Hideo Hamada
- Neurology top 2%
- Surgery top 10%
- Cellular and Molecular Neuroscience top 5%
- Pediatrics, Perinatology and Child Health top 10%
- Public Health, Environmental and Occupational Health top 10%
- Co-authors
- Shunro EndoNakamasa HayashiYutaka HirashimaMasanori KurimotoHideki OrigasaHiromichi YamamotoNobuhisa MatsumuraNaoya Kuwayama
- Topics
- Cerebrospinal fluid and hydrocephalus (34 papers)Fetal and Pediatric Neurological Disorders (18 papers)Intracranial Aneurysms: Treatment and Complications (15 papers)
- Partner nations
- JapanSwitzerlandPortugal
In The Last Decade
Hideo Hamada
71 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 81
- Neurology 556
- Surgery 411
- Cellular and Molecular Neuroscience 299
- Pediatrics, Perinatology and Child Health 156
- Public Health, Environmental and Occupational Health 150
Countries citing papers authored by Hideo Hamada
This map shows the geographic impact of Hideo Hamada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideo Hamada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideo Hamada more than expected).
Fields of papers citing papers by Hideo Hamada
This network shows the impact of papers produced by Hideo Hamada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideo Hamada. The network helps show where Hideo Hamada may publish in the future.
Co-authorship network of co-authors of Hideo Hamada
This figure shows the co-authorship network connecting the top 25 collaborators of Hideo Hamada. A scholar is included among the top collaborators of Hideo Hamada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hideo Hamada. Hideo Hamada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 13 | |
| 3 | 13 | |
| 4 | 23 | |
| 5 | 4 | |
| 6 | 21 | |
| 7 | 25 | |
| 8 | 30 | |
| 9 | 1 | |
| 10 | 4 | |
| 11 | 5 | |
| 12 | 173 | |
| 13 | 6 | |
| 14 | 32 | |
| 15 | 12 | |
| 16 | 1 | |
| 17 | 36 | |
| 18 | 20 | |
| 19 | 42 | |
| 20 | 9 |
About Hideo Hamada
Hideo Hamada is a scholar working on Neurology, Cellular and Molecular Neuroscience and Pediatrics, Perinatology and Child Health, having authored 73 papers that have together received 1.1k indexed citations. Recurring topics across this work include Cerebrospinal fluid and hydrocephalus (34 papers), Fetal and Pediatric Neurological Disorders (18 papers) and Intracranial Aneurysms: Treatment and Complications (15 papers). The work is most often cited by research in Neurology (556 citations), Cellular and Molecular Neuroscience (299 citations) and Surgery (411 citations). Hideo Hamada has collaborated with scholars based in Japan, Switzerland and Portugal. Frequent co-authors include Shunro Endo, Nakamasa Hayashi, Yutaka Hirashima, Masanori Kurimoto, Hideki Origasa, Hiromichi Yamamoto, Nobuhisa Matsumura, Naoya Kuwayama, Akira Takaku and Shoichi Nagai. Their work appears in journals such as Stroke, FEBS Letters and Journal of neurosurgery.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.