Hidetaka Sato

3.4k total citations · 1 hit paper
83 papers, 2.6k citations indexed

About

Hidetaka Sato is a scholar working on Molecular Biology, Cancer Research and Neurology. According to data from OpenAlex, Hidetaka Sato has authored 83 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 19 papers in Cancer Research and 15 papers in Neurology. Recurrent topics in Hidetaka Sato's work include Carcinogens and Genotoxicity Assessment (11 papers), Traumatic Brain Injury and Neurovascular Disturbances (11 papers) and Drug Transport and Resistance Mechanisms (7 papers). Hidetaka Sato is often cited by papers focused on Carcinogens and Genotoxicity Assessment (11 papers), Traumatic Brain Injury and Neurovascular Disturbances (11 papers) and Drug Transport and Resistance Mechanisms (7 papers). Hidetaka Sato collaborates with scholars based in Japan, United States and United Kingdom. Hidetaka Sato's co-authors include Gou Young Koh, Toshio Suda, Keisuke Ito, Atsushi Hirao, Keiyo Takubo, Sahoko Matsuoka, Masako Ohmura, Fumio Arai, Yûzo Hayashi and Kazuhiro Toyoda and has published in prestigious journals such as Cell, SHILAP Revista de lepidopterología and Biochemistry.

In The Last Decade

Hidetaka Sato

78 papers receiving 2.5k citations

Hit Papers

Tie2/Angiopoietin-1 Signaling Regulates Hematopoietic Ste... 2004 2026 2011 2018 2004 400 800 1.2k

Peers

Hidetaka Sato
John W. Forstrom United States
Weina Chen United States
Xi Huang United States
J Caen France
John W. Forstrom United States
Hidetaka Sato
Citations per year, relative to Hidetaka Sato Hidetaka Sato (= 1×) peers John W. Forstrom

Countries citing papers authored by Hidetaka Sato

Since Specialization
Citations

This map shows the geographic impact of Hidetaka Sato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hidetaka Sato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hidetaka Sato more than expected).

Fields of papers citing papers by Hidetaka Sato

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hidetaka Sato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hidetaka Sato. The network helps show where Hidetaka Sato may publish in the future.

Co-authorship network of co-authors of Hidetaka Sato

This figure shows the co-authorship network connecting the top 25 collaborators of Hidetaka Sato. A scholar is included among the top collaborators of Hidetaka Sato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hidetaka Sato. Hidetaka Sato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Iwase, Haruko, Takeshi Hirasawa, Hidetaka Sato, et al.. (2018). The Clinical Features of Recurrent EndometrialCancer in Japan. International Journal of Gynecological Cancer. 28(8). 1616–1623. 9 indexed citations
2.
Sato, Hidetaka, et al.. (2013). Pathophysiology, Mortality, Treatment of Acute Phase of Haemostatic Disorders of Traumatic Brain Injury. Japanese Journal of Neurosurgery. 22(11). 837–841. 7 indexed citations
3.
Mizumura, Sunao, Ryusuke Murakami, Hidetaka Sato, et al.. (2013). Voxel-based analysis of 201Tl SPECT for grading and diagnostic accuracy of gliomas: comparison with ROI analysis. Annals of Nuclear Medicine. 27(6). 493–501. 4 indexed citations
4.
Kojima, Satoko, Kei Kawana, Tomoyuki Fujii, et al.. (2011). Characterization of Gut-Derived Intraepithelial Lymphocyte (IEL) Residing in Human Papillomavirus (HPV)-Infected Intraepithelial Neoplastic Lesions. American Journal of Reproductive Immunology. 66(5). 435–443. 14 indexed citations
6.
Murai, Yasuo, Yukio Ikeda, Hidetaka Sato, Yasuhiro Yamamoto, & Akira Teramoto. (2005). Anomalous Internal Carotid Anastomosis to Contralateral Anterior Cerebral Artery. Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques. 32(3). 359–360. 1 indexed citations
7.
Onozawa, Mizuki, Toshihiko Kawamori, Masaki Baba, et al.. (1999). Effects of a Soybean Isoflavone Mixture on Carcinogenesis in Prostate and Seminal Vesicles of F344 Rats. Japanese Journal of Cancer Research. 90(4). 393–398. 65 indexed citations
8.
Ushijima, Toshikazu, Keiji Wakabayashi, Hidetaka Sato, et al.. (1996). Rare Activation of the Human c‐Ha‐ras Transgene of Mice in Hemangioendothelial Sarcomas and Liver Tumors Induced by Glu‐P‐1. Japanese Journal of Cancer Research. 87(6). 583–588. 6 indexed citations
9.
Hirose, M, Keiji Wakabayashi, Masako Ochiai, et al.. (1995). Formation and Removal of DNA Adducts in the Liver of Rats Chronically Fed the Food‐borne Carcinogen, 2‐Amino‐3,8‐dimethylimidazo[4,5‐f]quinoxaline. Japanese Journal of Cancer Research. 86(6). 516–522. 14 indexed citations
10.
Toyoda, Kazuhiro, Katsumi Imaida, Kunitoshi Mitsumori, et al.. (1992). Correlation between cataract and retinopathy due to lighting in F344 rats used in a long‐term carcinogenicity study. Journal of Toxicology and Environmental Health. 37(4). 495–509. 6 indexed citations
11.
Sirbasku, David A., et al.. (1992). Thyroid hormone and apotransferrin regulation of growth hormone secretion by GH1 rat pituitary tumor cells in iron restricted serum-free defined medium. In Vitro Cellular & Developmental Biology - Animal. 28(1). 67–71. 4 indexed citations
12.
Sato, Hidetaka, et al.. (1992). Apotransferrins from several species promote thyroid hormone-dependent rat pituitary tumor cell growth in iron-restricted serum-free defined culture. Molecular and Cellular Endocrinology. 83(2-3). 239–251. 8 indexed citations
13.
Sirbasku, David A., et al.. (1991). Thyroid hormone-dependent pituitary tumor cell growth in serum-free chemically defined culture. A new regulatory role for apotransferrin. Biochemistry. 30(30). 7466–7477. 28 indexed citations
14.
Nishi, Yoshisuke, et al.. (1991). Initiating activity of eight pyrolysates of carbohydrates in a two-stage mouse skin tumorigenesis model. Carcinogenesis. 12(7). 1169–1173. 19 indexed citations
16.
Hasegawa, Ryohei, Fumio Furukawa, Kazuhiro Toyoda, et al.. (1990). Urothelial Damage and Tumor Initiation by Urinary Metabolites of Sodium o‐Phenylphenate in the Urinary Bladder of Female Rats. Japanese Journal of Cancer Research. 81(5). 483–488. 6 indexed citations
17.
Takahashi, Michihito, Hidetaka Sato, Kazuhiro Toyoda, et al.. (1989). Sodium o-phenylphenate (OPP-Na) promotes skin carcinogenesis in CD-1 female mice initiated with 7, 12-dethylbenz[a]anthracene. Carcinogenesis. 10(7). 1163–1167. 8 indexed citations
18.
Imaida, Katsumi, Hidetaka Sato, Hideaki Okamiya, Michihito Takahashi, & Yûzo Hayashi. (1989). Enhancing Effect of High Fat Diet on 4‐Nitroquinoline 1‐Oxide‐induced Pulmonary Tumorigenesis in ICR Male Mice. Japanese Journal of Cancer Research. 80(6). 499–502. 13 indexed citations
20.
Jang, Ja June, Mami Takahashi, Fumio Furukawa, et al.. (1987). Long-term in vivo carcinogenicity study of phenytoin (5,5-diphenylhydantoin) in F344 rats. Food and Chemical Toxicology. 25(9). 697–702. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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