Hideo Otani

1.3k citations
22 papers · 905 indexed · h-index 15

Hideo Otani

22 papers receiving 879 citations

Peers

Hideo Otani
Comparison fields: 5 of 66
  • Cardiology and Cardiovascular Medicine 640
  • Developmental Neuroscience 39
  • Molecular Biology 632
  • Pathology and Forensic Medicine 151
  • Cellular and Molecular Neuroscience 119
Replace Andrew K. Snabaitis with:
Andrew K. Snabaitis United Kingdom
James S. Heisner United States
Geoffrey Teixeira France
Fearghas O’Cochlain United States
A Sen United States
Renee Wong United States
James W. Mockridge United Kingdom
Domenico M. Taglieri United States
Kenichi Hongo Japan
Irène Papageorgiou Switzerland
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Citations per field
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Citations per year

Countries citing papers authored by Hideo Otani

Since Specialization
Citations

This map shows the geographic impact of Hideo Otani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideo Otani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideo Otani more than expected).

Fields of papers citing papers by Hideo Otani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideo Otani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideo Otani. The network helps show where Hideo Otani may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Hideo Otani, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Hideo Otani Line = papers co-authored together Hideo Otani links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20207
2 20049
3 2003181
4 200218
5 200226
6 200270
7 200124
8 200153
9 200150
10 200020
11 200041
12 20009
13
Kono, Y. et al. The properties of the Kir6.1-6.2 tandem channel co-expressed with SUR2A. Pflugers. Arch. 440, 692-698
200014
14 199914
15 199837
16 199832
17 199843
18 19959
19 19901
20 19823

About Hideo Otani

Hideo Otani is a scholar working on Emergency Medicine, Cardiology and Cardiovascular Medicine and Pathology and Forensic Medicine, having authored 22 papers that have together received 905 indexed citations. Recurring topics across this work include Ion channel regulation and function (12 papers), Cardiac electrophysiology and arrhythmias (11 papers), Cardiac Arrest and Resuscitation (5 papers), Cardiac Ischemia and Reperfusion (5 papers), Neuroscience and Neuropharmacology Research (3 papers), Receptor Mechanisms and Signaling (3 papers), Cholesterol and Lipid Metabolism (2 papers) and Cardiac pacing and defibrillation studies (2 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (640 citations), Developmental Neuroscience (39 citations) and Molecular Biology (632 citations). Hideo Otani has collaborated with scholars based in Japan, United States and Croatia. Frequent co-authors include Minoru Horie, Shigetake Sasayama, Tomohiko Ai, Tomoyuki Kubota, Makoto Takano, T. Ninomiya, Keiko Tsuji, Kotoe Takenaka, Hidetada Yoshida and Atsushi Kobori. Their work appears in journals such as Circulation, Circulation Research and The Journal of Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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