citations. Recurring topics across this work include Drug-Induced Adverse Reactions (3 papers), Phosphorus and nutrient management (3 papers) and Immunodeficiency and Autoimmune Disorders (2 papers). The work is most often cited by research in Industrial and Manufacturing Engineering (49 citations), Mechanical Engineering (154 citations) and Materials Chemistry (132 citations). Hideyuki Ishikawa has collaborated with scholars based in Japan, Germany and United States. Frequent co-authors include Farghalli A. Mohamed, Terence G. Langdon, Toshihiro Tanaka, Yuji Miura, Michiko Aihara, Izumi Hirasawa, Kazunori Ohnishi, Naoko Inomata, Setsuko Matsukura and Yoshiaki Hamamoto. Their work appears in journals such as SHILAP Revista de lepidopterología, Journal of Cleaner Production and International Journal of Hydrogen Energy.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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